WEBVTT

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Hey guys, this right here is going to be PALS,

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the Pediatric Advanced Life Support. And the

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way it's going to go is I'm going to try to keep

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it in this order. The systematic approach to

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the pediatric patients, pediatric shock recognition,

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pediatric management of shock, recognizing respiratory

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distress, managing respiratory distress. recognizing

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cardiac arrest, management of cardiac arrest,

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arrhythmias, recognition of those arrhythmias,

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and then management of those arrhythmias. I don't

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remember PALS being this deep, but again, it's

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been almost two years since I've taken PALS,

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but it seems like it's a lot more in depth than

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just your normal ACLS. So this material is coming

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straight from the book. For the course just make

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sure that you guys are reading that material

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Because I did not add some stuff in there like

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team dynamics or resources for respiratory care

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or Management of the post cardiac arrest, you

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know stuff like that. I didn't include that into

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this stuff So if you guys need that or want that,

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then by all means, read the book or, you know,

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Google it or whatever. But this right here is

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going to be the intro for the pals stuff. So

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I hope you guys enjoy the pediatric advanced

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life support material for the course. This is

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all going to happen super fast. Welcome to the

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emergency room. Welcome to the deep dive. If

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you're joining us, you are probably bracing for

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the PLS certification exam. or maybe walking

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into a trauma bay right now. Exactly. And you

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need that lightning fast, high stakes clinical

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decision tree for pediatric arrhythmias. This

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isn't just about theory. No, not at all. This

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is about saving myocardium. And I mean, at the

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end of the day, saving a child's life. So this

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deep dive is really for that seasoned critical

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care or ER nurse. Right. We're not here to just,

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you know, recite algorithms from a card. We are

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here to unlock the clinical reasoning, the why

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behind every single critical step. Especially

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when that heart rate is threatening their stability.

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Yes. Our mission is singular, mastering the management

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of pediatric arrhythmias. We need to know how

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to quickly, and I mean quickly, determine instability

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and treat symptomatic bradycardia and tachycardia

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before they spiral. Before they go into irreversible

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shock or cardiac arrest. Presesimente. And we

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have to begin with the non -negotiable foundation

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of all pediatric critical care. Before we even

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glance at the monitor, before we think about

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a drug or a shock, what is the absolute priority?

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It's always the ABCs. Airway, breathing, and

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circulation. And at the same time, seeking and

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treating that underlying cause. That's the step

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that really separates the pros, isn't it? It

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is. It separates a true critical care provider

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from someone who just reads the card. You support

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oxygenation. You support ventilation. And then

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and only then do you look at the rhythm and decide

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if it's the main problem or just a symptom. OK,

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so let's really impact this. We're talking about

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an abnormal heart rate or rhythm. And pediatric

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hearts run faster than adult hearts to begin

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with. So what's the critical threshold? What's

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that specific line we cross where we go from,

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say, monitoring and diagnosing to emergency life

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-saving intervention? That line is hemodynamic

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instability, an abnormal rate or rhythm because

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a true emergency when it's causing significant

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cardiopulmonary compromise. And you have to figure

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that out in seconds. In seconds. You have to

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determine if the arrhythmia is the cause of the

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problem or just the result of it. The PLNS guidelines

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give us five, you know, need -to -know indicators

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that define instability. And these require immediate

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treatment. No waiting for labs, no... No waiting

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for an expert consult to call you back. You act.

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Okay, so let's detail those five signs, but with

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a focus on clinical insight. What do they actually

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look like at the bedside? Because sometimes these

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signs can be pretty subtle, especially in early

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shock. Absolutely. So number one is respiratory

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distress or failure. If that rapid heart rate

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is taxing the body so much that the child just

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can't keep up with their own oxygenation or ventilation,

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that's an emergency. Makes sense. The second

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is shock with poor end -organ perfusion. Now,

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this is often associated with hypotension, but

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here is the critical clinical parole for you.

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Okay. Shock can happen without hypotension. especially

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in kids. They compensate like crazy. They clamp

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down those vessels to maintain blood pressure

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right until the very end. So you can't wait for

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the cuff reading? You cannot wait for the cuff.

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You look for delayed capillary refill. You look

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for cool extremities, thready pulses. You look

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for those things before the blood pressure finally

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drops. That distinction is huge. I feel like

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there's always a tendency to wait for that number

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on the monitor, but the kid is often circling

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the drain long before that cuff reads low. They

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are. So what about the neurological indicators?

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The third is irritability, or decreased level

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of consciousness, you know, altered mental status.

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This is arguably the most powerful early indicator

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of poor cerebral perfusion. So a kid who's normally

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alert, but is now, what, lethargic? Agitated?

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Exactly. A child who suddenly becomes lethargic

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or agitated is basically screaming that their

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brain is not getting enough oxygenated blood.

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Fourth, we look for chest pain or a vague feeling

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of discomfort. More common in older kids, I imagine.

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Much more common in older kids who can actually

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tell you. It signifies myocardial ischemia because

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the heart is beating so fast it can't perfuse

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itself. And then finally, number five is sudden

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collapse. OK. If any of those five are present,

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the clock is running and you move straight to

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intervention. That makes the nursing priority.

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Crystal clear then. If we see symptomatic bradycardia

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or tachycardia, box one in the algorithm kicks

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in. Walk us through those mandatory initial steps

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at the bedside. So these steps have to happen

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simultaneously, right, as you're doing your assessment.

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First, maintain a patent airway and assist breathing.

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That might just be a manual airway maneuver.

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A jaw thrust? Simple jaw thrust. Then immediately

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provide high concentration oxygen. usually a

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non -rebreather, critically establish vascular

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access IV or IO immediately. You can't give life

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-saving drugs without it. You can't. And if you

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even suspect instability, you call for the IO

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supplies right away. Don't wait for three failed

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peripheral IV attempts. Right. And while the

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team is working on ABCs and getting access, we're

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getting the monitors ready? Precisely. Attach

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the cardiac monitor, the defibrillator, the pulse

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oximeter, get them on right away. And here's

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where we hit that diagnostic caveat. The 12 lead

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ECG. Right. Obtain a 12 lead ECG if available.

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But the pay the last guideline is rigid on this.

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Do not delay therapy waiting for it. If the patient

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is unstable, treatment comes first. Treatment

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always supersedes a precise diagnosis in that

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moment. You can get your blood pressure, your

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blood gas, glucose, tox screens, but urgent intervention

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is the priority. The information you get from

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a 12 lead does not outweigh the risk of delaying

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cardioversion in an unstable kid. That's such

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a critical point for the ER nurse. You might

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feel this pressure to get the perfect strip for

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the cardiologist, but if the patient is seizing

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from poor perfusion, the strip is irrelevant

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until you fix the rhythm. Exactly. Stabilize

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the patient first. Don't worry about perfecting

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the paperwork. OK, let's move on. Here's where

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we make a really fundamental physiological distinction.

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Radycardia in an adult, that's often a primary

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electrical failure, right? Like sick sinus syndrome

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or a block. Right. But in kids, it's a totally

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different ballgame. It is fundamentally different.

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Pediatric bradycardia is usually secondary to

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a respiratory or metabolic issue. It's that late

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bad sign that profound hypoxia has set in. That

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is the core clinical truth, isn't it? It is.

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You have to remember, pediatric cardiac output

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is highly rate dependent. They rely on a high

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heart rate to move volume around. They can't

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increase their stroke volume like adults can.

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Exactly. So when that rate slows down, it means

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the heart muscle is exhausted or it's ischemic

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or it's been poisoned. The pediatric bradycardia

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algorithm is specifically structured to treat

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that secondary cause first. So when you hit the

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algorithm, you immediately reassess for compromised

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hypertension, altered mental status, signs of

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shock. And if it's there, you move fast. You

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go very fast. Let's talk about the ultimate red

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flag in this algorithm, the CPR trick. That's

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box three. If everything we've done to improve

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oxygenation and ventilation has failed, when

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do we actually start chest compressions on a

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child who still technically has a pulse? This

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is a decision point that gives a lot of providers

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pause, but Paul is very clear on it. If bradycardia

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persists, and we define that as a heart rate

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less than 60 beats per minute. A hard line at

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60. A hard line at 60. If it's under 60 despite

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adequate oxygenation and ventilation, You start

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high quality CPR immediately. Because a persistent

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rate below 60 in a symptomatic child means the

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heart is failing to generate enough cardiac output

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to sustain life. External circulation is now

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required. The lack of oxygen has depressed the

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myocardium so severely that the body just can't

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compensate anymore. two minutes and we're sure

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their respiratory effort is supported, we compress.

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You compress. Let's quickly review the metrics

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for high quality CPR because execution here matters

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so much more than intention. These are foundational

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facts you have to know cold. Push fast. 100 to

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120 compressions per minute. Push hard. Yeah.

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At least one third the depth of the chest. Which

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is about 1 .5 inches in an infant or two inches

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in a child. Right. And you have to ensure complete

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chest recoil. Minimizing interruptions is vital.

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Aim for a compression fraction of at least 60%.

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And the ventilation ratio. If you're manually

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ventilating, it's 15 .2 for two providers, or

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30 .2 if you're a lone rescuer, that is, until

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they're intubated. After that, compressions are

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continuous and you give breath every two to three

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seconds. Okay, so once we're doing high quality

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CPR because of persistent bradycardia, we move

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immediately to the drugs. And the primary drug,

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the heavy hitter, is epinephrine. Epinephrine

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is the central nervous system drug in cardiac

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arrest and severe bradycardia. Its mechanism

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is beautifully dual. And if you understand this,

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you understand why it's the first choice. OK.

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Think of the alpha effect as tightening the pipes.

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Vasoconstriction. Exactly. Vasoconstriction.

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This increases systemic vascular resistance,

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which raises aortic diastolic pressure. That

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is crucial for perfusing the coronary arteries

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in the brain. Then you have the beta effect.

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The beta effect is speeding up the pump. Speeding

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up the pump. It increases heart rate and cardiac

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contractility. So this simultaneous action supports

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both oxygenation and perfusion when that myocardium

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is just completely compromised. So we're not

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just trying to make the heart beat faster. We're

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trying to literally squeeze the arteries to drive

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blood back into the heart muscle itself. You

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have to get that coronary perfusion pressure

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up. The indication is clear. Symptomatic bradycardia

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that persists despite effective oxygenation,

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ventilation, and CPR. And the dose. The standard

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IV or IO dose is 0 .01 millochi kilogram. That's

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0 .1 millochi IG of the standard 1 .00 concentration.

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And you repeat that dose every three to five

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minutes. And you have to be strict with that

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timing. You have to be. Stick to that three to

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five minute interval. What about the less reliable

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route, endotracheal? The ET route. Absorption

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is unreliable. It's inconsistent. and it needs

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a much higher dose. If you have absolutely no

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IV or IO access, the ET dose is 10 times higher,

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0 .1 milliliter kilogram. But that's a last resort.

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It has to be a rescue ride only. Your priority

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is to get IO access immediately. Okay, next up

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is atropine, the parasympathetic. Its role is

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very specific in PAELs. Very specific. Atropine

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sulfate is an anti -colonergic drug. Its mechanism

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is to accelerate the sinus or atrial pacemakers

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and enhance AV conduction by blocking vagal tone.

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So it's the drug of choice for bradycardia that's

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specifically caused by increased vagal tone.

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Right, like from intubation attempts or surgical

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stimulus or poisoning by colonergic agents or

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for a primary AV block. You mentioned before

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why epinephrine is generally preferred over atropine

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in the sort of general symptomatic bradycardia

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algorithm. Can you just reiterate that for us?

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Yeah. I mean, if the child is profoundly hypoxic

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or ischemic, atropine won't fix the underlying

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problem. The muscle is just too sick. Epinephrine's

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broader ALSA and beta effects are just superior

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for that generally compromised myocardium. So

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if the child doesn't respond to atropine? You

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pivot. You pivot immediately to epinephrine and

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compression. Atropine is a targeted drug for

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a very targeted cause. What's the dose for atropine?

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It's 0 .02mg in kilogram IV or IO. You must respect

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the minimum dose of 0 .1mg. If you give less,

00:13:43.139 --> 00:13:45.120
you can get a paradoxical slowing of the heart

00:13:45.120 --> 00:13:48.299
rate. Oh, wow. Yeah. And the maximum single dose

00:13:48.299 --> 00:13:51.120
is 0 .5mg. You can repeat it once. And the clinical

00:13:51.120 --> 00:13:53.480
pearl about its use in heart blocks? Atropine

00:13:53.480 --> 00:13:56.039
may be useful for a symptomatic second degree

00:13:56.039 --> 00:13:59.500
AV block. Either Mobitz is the first, and third

00:13:59.500 --> 00:14:02.539
degree AV block. But, and this is a big but,

00:14:02.940 --> 00:14:05.860
if the block is low down in the bundle of his

00:14:05.860 --> 00:14:08.779
or the Purkinje Fibers Atropine is unlikely to

00:14:08.779 --> 00:14:11.220
work. And then you're looking at pacing. Then

00:14:11.220 --> 00:14:13.700
you need to prepare for transcutaneous pacing,

00:14:13.960 --> 00:14:16.980
which is... Honestly, very challenging in pediatric

00:14:16.980 --> 00:14:19.820
patients and usually a bridge to getting transvenous

00:14:19.820 --> 00:14:22.419
pacing after they're stabilized. Okay, now we

00:14:22.419 --> 00:14:24.919
get to what I think is the core of critical nursing

00:14:24.919 --> 00:14:27.919
in PALS. If we give epinephrine without fixing

00:14:27.919 --> 00:14:29.799
the root problem, the code is going to fail.

00:14:30.159 --> 00:14:32.840
We have to master the H's and T's. This is everything.

00:14:33.200 --> 00:14:35.500
What are the two most common potentially reversible

00:14:35.500 --> 00:14:37.779
causes of pediatric bradycardia that we see in

00:14:37.779 --> 00:14:40.440
the trauma bay? Hypoxia and hydrogen ion, which

00:14:40.440 --> 00:14:43.379
is acidosis. If a child's rate drops below 80

00:14:43.379 --> 00:14:45.879
or certainly below 60, your first thought has

00:14:45.879 --> 00:14:47.759
to be their airway and their breathing. Those

00:14:47.759 --> 00:14:49.919
two have to be addressed aggressively. Immediately

00:14:49.919 --> 00:14:51.840
and aggressively. So let's run through the full

00:14:51.840 --> 00:14:54.539
table of reversible causes. Give us the why and

00:14:54.539 --> 00:14:56.600
the specific treatment for the ER nurse on the

00:14:56.600 --> 00:14:59.259
front line. Okay, for hypoxia, the treatment

00:14:59.259 --> 00:15:02.879
is foundational. Give high concentration supplementary

00:15:02.879 --> 00:15:05.360
oxygen with assisted ventilation. You must ensure

00:15:05.360 --> 00:15:07.740
the chest rises symmetrically and that the pulse

00:15:07.740 --> 00:15:10.139
ox improves. And if it doesn't? If it doesn't,

00:15:10.200 --> 00:15:12.480
you check for a misplaced tube or a foreign body

00:15:12.480 --> 00:15:14.779
obstruction. You troubleshoot that airway. Okay,

00:15:15.299 --> 00:15:18.559
next. Hydrogen ion. Acidosis. Right. Remember,

00:15:18.879 --> 00:15:20.980
acidosis depresses myocardial function. It makes

00:15:20.980 --> 00:15:23.600
the heart muscle weak. So you treat the respiratory

00:15:23.600 --> 00:15:26.080
component first. You provide ventilation to blow

00:15:26.080 --> 00:15:28.820
off CO2 and correct that respiratory acidosis.

00:15:28.899 --> 00:15:31.470
What about sodium bicarbonate? Sodium bicarb

00:15:31.470 --> 00:15:34.610
may be considered in a severe documented metabolic

00:15:34.610 --> 00:15:37.629
acidosis or for hyperkalemia, but you always

00:15:37.629 --> 00:15:40.830
ensure ventilation is maximized first. Bicarb

00:15:40.830 --> 00:15:43.009
generates CO2, and if you can't ventilate that

00:15:43.009 --> 00:15:45.490
out, you paradoxically make the intracellular

00:15:45.490 --> 00:15:49.210
acidosis worse. Good point. Okay, moving to electrolytes

00:15:49.210 --> 00:15:51.929
hyperkalemia. We see this in renal failure or

00:15:51.929 --> 00:15:54.649
severe crush injuries. Why does it cause such

00:15:54.649 --> 00:15:57.740
profound bradycardia? I potassium hyperkalemia.

00:15:57.740 --> 00:16:00.960
It destabilizes the cardiac cell membrane. On

00:16:00.960 --> 00:16:03.879
the ECG, you'll see those classic tall peak T

00:16:03.879 --> 00:16:06.759
waves followed by a widening of the QRS and then

00:16:06.759 --> 00:16:09.299
ultimately a sine wave arrest. And the treatment

00:16:09.299 --> 00:16:12.159
is multifaceted. Very. You have to stabilize

00:16:12.159 --> 00:16:14.379
the myocardium first with calcium chloride or

00:16:14.379 --> 00:16:16.840
calcium gluconate that protects the heart from

00:16:16.840 --> 00:16:19.220
the potassium. Then you shift the potassium back

00:16:19.220 --> 00:16:21.899
into the cells using insulin and dexrose and

00:16:21.899 --> 00:16:25.289
potentially sodium bicarb or albuterol. But calcium

00:16:25.289 --> 00:16:27.850
is the immediate lifesaver. Calcium is the first

00:16:27.850 --> 00:16:30.029
thing you give. It's the immediate lifesaver.

00:16:30.350 --> 00:16:33.590
Next, temperature. Hypothermia. I feel like this

00:16:33.590 --> 00:16:36.149
is an often ignored cause, especially if the

00:16:36.149 --> 00:16:38.250
patient was found down. It is. If the child is

00:16:38.250 --> 00:16:40.549
hypothermic, you have to warm them gently but

00:16:40.549 --> 00:16:43.309
effectively. Core temperatures below 30 degrees

00:16:43.309 --> 00:16:45.590
Celsius make the myocardium highly irritable

00:16:45.590 --> 00:16:47.950
and refractory to standard resuscitation drugs.

00:16:48.049 --> 00:16:51.029
So you defer shocking and drugs. You defer shocking

00:16:51.029 --> 00:16:53.809
and drugs until the core temp is above 30 degrees

00:16:53.809 --> 00:16:56.370
Celsius, if possible. And you have to be careful

00:16:56.370 --> 00:16:58.990
not to trigger dysrhythmia by moving the patient

00:16:58.990 --> 00:17:01.850
roughly during warming. That cold myocardium

00:17:01.850 --> 00:17:04.890
can flip into VFib very easily. OK. Then we have

00:17:04.890 --> 00:17:07.750
the massive category of toxins, poisons, and

00:17:07.750 --> 00:17:10.269
drugs. How do we even begin to narrow the treatment

00:17:10.269 --> 00:17:13.450
here? This requires a highly specific antidote

00:17:13.450 --> 00:17:16.750
or very targeted supportive care. If it's a calcium

00:17:16.750 --> 00:17:19.950
channel blocker or beta blocker overdose, your

00:17:19.950 --> 00:17:22.369
first line therapy is often high -dose glucagon,

00:17:22.430 --> 00:17:25.309
calcium, or high -dose insulin euglycemic therapy,

00:17:25.710 --> 00:17:28.140
not just epi. And opioids. If it's an opioid

00:17:28.140 --> 00:17:30.779
overdose, you give Narcan. You have to ask about

00:17:30.779 --> 00:17:33.799
recent medications, access to drugs, or suicidal

00:17:33.799 --> 00:17:36.119
ideation immediately. Finally, the warning sign

00:17:36.119 --> 00:17:39.180
in the neuro or trauma patient, bradycardia associated

00:17:39.180 --> 00:17:41.480
with the head injury. This is the classic Cushing's

00:17:41.480 --> 00:17:44.099
Reflex Hypertension, bradycardia, and irregular

00:17:44.099 --> 00:17:47.859
respirations. It signifies severe increased intracranial

00:17:47.859 --> 00:17:50.359
pressure or ICP. So the body is clamping down

00:17:50.359 --> 00:17:52.500
the arteries to try and force blood into the

00:17:52.500 --> 00:17:54.880
skull. causing the hypertension, and that high

00:17:54.880 --> 00:17:57.420
blood pressure triggers a reflex slowing of the

00:17:57.420 --> 00:17:59.579
heart rate. So the bradycardia is actually a

00:17:59.579 --> 00:18:01.319
protective mechanism, even though it's failing.

00:18:01.599 --> 00:18:03.940
What's the nursing intervention? Your first steps

00:18:03.940 --> 00:18:07.180
are oxygenation and ventilation. If there are

00:18:07.180 --> 00:18:09.539
signs of impending herniation, like pupillary

00:18:09.539 --> 00:18:12.799
changes or posturing, a brief period of mild

00:18:12.799 --> 00:18:15.200
hyperventilation can be used as a temporary rescue

00:18:15.200 --> 00:18:19.119
therapy to acutely decrease that ICP. But ultimately,

00:18:19.339 --> 00:18:22.430
this child needs a surgeon. The bradycardia isn't

00:18:22.430 --> 00:18:24.349
a heart problem. It's a brain problem. You have

00:18:24.349 --> 00:18:26.970
to treat the head. Okay, switching gears completely

00:18:26.970 --> 00:18:30.029
to fast rates. With tachycardia, speed matters.

00:18:30.589 --> 00:18:32.589
A heart rate that's too fast doesn't allow enough

00:18:32.589 --> 00:18:34.869
diastolic filling time, and that leads to shock

00:18:34.869 --> 00:18:37.970
really fast. Right. PLS is very clear here. If

00:18:37.970 --> 00:18:40.589
the child is unstable, do not delay emergency

00:18:40.589 --> 00:18:42.829
treatment while you wait for an expert consult.

00:18:43.029 --> 00:18:46.450
The initial management is still your ABCs, monitoring,

00:18:46.690 --> 00:18:49.529
and getting vascular access. The problem here

00:18:49.529 --> 00:18:51.809
is that the rapid rate reduces stroke volume

00:18:51.809 --> 00:18:54.390
so drastically that cardiac output just falls

00:18:54.390 --> 00:18:56.630
off a cliff. So unlike bradycardia, where we

00:18:56.630 --> 00:18:58.829
focus on the secondary cause, with tachycardia,

00:18:58.849 --> 00:19:00.630
we're really focused on the rhythm itself if

00:19:00.630 --> 00:19:03.009
it's pathological. Exactly. The critical moment

00:19:03.009 --> 00:19:06.369
in the tachycardia algorithm is box two. Evaluate

00:19:06.369 --> 00:19:09.730
the QRS duration. This immediately splits our

00:19:09.730 --> 00:19:12.410
pathway and dictates our entire therapeutic approach.

00:19:12.630 --> 00:19:15.609
It does. The QRS duration tells us where the

00:19:15.609 --> 00:19:18.420
electrical impulse is coming from. If it originates

00:19:18.420 --> 00:19:20.740
above the ventricles, the path down is normal

00:19:20.740 --> 00:19:23.539
and fast, so you get a narrow QRS. And if it

00:19:23.539 --> 00:19:25.460
originates in the ventricles? Then the path is

00:19:25.460 --> 00:19:28.839
slow and aberrant and you get a wide QRS. So

00:19:28.839 --> 00:19:31.279
an narrow complex is defined as less than or

00:19:31.279 --> 00:19:35.259
equal to 0 .09 seconds. This suggests probable

00:19:35.259 --> 00:19:37.980
sinus tachycardia or supraventricular tachycardia

00:19:37.980 --> 00:19:41.039
SVT. And a wide complex is greater than 0 .09

00:19:41.039 --> 00:19:43.900
seconds. Which is presumed ventricular tachycardia

00:19:43.900 --> 00:19:47.079
VT or maybe SVT with some aberrant conduction.

00:19:47.319 --> 00:19:49.220
Okay, let's talk about the stable patient. If

00:19:49.220 --> 00:19:51.539
the child has good pulses, good perfusion, their

00:19:51.539 --> 00:19:54.220
mentation is intact. We have a little bit of

00:19:54.220 --> 00:19:56.119
time, precious time to think and differentiate

00:19:56.119 --> 00:19:59.039
between sinus tach and SVT. And this distinction

00:19:59.039 --> 00:20:01.559
is the single most common diagnostic error I

00:20:01.559 --> 00:20:04.720
see in the PEDS ER. I believe it! It is! Because

00:20:04.720 --> 00:20:07.180
if you treat ST like it's SVT, you're treating

00:20:07.180 --> 00:20:09.700
the compensation, not the problem, and you risk

00:20:09.700 --> 00:20:12.029
a really serious error. So we have to distinguish

00:20:12.029 --> 00:20:14.349
them. OK, let's nail down those differentiating

00:20:14.349 --> 00:20:17.410
factors. Sinus tachycardia, or ST, is compatible

00:20:17.410 --> 00:20:20.809
with a known cause. Fever, pain, anxiety, dehydration.

00:20:21.230 --> 00:20:23.490
You look for normal P waves. Crucially, the R

00:20:23.490 --> 00:20:25.869
to R interval is variable. The heart rate speeds

00:20:25.869 --> 00:20:28.349
up and slows down. It does, depending on the

00:20:28.349 --> 00:20:30.670
child's activity or hydration. And the rate limits

00:20:30.670 --> 00:20:33.529
are usually less than 220 in infants and less

00:20:33.529 --> 00:20:36.529
than 180 in children. ST is the heart just doing

00:20:36.529 --> 00:20:40.130
its job to compensate. Okay, and SVT is the pathological

00:20:40.130 --> 00:20:43.150
one, the sudden rhythm. SVT is sudden onset.

00:20:43.829 --> 00:20:46.750
The history is also not compatible with ST. The

00:20:46.750 --> 00:20:48.549
heart rate is usually constant and does not vary

00:20:48.549 --> 00:20:51.509
with activity. The RR is fixed? It's fixed. P

00:20:51.509 --> 00:20:53.750
waves are usually absent, or they're merged with

00:20:53.750 --> 00:20:56.109
the QRS, or they look abnormal, and the rates

00:20:56.109 --> 00:20:59.089
are extreme. Usually 220 are greater in infants

00:20:59.089 --> 00:21:02.089
and 180 are greater in children. If the rate

00:21:02.089 --> 00:21:04.890
is that high and it was sudden, it's SVT until

00:21:04.890 --> 00:21:07.599
you prove otherwise. So if it's sinus tachycardia,

00:21:07.740 --> 00:21:10.200
we have to resist that urge to slow the heart

00:21:10.200 --> 00:21:12.519
down with drugs. You absolutely have to. You

00:21:12.519 --> 00:21:15.359
treat the cause. You give a fluid bolus if they're

00:21:15.359 --> 00:21:17.660
dehydrated or acetaminophen if they're febrile.

00:21:18.000 --> 00:21:20.319
You do not give adenosine or other antiarrhythmics.

00:21:20.400 --> 00:21:23.200
If you do, you take away the child's essential

00:21:23.200 --> 00:21:25.680
compensatory mechanism and you risk sending them

00:21:25.680 --> 00:21:29.529
into shock. But if we have stable SVT, we move

00:21:29.529 --> 00:21:32.410
to rhythm control, starting with vagal maneuvers.

00:21:32.569 --> 00:21:35.069
Always attempt these first. They stimulate the

00:21:35.069 --> 00:21:37.509
vagus nerve and slow down that AV conduction.

00:21:38.130 --> 00:21:40.329
If they work, they terminate the rhythm instantly.

00:21:40.869 --> 00:21:43.990
No drugs needed. But the technique is very age

00:21:43.990 --> 00:21:46.920
dependent. So what are the specific nursing coaching

00:21:46.920 --> 00:21:49.579
techniques here? For infants, the safest most

00:21:49.579 --> 00:21:51.900
effective method is the ice water facial immersion.

00:21:52.559 --> 00:21:54.660
You place a bag of ice water over the upper half

00:21:54.660 --> 00:21:57.480
of the face for about 15 to 20 seconds. But don't

00:21:57.480 --> 00:21:59.839
cover their nose or mouth? Critically, do not

00:21:59.839 --> 00:22:01.730
cover their nose or mouth. For older children

00:22:01.730 --> 00:22:03.849
who can follow directions, the modern approach

00:22:03.849 --> 00:22:06.210
is a modified Valsalva maneuver. How does that

00:22:06.210 --> 00:22:08.549
work? You have them blow forcefully into a 10

00:22:08.549 --> 00:22:11.269
millil syringe trying to move the plunger, or

00:22:11.269 --> 00:22:13.809
through an obstructed straw for 15 seconds while

00:22:13.809 --> 00:22:15.769
they're lying flat. And then you immediately

00:22:15.769 --> 00:22:17.809
raise their legs up to 45 degrees for another

00:22:17.809 --> 00:22:21.670
15 seconds. That combination significantly increases

00:22:21.670 --> 00:22:24.609
the success rate. Okay, if vagal maneuvers fail...

00:22:24.640 --> 00:22:27.500
We move to the definitive drug for stable SVT,

00:22:27.740 --> 00:22:29.940
Adenosine. Adenosine is the drug of choice for

00:22:29.940 --> 00:22:34.160
reentrant SVT. Its mechanism is pretty dramatic.

00:22:34.359 --> 00:22:36.960
It blocks conduction through the AV node and

00:22:36.960 --> 00:22:39.190
temporarily stops the heart. Let's spend some

00:22:39.190 --> 00:22:41.490
time on the dosing and maybe more importantly

00:22:41.490 --> 00:22:43.430
the nursing technique. This is where clinical

00:22:43.430 --> 00:22:46.130
failure so often happens. It really is. Dosing

00:22:46.130 --> 00:22:50.289
is 0 .1mgkg IV or IO with a max first dose of

00:22:50.289 --> 00:22:52.930
6mg. If it's ineffective, you immediately give

00:22:52.930 --> 00:22:56.190
the second dose, 0 .2mgkg with a max second dose

00:22:56.190 --> 00:22:59.049
of 12mg. And the technique is everything. It's

00:22:59.049 --> 00:23:01.759
paramount. A tenocene has an incredibly short

00:23:01.759 --> 00:23:04.619
half -life, less than 10 seconds. You must use

00:23:04.619 --> 00:23:07.619
a central or proximal IV line if you can. You

00:23:07.619 --> 00:23:09.720
give the doses a rapid IV bolus and you follow

00:23:09.720 --> 00:23:12.720
it immediately with a rapid flush of 5 to 10

00:23:12.720 --> 00:23:15.240
milliliter of normal saline, elevating the arm

00:23:15.240 --> 00:23:18.319
at the same time. This just shoves the drug into

00:23:18.319 --> 00:23:20.460
the central circulation so it can get to the

00:23:20.460 --> 00:23:22.980
AV node before it gets metabolized. Okay, now

00:23:22.980 --> 00:23:25.859
let's shift to the wide QRS, greater than 0 .09

00:23:25.859 --> 00:23:29.079
seconds, but in a stable patient. This is presumed

00:23:29.079 --> 00:23:31.740
VT until proven otherwise, and the stakes just

00:23:31.740 --> 00:23:33.599
immediately go up, even if the patient looks

00:23:33.599 --> 00:23:36.259
okay for the moment. A wide QRS strongly suggests

00:23:36.259 --> 00:23:38.940
a ventricular origin, which carries a high risk

00:23:38.940 --> 00:23:41.599
of degenerating into VFib. If the rhythm has

00:23:41.599 --> 00:23:45.319
a uniform QRS morphology and a regular RR interval,

00:23:45.599 --> 00:23:48.200
and the patient is stable, we treat it as VT.

00:23:48.599 --> 00:23:51.220
This situation, box 12 demands immediate expert

00:23:51.220 --> 00:23:54.289
consultation. A cardiologist or an ICU doc? Yes,

00:23:54.410 --> 00:23:55.690
but you have to start treatment while you're

00:23:55.690 --> 00:23:57.329
waiting for them to arrive. And provided the

00:23:57.329 --> 00:23:59.670
patient is truly stable, we consider pharmacologic

00:23:59.670 --> 00:24:03.450
conversion here. Yes. The primary drugs are amiodarone

00:24:03.450 --> 00:24:07.349
or prokanamide. You consider amiodarone at 5

00:24:07.349 --> 00:24:11.049
millijoy kilogram given IV or IO, but slowly,

00:24:11.190 --> 00:24:14.750
over 20 to 60 minutes. Or, prokanamide at 15

00:24:14.750 --> 00:24:17.670
millijoy kilograms infused over 30 to 60 minutes.

00:24:18.529 --> 00:24:21.690
The key word here is slow. Why slow? Because

00:24:21.690 --> 00:24:24.170
these drugs can induce pretty severe hypotension

00:24:24.170 --> 00:24:26.109
and just make the situation worse if you push

00:24:26.109 --> 00:24:28.789
them too fast. What about the absolute caution

00:24:28.789 --> 00:24:31.549
regarding combining these two powerful drugs?

00:24:31.690 --> 00:24:34.230
This is a huge safety concern. Do not routinely

00:24:34.230 --> 00:24:36.650
administer amyterone and procainamide together.

00:24:36.829 --> 00:24:39.349
Both are strong sodium and potassium channel

00:24:39.349 --> 00:24:41.650
blockers, and they both prolong the QT interval.

00:24:41.809 --> 00:24:43.809
So you get an additive effect. A very dangerous

00:24:43.809 --> 00:24:46.170
additive effect. You significantly increase the

00:24:46.170 --> 00:24:48.269
chance of inducing a lethal rhythm, specifically

00:24:48.269 --> 00:24:51.250
polymorphic VT or torsades de pointes. If one

00:24:51.250 --> 00:24:52.950
fails, you can switch to the other, but you never

00:24:52.950 --> 00:24:55.109
combine them. And what about using adenosine

00:24:55.109 --> 00:24:57.789
in a wide complex tachycardia? I know a lot of

00:24:57.789 --> 00:24:59.829
providers get tripped up there. It's high risk.

00:25:00.009 --> 00:25:02.769
I mean, if the wide complex rhythm is truly VT,

00:25:03.049 --> 00:25:04.890
adenosine won't terminate it, and it might even

00:25:04.890 --> 00:25:07.470
accelerate it, which is dangerous. The only time

00:25:07.470 --> 00:25:09.769
you might even consider it is if you have really

00:25:09.769 --> 00:25:12.690
strong evidence that the rhythm is actually SVT

00:25:12.690 --> 00:25:15.089
with aberrancy. Meaning the patient has a pre

00:25:15.089 --> 00:25:17.690
-existing bundle branch block or something? Exactly.

00:25:17.829 --> 00:25:20.809
But honestly, if you're not sure, you treat the

00:25:20.809 --> 00:25:24.930
wide QRS as VT, you use the designated antiarrhythmics,

00:25:25.069 --> 00:25:28.039
or you prepare for cardioversion. And this is

00:25:28.039 --> 00:25:30.819
that critical transition. When the stable tachycardia

00:25:30.819 --> 00:25:33.480
patient suddenly becomes unstable, poor perfusion,

00:25:33.720 --> 00:25:36.559
hypotension, signs of shock, we skip all the

00:25:36.559 --> 00:25:38.539
drug trials. We move straight to electricity.

00:25:39.119 --> 00:25:41.559
Time is myocardium. That's right. When we hit

00:25:41.559 --> 00:25:44.000
cardiopulmonary compromise, we go straight to

00:25:44.000 --> 00:25:46.539
synchronized cardioversion. This is the definitive

00:25:46.539 --> 00:25:49.519
intervention for hemodynamically unstable SVT,

00:25:49.740 --> 00:25:52.539
atrial flutter, or VT with a pulse. It takes

00:25:52.539 --> 00:25:55.720
seconds, not minutes, to deliver. Now, cardioversion

00:25:55.720 --> 00:25:58.000
is a painful procedure, so how do we balance

00:25:58.000 --> 00:26:00.339
the need for sedation and analgesia with the

00:26:00.339 --> 00:26:02.680
urgency of delivering that shock? That is the

00:26:02.680 --> 00:26:05.519
art of critical care, right there. If the patient

00:26:05.519 --> 00:26:08.480
is unstable and has a depressed level of consciousness,

00:26:08.920 --> 00:26:11.039
you skip sedation and you shock immediately.

00:26:11.200 --> 00:26:13.299
And if they're awake? If the child is unstable

00:26:13.299 --> 00:26:15.880
but awake, you should quickly deliver a rapid

00:26:15.880 --> 00:26:18.579
sequence, analgesic or sedative, something like

00:26:18.579 --> 00:26:22.339
ketamine or midozolam. if it's available, but

00:26:22.339 --> 00:26:25.380
the pay -alist instruction is firm. You do not

00:26:25.380 --> 00:26:27.640
delay the shock waiting for the sedation to kick

00:26:27.640 --> 00:26:30.359
in or for the perfect airway setup if the patient

00:26:30.359 --> 00:26:33.319
is crashing. A brief moment of pain is worth

00:26:33.319 --> 00:26:36.039
avoiding a full -blown cardiac arrest. Absolutely.

00:26:36.099 --> 00:26:37.920
Let's focus on the energy dose. It's lower than

00:26:37.920 --> 00:26:40.519
for V -fib defibrillation. Right. Cardiaversion

00:26:40.519 --> 00:26:42.599
requires less energy because we're just trying

00:26:42.599 --> 00:26:45.019
to reset the electrical rhythm, not completely

00:26:45.019 --> 00:26:48.119
depolarize the entire heart. The initial dose

00:26:48.119 --> 00:26:51.400
is 0 .5 to 1 joule. per kilogram. And if that

00:26:51.400 --> 00:26:53.740
doesn't work? If that shock is ineffective, you

00:26:53.740 --> 00:26:55.599
have to increase the dose for the next attempt.

00:26:56.099 --> 00:26:58.000
You escalate to the second dose of two joules

00:26:58.000 --> 00:27:00.400
per kilogram. Always use the lowest effective

00:27:00.400 --> 00:27:02.880
dose. Okay, now for the critical nursing steps

00:27:02.880 --> 00:27:05.759
for synchronized cardioversion. What are the

00:27:05.759 --> 00:27:08.299
common pitfalls you see that jeopardize the shock?

00:27:08.740 --> 00:27:11.700
Pitfall number one. And it's a big one, not selecting

00:27:11.700 --> 00:27:14.299
synchronization. You have to turn on the defibrillator,

00:27:14.539 --> 00:27:16.839
select your pads, and most importantly, select

00:27:16.839 --> 00:27:18.779
the synchronize mode. And you have to see the

00:27:18.779 --> 00:27:20.779
sync marks. You must look for the sync marks

00:27:20.779 --> 00:27:23.680
to appear above the R waves on the monitor. If

00:27:23.680 --> 00:27:26.099
you can't see them, increase the gain or switch

00:27:26.099 --> 00:27:28.680
leads until you can. And if you can't see them

00:27:28.680 --> 00:27:31.519
and you shock anyway, what's the risk? The machine

00:27:31.519 --> 00:27:34.180
will deliver an unsynchronized shock. That means

00:27:34.180 --> 00:27:36.700
the energy could land exactly on that vulnerable

00:27:36.700 --> 00:27:40.279
period of the T wave. R on T phenomenon. Exactly.

00:27:40.660 --> 00:27:43.539
Hitting the T wave can induce ventricular fibrillation,

00:27:43.819 --> 00:27:46.619
turning a salvageable rhythm into a full cardiac

00:27:46.619 --> 00:27:49.599
arrest. Confirming synchronization is a mandatory

00:27:49.599 --> 00:27:52.640
safety check. OK, sync is confirmed. We charge

00:27:52.640 --> 00:27:55.259
and deliver. You announce charging defibrillator,

00:27:55.480 --> 00:27:58.160
make sure everyone is clear, and crucially, you

00:27:58.160 --> 00:28:00.299
have to hold the shock button down until it delivers.

00:28:00.960 --> 00:28:03.180
The machine is waiting for the next R wave. If

00:28:03.180 --> 00:28:05.500
you press and release too quickly, it won't fire.

00:28:05.619 --> 00:28:08.259
And then immediately after. Immediately after,

00:28:08.460 --> 00:28:11.980
you check the monitor. If VF develops a total

00:28:11.980 --> 00:28:14.480
catastrophe, you must immediately turn off the

00:28:14.480 --> 00:28:17.359
synchronization mode and proceed to unsynchronized

00:28:17.359 --> 00:28:20.640
defibrillation and CPR. And remember, the defibrillator

00:28:20.640 --> 00:28:22.859
defaults back to unsynchronized mode after every

00:28:22.859 --> 00:28:25.660
shock. You have to reselect synchronization for

00:28:25.660 --> 00:28:27.900
every subsequent cardioversion attempt. Okay,

00:28:27.960 --> 00:28:29.839
let's dedicate this section to the high -level

00:28:29.839 --> 00:28:32.079
nursing implications of our PANS drug arsenal.

00:28:32.420 --> 00:28:34.619
We've covered the doses. Now let's focus on the

00:28:34.619 --> 00:28:36.799
how and the what to watch for. The titration,

00:28:36.980 --> 00:28:38.960
the side effects, the complex stuff. Great idea.

00:28:39.319 --> 00:28:42.119
Let's start with adenosine for SVT. So we know

00:28:42.119 --> 00:28:44.400
adenosine blocks conduction through the AV node

00:28:44.400 --> 00:28:47.539
and causes that transient assistal. The nursing

00:28:47.539 --> 00:28:49.619
management is all centered around its rapid decay.

00:28:50.099 --> 00:28:52.319
That half -life is so short, you need to use

00:28:52.319 --> 00:28:54.940
a three -way stopcock or a proximal port. Pushing

00:28:54.940 --> 00:28:57.839
the drug, spinning the stopcock, flushing immediately.

00:28:58.140 --> 00:29:00.240
Rapidly, yes. What should the nurse warn the

00:29:00.240 --> 00:29:02.220
patient or the family about, especially if the

00:29:02.220 --> 00:29:04.519
child is conscious during this? The child will

00:29:04.519 --> 00:29:07.259
likely feel a sensation of profound momentary

00:29:07.259 --> 00:29:10.079
chest pressure or dizziness as their heart stops.

00:29:10.460 --> 00:29:12.859
You have to warn them about this brief cardiac

00:29:12.859 --> 00:29:15.160
standstill which lasts maybe 10 to 15 seconds.

00:29:15.849 --> 00:29:17.869
explain that it's necessary to reset the rhythm.

00:29:18.670 --> 00:29:20.950
That anxiety management is key. And you're glued

00:29:20.950 --> 00:29:23.710
to the ECG monitor. Continuous ECG monitoring

00:29:23.710 --> 00:29:27.089
is essential. If the SVT converts, your immediate

00:29:27.089 --> 00:29:29.829
priority is monitoring for recurrence. Okay,

00:29:29.950 --> 00:29:33.509
Omiodurone for a wide QRS. It's a complex drug.

00:29:34.250 --> 00:29:36.650
For the nurse, what are the primary concerns

00:29:36.650 --> 00:29:39.250
when you're infusing this for a perfusing VT?

00:29:39.990 --> 00:29:42.049
Amidurone affects pretty much everything, sodium,

00:29:42.190 --> 00:29:44.890
potassium, calcium channels, and it has anti

00:29:44.890 --> 00:29:47.230
-adrenergic properties. So our immediate concerns

00:29:47.230 --> 00:29:50.269
are twofold. First, hypotension. Okay. Rapid

00:29:50.269 --> 00:29:53.109
administration, even over 20 to 60 minutes, can

00:29:53.109 --> 00:29:55.589
cause significant peripheral vasodilation and

00:29:55.589 --> 00:29:57.450
decreased contractility. You have to monitor

00:29:57.450 --> 00:29:59.369
their blood pressure every five minutes during

00:29:59.369 --> 00:30:01.869
that infusion. And the second major concern related

00:30:01.869 --> 00:30:04.309
to its mechanism. The second concern is related

00:30:04.309 --> 00:30:07.710
to its effect on potassium channels, QT prolongation.

00:30:08.700 --> 00:30:11.279
Amiodarone increases the risk of dangerous arrhythmias,

00:30:11.640 --> 00:30:14.140
particularly torsades to point. The nurse has

00:30:14.140 --> 00:30:16.519
to ensure the patient isn't also hypokalemic

00:30:16.519 --> 00:30:18.920
or hypomagnesemic, because those electrolyte

00:30:18.920 --> 00:30:20.819
issues just exacerbate the risk of torsades.

00:30:20.960 --> 00:30:24.000
Right, and the dose is 5 mg of a kilogram IVIO

00:30:24.000 --> 00:30:27.079
infused slowly. Very slowly. The slower the better

00:30:27.079 --> 00:30:29.480
if they're perfusing. And remember, the maximum

00:30:29.480 --> 00:30:33.839
cumulative dose is 15 mg per day. Okay, prokainamide.

00:30:34.380 --> 00:30:37.859
Our other strong option for wide QRS, how does

00:30:37.859 --> 00:30:40.720
its mechanism, mainly sodium channel blockade,

00:30:40.880 --> 00:30:42.980
translate to different nursing management concerns

00:30:42.980 --> 00:30:45.240
compared to amiodarone? Procatamide primarily

00:30:45.240 --> 00:30:47.400
blocks a sodium channel, so it slows conduction

00:30:47.400 --> 00:30:50.079
velocity and it prolongs the QRS duration. It's

00:30:50.079 --> 00:30:52.240
highly effective, but it comes with significant

00:30:52.240 --> 00:30:54.940
risks. The main nursing focus is just preventing

00:30:54.940 --> 00:30:57.920
toxicity by infusing it slowly. So you're infusing

00:30:57.920 --> 00:31:01.099
15 milligrams over 30 to 60 minutes. Right. What

00:31:01.099 --> 00:31:03.279
are the key signs of procatamide toxicity that

00:31:03.279 --> 00:31:05.019
would make you stop the infusion immediately.

00:31:05.420 --> 00:31:07.720
The three big ones are hypotension just like

00:31:07.720 --> 00:31:11.640
amiodarone and then excessive QRS or QT interval

00:31:11.640 --> 00:31:15.640
prolongation. If the QRS complex widens by 50

00:31:15.640 --> 00:31:18.890
% or more from baseline Or if it widens to greater

00:31:18.890 --> 00:31:22.269
than 0 .12 seconds, you stop the infusion immediately.

00:31:22.490 --> 00:31:24.009
Because that's a sign of dangerous conduction

00:31:24.009 --> 00:31:26.150
depression. Exactly. And again, the combination

00:31:26.150 --> 00:31:28.730
of amiodarone and brocanamide is strictly avoided

00:31:28.730 --> 00:31:31.210
because of that additive QT risk. Okay, let's

00:31:31.210 --> 00:31:34.089
touch on magnesium and sodium bicarb. These have

00:31:34.089 --> 00:31:36.690
really specific roles, often tied to the H's

00:31:36.690 --> 00:31:39.509
and T's. Right. Magnesium sulfate is specifically

00:31:39.509 --> 00:31:42.630
indicated for two situations, torsades de pointe,

00:31:42.789 --> 00:31:45.890
that polymorphic VT, and severe refractory asthma.

00:31:46.109 --> 00:31:49.130
It acts as a physiologic calcium channel blocker,

00:31:49.210 --> 00:31:51.069
and it's crucial for stabilizing the membrane

00:31:51.069 --> 00:31:54.009
when torsides is present. And sodium bicarb.

00:31:54.390 --> 00:31:56.769
Sodium bicarbonate is used to treat severe documented

00:31:56.769 --> 00:31:59.750
metabolic acidosis or specific drug overdoses,

00:32:00.069 --> 00:32:02.210
like presciclic antidepressants, where it helps

00:32:02.210 --> 00:32:04.549
narrow the QRS and counter that tartar toxicity.

00:32:05.029 --> 00:32:07.069
But remember, it's not a generalized drug. Maximize

00:32:07.069 --> 00:32:09.630
ventilation first. Finally, let's address the

00:32:09.630 --> 00:32:12.009
drug that must be avoided in a specific pediatric

00:32:12.009 --> 00:32:15.230
population and explain the critical physiological

00:32:15.230 --> 00:32:19.440
why. Varepamil. It's a calcium channel blocker,

00:32:19.700 --> 00:32:21.960
and it should not be used routinely to treat

00:32:21.960 --> 00:32:25.579
SVT in infants. This is a Palau certification

00:32:25.579 --> 00:32:27.859
deal -breaker question. And the physiological

00:32:27.859 --> 00:32:31.539
reason is profound. It is. Infants, especially

00:32:31.539 --> 00:32:34.220
those under one year, are highly rate dependent,

00:32:34.579 --> 00:32:37.920
and they rely heavily on adequate cardiac contractility.

00:32:38.240 --> 00:32:41.210
They're anotropy. Calcium channel blockers kill

00:32:41.210 --> 00:32:43.769
contractility. So when you combine that severe

00:32:43.769 --> 00:32:46.690
decrease in squeeze with a rapid SDT -induced

00:32:46.690 --> 00:32:49.970
rate, you cause immediate refractory hypotension

00:32:49.970 --> 00:32:52.430
and cardiac collapse. It's just too risky. Far

00:32:52.430 --> 00:32:54.529
too risky in that age group. In older children,

00:32:54.730 --> 00:32:56.930
if it's used for stable SVT that's resistant

00:32:56.930 --> 00:32:59.849
to adenosine, it requires an immediate cardiologist

00:32:59.849 --> 00:33:01.789
consult. Okay, this is where we bring the textbook

00:33:01.789 --> 00:33:04.069
back to the bedside. We've covered the mechanics,

00:33:04.190 --> 00:33:06.650
but algorithms fail when humans make high pressure

00:33:06.650 --> 00:33:09.250
errors. Let's talk about the common traps and

00:33:09.250 --> 00:33:11.750
pitfalls we see in real -life codes. The algorithms

00:33:11.750 --> 00:33:14.150
are clear, but clinical pressure definitely warps

00:33:14.150 --> 00:33:16.470
decision -making. Let's start with the bradycardia

00:33:16.470 --> 00:33:18.990
trap. The natural instinct is to grab the epinephrine

00:33:18.990 --> 00:33:21.250
when you see that heart rate dropping. And that's

00:33:21.250 --> 00:33:23.529
the trap. You see a low number on the monitor

00:33:23.529 --> 00:33:25.910
and you react to the monitor. But you have to

00:33:25.910 --> 00:33:29.299
remember Pediatric bradycardia is almost always

00:33:29.299 --> 00:33:31.579
a sign of a ventilation or oxygenation failure

00:33:31.579 --> 00:33:33.740
first. So don't jump to epinephrine. Don't jump

00:33:33.740 --> 00:33:36.440
to epinephrine if you haven't maximized oxygenation

00:33:36.440 --> 00:33:38.839
and ventilation. I remember a case where the

00:33:38.839 --> 00:33:41.180
teen treated the low heart rate first, forgetting

00:33:41.180 --> 00:33:43.839
the patient was profoundly hypoxemic from an

00:33:43.839 --> 00:33:46.400
unsecured airway. The pump was pressed, but the

00:33:46.400 --> 00:33:49.220
gas tank was empty. Exactly. Always maximize

00:33:49.220 --> 00:33:52.920
your ABCs first, then compress or medicate. Then

00:33:52.920 --> 00:33:55.160
there's the more subtle but equally dangerous

00:33:55.160 --> 00:33:59.440
tachycardia trap. Mistaking ST for SVT. This

00:33:59.440 --> 00:34:01.400
is where treating the patient, not the monitor,

00:34:01.640 --> 00:34:04.660
is absolutely paramount. Sinus tachycardia is

00:34:04.660 --> 00:34:07.519
a sign of an underlying systemic illness sepsis,

00:34:07.720 --> 00:34:10.699
hemorrhage, severe dehydration. And if you mistakenly

00:34:10.699 --> 00:34:12.900
treat that with adenosine? You interrupt the

00:34:12.900 --> 00:34:15.199
child's attempt to maintain cardiac output via

00:34:15.199 --> 00:34:17.800
rate. You've taken away their only coping mechanism.

00:34:18.219 --> 00:34:20.280
You have to treat the underlying cause -give

00:34:20.280 --> 00:34:23.199
fluids for dehydration, blood products for hemorrhage.

00:34:23.440 --> 00:34:26.480
A continuous ECG should show that heart rate

00:34:26.480 --> 00:34:29.340
coming down as the underlying issue is resolved.

00:34:29.699 --> 00:34:32.239
And the cardinal sin during electrical intervention,

00:34:33.199 --> 00:34:35.840
the cardioversion mistake. Forgetting to hit

00:34:35.840 --> 00:34:38.460
the sync button. It seems so obvious when we're

00:34:38.460 --> 00:34:40.239
sitting here talking about it, but in the chaos

00:34:40.239 --> 00:34:43.639
of a code, providers forget. And the result is

00:34:43.639 --> 00:34:46.900
a potentially harmful unsynchronized shock. Right.

00:34:47.079 --> 00:34:50.230
Risking induced V -fib. Always confirm those

00:34:50.230 --> 00:34:52.150
sync marks are visible before you charge and

00:34:52.150 --> 00:34:54.110
deliver, and remember to check it again after

00:34:54.110 --> 00:34:56.090
the first shock, as the machine always reverts

00:34:56.090 --> 00:34:58.750
back to unsynchronized mode. As the ER nurse,

00:34:59.530 --> 00:35:01.969
documentation is essential for quality improvement

00:35:01.969 --> 00:35:04.719
for legal defense. What are the absolute essentials

00:35:04.719 --> 00:35:06.699
that have to be captured during a high stakes,

00:35:07.000 --> 00:35:09.480
tacky, or bratty event? We need objective evidence

00:35:09.480 --> 00:35:12.139
of the rhythm and the interventions effect. So

00:35:12.139 --> 00:35:14.800
that means an ECG strip before and after every

00:35:14.800 --> 00:35:17.440
single intervention. Bagel maneuver, every drug,

00:35:17.679 --> 00:35:20.610
every shock. Every single one. For adenosine,

00:35:20.829 --> 00:35:23.130
you have to record the exact timing and dosage

00:35:23.130 --> 00:35:26.150
and the precise size of that flush, like six

00:35:26.150 --> 00:35:28.150
milligram adenosine followed immediately by 10

00:35:28.150 --> 00:35:30.510
milliliter LNS flush. For electrical therapy,

00:35:30.889 --> 00:35:33.289
the exact energy dose for every single attempt

00:35:33.289 --> 00:35:35.730
is mandatory. So what's the final thought we

00:35:35.730 --> 00:35:37.789
leave the ER nurse with as they're walking into

00:35:37.789 --> 00:35:40.320
that critical situation? When you're managing

00:35:40.320 --> 00:35:43.260
pediatric arrhythmias, you have to master the

00:35:43.260 --> 00:35:46.079
distinction between adequate and poor perfusion.

00:35:46.539 --> 00:35:48.900
That single assessment determines your timeline.

00:35:49.079 --> 00:35:51.000
Poor perfusion means you have seconds. You have

00:35:51.000 --> 00:35:53.400
30 seconds to deliver a life -saving shock or

00:35:53.400 --> 00:35:56.019
start compressions. If perfusion is adequate,

00:35:56.300 --> 00:35:58.360
you have little time for precise diagnosis, for

00:35:58.360 --> 00:36:01.239
an expert consult, for non -electrical interventions.

00:36:01.820 --> 00:36:04.599
Use that QRS split wide versus narrow to narrow

00:36:04.599 --> 00:36:07.099
your drug choice. And never forget that pediatric

00:36:07.099 --> 00:36:10.289
bradycardia is most often airway problem disguised

00:36:10.289 --> 00:36:12.869
as a heart problem. We covered a tremendous amount

00:36:12.869 --> 00:36:15.269
of ground today. The critical signs of instability,

00:36:15.610 --> 00:36:18.570
respiratory failure, shock with or without hypotension,

00:36:18.690 --> 00:36:21.369
and altered mental status. We emphasize that

00:36:21.369 --> 00:36:24.269
in bradycardia, the ABCs come before drugs, and

00:36:24.269 --> 00:36:26.769
that a persistent bradycardia below 60 means

00:36:26.769 --> 00:36:30.530
immediate, high -quality CPR. Yes. We broke down

00:36:30.530 --> 00:36:33.210
the critical QRS split, the high -stakes technique

00:36:33.210 --> 00:36:36.070
of synchronized cardioversion, and detailed the

00:36:36.070 --> 00:36:39.050
crucial nursing implications for amiodarone and

00:36:39.050 --> 00:36:41.110
procainamide. And if you take one lesson away

00:36:41.110 --> 00:36:44.489
from this... Let it be this. Always remember

00:36:44.489 --> 00:36:47.469
the reversible causes of bradycardia. Often the

00:36:47.469 --> 00:36:49.989
best cardiac care you can provide is swift and

00:36:49.989 --> 00:36:52.190
effective pulmonary care. Good ventilation and

00:36:52.190 --> 00:36:55.309
oxygenation. Exactly. Never forget that the child's

00:36:55.309 --> 00:36:57.809
slow heart rate is usually their body's final

00:36:57.809 --> 00:37:01.010
desperate attempt to survive hypoxia. The solution

00:37:01.010 --> 00:37:03.849
is often right there in your hands managing the

00:37:03.849 --> 00:37:06.269
airway and breathing long before you ever reach

00:37:06.269 --> 00:37:08.570
for the epinephrine. An excellent point to end

00:37:08.570 --> 00:37:10.469
on. Thank you for going on this deep dive with

00:37:10.469 --> 00:37:12.449
us, focusing on the details that truly matter

00:37:12.449 --> 00:37:15.329
when facing critical pediatric arrhythmias. Stay

00:37:15.329 --> 00:37:15.989
sharp out there.
