WEBVTT

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Welcome to Deep Dive Ortho. Today, we're getting

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to grips with a really common yet, well, often

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quite frustrating wrist injury, the trichetral

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fracture. That's right. It might actually surprise

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you just how prevalent these are. We're talking

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the second most common carpal bone fracture.

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Indeed. About 15 % to 18 % of all carpal fractures,

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give or take. Which is significant. But despite

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that, they're Let's face it, notoriously tricky

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to diagnose, often just labeled a wrist sprain.

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Exactly. Or if they're simply radiographically

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occult, just don't show up on the initial x -rays.

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So our aim today is to really give you a deep

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understanding of these elusive injuries. We want

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to look at the subtle diagnostic clues, the,

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well, the surprisingly complex ways they happen.

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Mechanisms, yes. Yeah. Which have evolved quite

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a bit. And the latest thinking on management,

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both conservative and surgical. We've got our

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expert here to guide us through all this. Thank

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you. It's great to be here. We're talking about

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a small bone, the trichobrum, but one that plays

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a really pivotal role in how the wrist works.

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Can you just remind us where it sits anatomically?

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Of course. So if you picture the wrist, the trichotrum

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is this sort of wedge -shaped bone. It's in the

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proximal carpal row over on the ulnar side, the

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little finger side. OK. And its position is key.

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It connects with several neighbors. The lunid

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bone next to it, the pisiform sits right on top.

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and the hamate is distal to it. Right. And it's

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connection to the ulna. Ah, well that's interesting.

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It doesn't directly touch the ulna. Instead,

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it connects via the TFCC, the triangular fibrocartilage

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complex. Disc. Precisely. Which acts as a crucial

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shock absorber and stabilizer for that medial

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side of the wrist. And another really important

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point, its dorsum medial surface, the back, has

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these two significant areas where ligaments attach.

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strong ligaments. Ah okay so not just bone -on

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-bone articulation but critical ligament anchor

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points too. Exactly and that's why when the tricotrum

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gets injured it's well it's really just the bone

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itself the surrounding structures especially

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those ligaments are often involved. It sort of

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sets the scene for why these injuries can be

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complex. That's a perfect setup. It immediately

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tells us, we're not just dealing with a simple

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chip. So let's dive into that complexity. How

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do these fractures actually happen? There's been

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quite a debate over the years, hasn't there?

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Oh, absolutely. A real sort of historical back

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and forth. It mainly centered around two competing

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ideas, ligament avulsion versus impaction. Pull

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versus push, essentially. You could put it like

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that, yes. The traditional views were fairly

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distinct. First, you had the ligament avulsion

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theory. Right. This suggested that these fractures

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particularly the common dorsal ones, happened

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when a ligament attaching to the trichotrum was

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pulled so hard, perhaps during a fall with the

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wrist bent sharply forward and inwards, palmar

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flexion and radial deviation, that the ligament

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just pulled a fragment of bone off with it. Simple

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tensile failure at the insertion site. Okay,

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so a pulling mechanism. What was the alternative?

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The alternative was the endocarpal impaction

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theory. Garcia Elias, a well -known hand surgeon,

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really detailed this. He proposed a chisel action.

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Yes, the idea was that the ulnar styloid process,

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that bony bump on the end of your ulna, acted

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like a chisel. During forceful dorsiflexion,

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bending the wrist back hard combined with ulnar

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deviation towards the little finger. That classic

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fall mechanism. Exactly. The styloid would supposedly

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strike the dorsal aspect of the trichatrum and

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chip a piece off. And this theory gained some

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traction because some studies noted a slightly

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longer ulnar styloid in these patients. They

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measured the ulnar styloid process index, the

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USPI. Right, I remember reading about that. I

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mean USPI of about .29 was reported. That's the

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figure often cited, suggesting a longer styloid

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might predispose to this impaction. Then later,

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Hucker and Menchik sort of expanded on this impaction

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idea. How so? They suggested it wasn't maybe

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just the ulnar styloid. They argued that the

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edge of the hamat bone, the dorsoproximal edge,

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could also act as a chisel against the trichatrum

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in that same extended ulnar deviated position.

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OK, so a similar impact mechanism, but potentially

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from a different bony structure. So historically

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it was pull versus impact, either from the ulna

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or the hermit. That was the basic picture, yes.

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But as often happens in medicine, things aren't

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usually that simple, are they? What have newer

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techniques, especially MRI, shown us? Has it

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clarified things or, dare I say, made it even

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muddier? Huh. Well, complex might be a better

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word than muddy. But yes, you're absolutely right.

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Modern MRI studies, like the one by Bess and

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colleagues, have really peeled back the layers

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here. OK. And their findings don't just tweak

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the old theories. They, well, they challenge

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some of the fundamental assumptions and show

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it's often a much more mixed, multifactorial

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picture. How so? Let's take the ulnar styloid

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impaction theory first. What did the MRI data

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show there? Well, firstly, the average USPI they

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found in their patients was 0 .21. Which is normal.

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Exactly. Within the normal range. Yeah. Not consistently

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high like the theory predicted. But here's the

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really interesting bit. The median USPI was actually

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higher in patients whose injury story sounded

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more like an avulsion 0 .27. Higher in the avulsion

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group. Yes. Compared to those whose story suggested

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impaction. where it was .20. So that directly

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contradicts the simple idea that a long styloid

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equals impaction fracture. It's clearly not that

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straightforward. Okay, that's definitely counterintuitive.

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What else did the MRI reveal about the ulnar

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styloid's role? This is perhaps the most damning

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evidence against the direct ulnar styloid chisel

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idea. at least in the cases they studied. They

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found no fracture or even bone marrow edema bone

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bruising in the ulnar styloid process itself

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in any of their patients with trichotral fractures.

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None at all. None. And you'd really expect to

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see some sign of trauma on the styloid if it

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was directly hitting the trichotrum hard enough

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to break it. That seems pretty conclusive for

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those cases, doesn't it? It makes direct impaction

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from the ulna seem unlikely as the sole cause.

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It certainly makes it much less likely in those

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instances, yes. However, The heme impaction theory

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did get some support. Oh, OK. How? They did find

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bone marodema in the hamat bone in nearly a quarter

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of the patients, 23 .8%, which does suggest that

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the hemo hitting the trichotrum, as Hucker and

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Menchik proposed, could well be a contributing

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factor in that extended ulnar deviated position.

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So maybe the heme is the more common impactor

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than the ulna. Okay, so the picture is shifting.

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Less ulna, maybe more heme involvement in impaction.

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What else complicated the simple avulsion versus

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impaction story? Well, they looked at specific

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fracture patterns using the Gersi -Alias classification

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we'll discuss later. He'd suggested type III

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fractures partially displaced at the distal end

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were likely evulsions. And Besse's group did

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find a lower mean USPI for these, .17, which

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sort of fits. But crucially, they found this

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type III pattern was significantly more common

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in the group whose history suggested impaction.

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Oh, right. So the pattern associated with a lower

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USPI thought to be evulsion was actually more

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common in the impaction group. Precisely. Yeah.

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doesn't fit neatly into either box. It suggests

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a real overlap, maybe a combination of forces

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rather than one single mechanism defining the

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fracture type. Okay, it sounds truly multifactorial.

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And what about the ligaments in all this? Did

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they find differences between the evulsion and

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impaction groups there? This is probably the

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most critical finding. Clinically speaking, they

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found no significant difference in how often

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associated ligaments were torn between the supposed

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avulsion and impaction groups. Really? So regardless

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of whether it looked more like a pull or a push

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fracture? The surrounding ligaments were just

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as likely to be injured. It strongly points towards

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a complex combined injury mechanism where the

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soft tissues nearly always take a hit, whatever

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the primary force on the bone appears to be.

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Wow. So the key takeaway is forget simple either.

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It's a complex interplay and assume ligaments

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are involved. That sums it up perfectly. It's

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a dynamic event affecting the whole complex.

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So if the mechanism is this complex, does that

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translate into different types of tricatural

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fractures? And more importantly, given what we've

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just heard about ligaments, what associated injuries

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should we really be looking for? Absolutely.

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Yeah. Understanding the different fracture types

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is key for management. For the common dorsal

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cortical fractures, the Garcia -Elias system

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is useful. It goes from type 1 Undisplaced. Just

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a crack. Essentially, yes. Types 2 and 3 are

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partially displaced, either proximally or distally.

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Type 4 is completely displaced. Type 5 means

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multiple fragments, more committed. And type

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6 is a more significant vertical frontal fracture

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involving the whole dorsal surface. Right. But

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beyond those dorsal ones, what are the main anatomical

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types? Broadly, we think of three main types.

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First, the dorsal cortical fractures we've been

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discussing, they make up the vast majority, 93

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to 95%. And also called benign. Historically,

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yes, but maybe that's misleading given the ligament

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issue. They typically happen from falls onto

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an extended ulnar deviated wrist, the impaction

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scenario. But they can also result from avulsion

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of the dorsal radiotricotrol or scaphotricotrol

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ligaments, particularly in wrist flexion with

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radial deviation. So same fracture appearance.

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Potentially different initiating force. Understood.

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What's the second type? Second are trichotral

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body fractures. These are less common, but generally

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indicate higher energy trauma. More serious,

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then? Definitely. Because they involve the main

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structural part of the bone. You often see these

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alongside really significant wrist injuries,

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like peri -lunar fracture dislocations. Ah, right.

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Part of a bigger picture. Exactly. A trichotral

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body fracture is found in something like 12 to

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25 percent of peri -lunar injuries. It's a red

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flag for major carpal disruption. The mechanism

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is usually direct compression, axial loading,

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or crush injuries. OK. And because they're central

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in high energy, they're often associated with

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a whole host of other problems. Lunar trichotrial

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ligament tears, scaphoid fractures, distal radius

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fractures. You have to look carefully for these

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associated injuries. Missing them is bad news.

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Absolutely. And the third type. The least common,

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but potentially the most problematic for stability

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are volar cortical fractures. On the palm side?

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Yes. Usually caused by avulsion of the volar

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ligaments, like the ulnotrichotrol or lunotrichotrol

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ligament. Why are they more problematic? Because

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those volar ligaments are absolutely critical

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for maintaining the alignments of the carpal

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bones. Injuring them carries a much higher risk

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of leading to carpal instability, which can cause

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long -term pain and dysfunction if it's not picked

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up and managed correctly. They need a high index

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of suspicion. So it really hammers home that

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point about associated injuries. It's not just

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the bone. And that best study you mentioned really

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quantified this ligament issue, didn't it? It

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really did. And the figures are, well, quite

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sobering. They found that an incredible 95 .2

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% of patients with dorsal trichatrial fractures

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had at least one associated dorsal carpal ligament

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injury. 95%. 20 out of 21 patients. It essentially

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means if you diagnose a dorsal trichatral fracture,

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you should assume there's significant ligament

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damage until proven otherwise. That completely

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changes the perspective from a simple fracture

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or sprain? It absolutely should. The specific

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breakdown is also revealing. Dorsal ulnortrichatral

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ligament tears were seen in 81%. Dorsal intercarpal

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ligament tears in 76%. And dorsal radiocarpal

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ligament tears in about 67%. These ligaments

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are almost routinely injured alongside the bone.

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Is there any correlation between where the bone

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bruising is on MRI and which ligament is torn?

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Interestingly, no. They looked for that but found

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no statistically significant link. Where the

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bone marrow oedema was located didn't reliably

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predict which specific ligament was injured.

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So you can't rely on the bone oedema pattern

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to guide you on the ligaments? Not reliably,

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no. The implication is clear. High suspicion

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of ligament injury is warranted in nearly all

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cases, irrespective of the precise edema pattern

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on the MRI. Okay, that 95 % figure is the one

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I'll take away. Assume ligament injury. Now,

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if we need to assess these ligaments, especially

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with MRI, how well can we actually see them?

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Are there imaging pitfalls we need to be aware

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of? That's a very practical question. Generally,

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3D gadolinium enhanced MRI using sequences like

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VBE is pretty good. The best study identified

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over 92 % of these ligaments in healthy controls.

00:12:21.710 --> 00:12:24.279
So MRI is the tool. It's definitely our best

00:12:24.279 --> 00:12:27.799
tool for soft tissues, yes. But, and it's important,

00:12:28.220 --> 00:12:30.159
but visibility isn't perfect for all the ligaments.

00:12:31.139 --> 00:12:34.000
Which ones are tricky? Well, the dorsal intercarpal

00:12:34.000 --> 00:12:36.340
ligaments showed up very well. completely visible

00:12:36.340 --> 00:12:39.740
in almost 97 % of cases. The dorsal radiocarpal

00:12:39.740 --> 00:12:42.899
was also good, 80 % complete visibility. But

00:12:42.899 --> 00:12:45.259
the dorsal ulno -tree control ligament was much

00:12:45.259 --> 00:12:47.460
more challenging. It was only completely visible

00:12:47.460 --> 00:12:51.500
in about 53 % of cases. And worryingly, it wasn't

00:12:51.500 --> 00:12:55.440
seen at all in nearly a quarter. 23 .3 % of the

00:12:55.440 --> 00:12:57.600
healthy controls. Not seen at all, even when

00:12:57.600 --> 00:13:00.159
presumably intact. Exactly. So if you don't see

00:13:00.159 --> 00:13:02.519
it clearly on the scan, you can't automatically

00:13:02.519 --> 00:13:05.220
assume it's torn or absent. There might be technical

00:13:05.220 --> 00:13:08.139
limitations. Why is that one harder to see? It's

00:13:08.139 --> 00:13:10.279
likely due, at least in part, to something called

00:13:10.279 --> 00:13:12.639
the magic angle artifact. Right, I've heard of

00:13:12.639 --> 00:13:15.139
that. Because ligaments are ordered collagen

00:13:15.139 --> 00:13:18.629
fibers, If they lie at roughly 55 degrees to

00:13:18.629 --> 00:13:21.730
the main magnetic field of the MRI scanner, they

00:13:21.730 --> 00:13:23.950
can produce an artificially high signal. Making

00:13:23.950 --> 00:13:26.309
it look like a tear. Precisely. Or just making

00:13:26.309 --> 00:13:29.149
it blurry. The dorsal ulnitricular ligament has

00:13:29.149 --> 00:13:31.970
a curved path, and combined with the short echo

00:13:31.970 --> 00:13:35.570
time, TE, used in some sequences, it's particularly

00:13:35.570 --> 00:13:38.769
susceptible. So you have to interpret findings

00:13:38.769 --> 00:13:41.269
relating to that specific ligament with a degree

00:13:41.269 --> 00:13:44.389
of caution. Reported tear rates might be slightly

00:13:44.389 --> 00:13:46.889
inflated due to this artifact. That's a really

00:13:46.889 --> 00:13:50.389
crucial point for interpreting MRI reports. Combine

00:13:50.389 --> 00:13:52.549
the imaging with the clinical picture and be

00:13:52.549 --> 00:13:54.970
aware of potential artifacts. Absolutely. Clinical

00:13:54.970 --> 00:13:57.009
correlation is paramount. Know the limitations

00:13:57.009 --> 00:14:00.049
of the test. Which leads us perfectly into evaluation

00:14:00.049 --> 00:14:03.230
and diagnosis. Given these challenges, occult

00:14:03.230 --> 00:14:05.940
fractures, tricky ligaments. What should alert

00:14:05.940 --> 00:14:08.120
us clinically? What's the typical presentation?

00:14:08.379 --> 00:14:10.039
Well, the starting point is always that high

00:14:10.039 --> 00:14:12.299
index of suspicion, especially with the right

00:14:12.299 --> 00:14:14.919
history. Clinically, patients usually complain

00:14:14.919 --> 00:14:17.720
of quite specific ulnar -sided wrist pain. Often,

00:14:17.879 --> 00:14:20.639
yes. They might say it's worth with bending the

00:14:20.639 --> 00:14:23.519
wrist, flexion, or extension. You often find

00:14:23.519 --> 00:14:25.919
some swelling on the dorsal side. And the key

00:14:25.919 --> 00:14:28.480
sign is usually localized tenderness right over

00:14:28.480 --> 00:14:31.139
the dorsal aspect of the trichotrum. If you press

00:14:31.139 --> 00:14:34.009
there, they'll likely jump. OK. And the history?

00:14:34.710 --> 00:14:36.750
Typically, it involves excessive load or the

00:14:36.750 --> 00:14:39.350
classic fall onto an outstretched hand injury,

00:14:39.769 --> 00:14:41.850
often with the wrist extended and pushed towards

00:14:41.850 --> 00:14:44.029
the little finger side, that ulnar deviation.

00:14:44.169 --> 00:14:46.629
Right. Think of someone falling backwards. But

00:14:46.629 --> 00:14:49.309
remember, the flexion radial deviation mechanism

00:14:49.309 --> 00:14:52.809
can also occur. So keep an open mind. Some also

00:14:52.809 --> 00:14:55.730
say wrist flexion can be more painful if it displaces

00:14:55.730 --> 00:14:58.809
a dorsal fragment. Good tip. Now, the radiographic

00:14:58.809 --> 00:15:01.090
challenges. You mentioned occult fractures. How

00:15:01.090 --> 00:15:03.529
often are we talking? It's surprisingly often.

00:15:03.750 --> 00:15:06.549
Some older studies suggested up to 80 % might

00:15:06.549 --> 00:15:09.409
be missed on initial plane x -rays. 80%. That's

00:15:09.409 --> 00:15:13.129
huge. It is. Even the best MRI study found 19

00:15:13.129 --> 00:15:15.769
% were occult on the initial radiographs they

00:15:15.769 --> 00:15:18.490
reviewed. It just highlights how insensitive

00:15:18.490 --> 00:15:22.210
standard x -rays can be for these small, complexly

00:15:22.210 --> 00:15:26.190
located bones. Compared to, say, a scaphoid fracture,

00:15:26.399 --> 00:15:29.100
The combined sensitivity of clinical exam and

00:15:29.100 --> 00:15:31.740
x -ray is often lower for the trichotrum. So

00:15:31.740 --> 00:15:34.440
what x -ray views should we be ordering to maximize

00:15:34.440 --> 00:15:37.139
our chances? You definitely need more than just

00:15:37.139 --> 00:15:39.519
the standard PA and lateral, although the lateral

00:15:39.519 --> 00:15:42.080
is often the most useful for seeing dorsal fragments

00:15:42.080 --> 00:15:44.960
displaced posteriorly. Okay. You should also

00:15:44.960 --> 00:15:48.259
get a 45 -degree pronated oblique view. This

00:15:48.259 --> 00:15:50.279
can sometimes rotate the trichoderm out from

00:15:50.279 --> 00:15:53.220
overlapping bones, and a radial deviation view

00:15:53.220 --> 00:15:56.019
might help highlight volar fractures by tensioning

00:15:56.019 --> 00:15:58.639
those ligaments. Any specific signs to look for?

00:15:58.740 --> 00:16:01.480
There's the slightly amusingly named pooping

00:16:01.480 --> 00:16:04.460
duck sign. The what now? The pooping duck. On

00:16:04.460 --> 00:16:06.539
the lateral view, sometimes the trigger profile

00:16:06.539 --> 00:16:08.960
looks a bit like a duck's head, and a dorsal

00:16:08.960 --> 00:16:11.399
cortical fragment can look like it's, well, exiting

00:16:11.399 --> 00:16:13.570
posteriorly. It's quite memorable. if you see

00:16:13.570 --> 00:16:15.649
it. I'll definitely remember that one. But even

00:16:15.649 --> 00:16:18.049
with these views, many are still missed. Yes,

00:16:18.129 --> 00:16:20.990
a significant number. So the rule has to be,

00:16:21.450 --> 00:16:24.389
if you strongly suspect a trichetral fracture

00:16:24.389 --> 00:16:27.389
based on the clinical picture, but the x -rays

00:16:27.389 --> 00:16:30.690
are normal or equivocal, you must pursue further

00:16:30.690 --> 00:16:35.149
imaging. Which brings us to CT and MRI. If x

00:16:35.149 --> 00:16:37.250
-rays aren't enough, what's the specific role

00:16:37.250 --> 00:16:39.710
for each of these? When do you choose one over

00:16:39.710 --> 00:16:42.769
the other? Excellent question. They both have

00:16:42.769 --> 00:16:46.269
critical but slightly different roles. CT is

00:16:46.269 --> 00:16:48.950
brilliant for bone detail. It's really the next

00:16:48.950 --> 00:16:51.409
step if you suspect an occult fracture that X

00:16:51.409 --> 00:16:53.600
-ray missed. better at seeing the fracture line.

00:16:53.720 --> 00:16:56.340
Much better. The cross -sectional views cut through

00:16:56.340 --> 00:16:59.299
the overlap issues. CT is also more accurate

00:16:59.299 --> 00:17:01.519
for measuring the exact size and volume of any

00:17:01.519 --> 00:17:03.600
fragments, which can be really important if you're

00:17:03.600 --> 00:17:05.859
considering surgery deciding if a piece is big

00:17:05.859 --> 00:17:08.039
enough to fix, for example. So high clinical

00:17:08.039 --> 00:17:10.660
suspicion, negative x -ray CT for bone. That's

00:17:10.660 --> 00:17:13.460
a very common and logical pathway, yes. But MRI

00:17:13.460 --> 00:17:15.460
comes into its own, crucially, when you're worried

00:17:15.460 --> 00:17:18.160
about instability or, as we've discussed extensively,

00:17:18.980 --> 00:17:20.700
associated ligament injury. Which is almost always.

00:17:20.910 --> 00:17:23.930
Which is in over 95 % of cases, according to

00:17:23.930 --> 00:17:27.710
that best study. So MRI is often essential. It's

00:17:27.710 --> 00:17:30.329
also useful if CT is negative, but you still

00:17:30.329 --> 00:17:33.410
suspect an occult fracture, as MRI can pick up

00:17:33.410 --> 00:17:36.500
bone marrow edema. the bruising, even before

00:17:36.500 --> 00:17:38.599
a clear fractal line forms. And it shows the

00:17:38.599 --> 00:17:42.039
soft tissues. Exactly. Ligaments, the TFCC, cartilage.

00:17:42.599 --> 00:17:44.980
Given the high rate of ligament tears, you could

00:17:44.980 --> 00:17:47.440
argue MRI is needed in most cases to get the

00:17:47.440 --> 00:17:50.000
full picture and planned treatment probably to

00:17:50.000 --> 00:17:52.279
avoid long -term problems like instability. So

00:17:52.279 --> 00:17:55.140
it's not really an either with CT and MRI sometimes?

00:17:55.299 --> 00:17:57.779
Often not. You might need both, depending on

00:17:57.779 --> 00:18:00.099
the clinical question. CT for the bone detail,

00:18:00.559 --> 00:18:02.740
MRI for the soft tissues, and subtle bone injury.

00:18:02.880 --> 00:18:04.900
And don't forget to look at the other carpal

00:18:04.900 --> 00:18:07.900
bones too. Best found edema in the hemat in nearly

00:18:07.900 --> 00:18:11.799
24 % of cases and in other wrist bones in 19%.

00:18:11.799 --> 00:18:14.579
It's often a wider injury pattern. Right, a comprehensive

00:18:14.579 --> 00:18:17.000
assessment is key. Okay, let's say we've navigated

00:18:17.000 --> 00:18:19.000
the diagnosis, we got the imaging, we know the

00:18:19.000 --> 00:18:21.180
fracture type and any associated injuries. How

00:18:21.180 --> 00:18:23.140
do we manage these? What's the first line approach

00:18:23.140 --> 00:18:26.920
usually? For the majority, especially those undisplaced

00:18:26.920 --> 00:18:29.720
or minimally displaced dorsal cortical fractures

00:18:29.720 --> 00:18:32.700
without instability. non -displaced body fractures,

00:18:33.279 --> 00:18:36.380
and stable volar fractures, non -operative management

00:18:36.380 --> 00:18:38.619
is definitely the way to go. Good news for the

00:18:38.619 --> 00:18:41.380
patient. Absolutely. The trichotrum generally

00:18:41.380 --> 00:18:44.339
heals well thanks to a decent blood supply. The

00:18:44.339 --> 00:18:47.319
mainstay is immobilization, usually for about

00:18:47.319 --> 00:18:49.779
four to six weeks. How is that typically done?

00:18:49.880 --> 00:18:52.740
Just a cast? It's usually phased. Yeah. Initially,

00:18:52.980 --> 00:18:54.680
say for the first few weeks while things are

00:18:54.680 --> 00:18:57.519
swollen and sore, a volar splint is often preferred.

00:18:57.759 --> 00:19:00.380
wrist in slight extension. Oh, movable. Yes,

00:19:00.400 --> 00:19:02.920
which allows for gentle range of motion exercises,

00:19:03.200 --> 00:19:05.759
just wiggling fingers and thumb, and for hygiene.

00:19:06.299 --> 00:19:08.240
Cold packs are also really helpful in this early

00:19:08.240 --> 00:19:10.579
phase, especially the first 72 hours, to manage

00:19:10.579 --> 00:19:13.259
that swelling and pain. Okay, split first, then

00:19:13.259 --> 00:19:15.650
what? Once the initial swelling settles, maybe

00:19:15.650 --> 00:19:17.630
around the 2 -4 week mark, depending on the case,

00:19:18.109 --> 00:19:20.170
you might transition to a proper short arm cast

00:19:20.170 --> 00:19:22.150
for more rigid support as healing progresses.

00:19:22.430 --> 00:19:24.490
And after the cast comes off, usually around

00:19:24.490 --> 00:19:27.089
four weeks, maybe slightly longer, you'd move

00:19:27.089 --> 00:19:30.069
to a removable wrist splinter brace. This allows

00:19:30.069 --> 00:19:32.369
for gradual increases in movement and starting

00:19:32.369 --> 00:19:36.059
gentle strengthening exercises. The goal is regaining

00:19:36.059 --> 00:19:38.579
mobility without stressing the healing site too

00:19:38.579 --> 00:19:41.480
much. When can patients expect to get back to

00:19:41.480 --> 00:19:43.839
normal activities? Weight -bearing activities

00:19:43.839 --> 00:19:46.240
can usually be gradually resumed around eight

00:19:46.240 --> 00:19:48.799
weeks post -injury, assuming things are progressing

00:19:48.799 --> 00:19:52.180
well and pain allows. Full unrestricted activity

00:19:52.180 --> 00:19:54.859
might take a bit longer. And the outcomes with

00:19:54.859 --> 00:19:57.180
this conservative approach? Generally very good.

00:19:57.319 --> 00:20:00.640
Most fractures manage this way heal well, often

00:20:00.640 --> 00:20:03.099
with what's called a fibrous union. Not solid

00:20:03.099 --> 00:20:05.440
bone. Not always solid bone, but strong fibrous

00:20:05.440 --> 00:20:07.839
tissue connecting the fragments. And crucially,

00:20:07.839 --> 00:20:10.720
this is usually completely asymptomatic. Patients

00:20:10.720 --> 00:20:13.000
typically get significant pain relief and recover

00:20:13.000 --> 00:20:15.299
good wrist motion, often within six to eight

00:20:15.299 --> 00:20:18.019
weeks of starting treatment. So for most, surgery

00:20:18.019 --> 00:20:20.980
isn't needed. That's very reassuring. But inevitably,

00:20:21.259 --> 00:20:23.900
some cases do require surgery. What are the main

00:20:23.900 --> 00:20:25.880
indications for going down the operative route?

00:20:26.059 --> 00:20:29.500
Yes, surgery definitely has its place. The main

00:20:29.500 --> 00:20:31.660
triggers are significant displacement of the

00:20:31.660 --> 00:20:34.079
fracture fragments, clear evidence of carpal

00:20:34.079 --> 00:20:36.940
instability, associated fracture dislocations

00:20:36.940 --> 00:20:40.119
involving the trichotrum, or those relatively

00:20:40.119 --> 00:20:43.640
rare cases of symptomatic non -union where conservative

00:20:43.640 --> 00:20:45.980
treatment has failed and the patient still has

00:20:45.980 --> 00:20:49.079
pain or mechanical issues. So instability and

00:20:49.079 --> 00:20:52.220
significant displacement are key flags. Absolutely.

00:20:52.589 --> 00:20:54.750
The goal of surgery is to put things back where

00:20:54.750 --> 00:20:57.970
they belong, make the wrists stable, and optimize

00:20:57.970 --> 00:21:00.750
long -term function. What surgical techniques

00:21:00.750 --> 00:21:03.109
are typically used? It depends on the specifics,

00:21:03.170 --> 00:21:05.609
of course. Open reduction internal fixation,

00:21:05.849 --> 00:21:08.670
or ORAC -A, is common for displaced body fractures

00:21:08.670 --> 00:21:10.950
or instability. So opening it up and fixing it.

00:21:11.170 --> 00:21:13.170
Exactly. You make an incision, carefully reduce

00:21:13.170 --> 00:21:15.430
the fragments back into place, and then fix them

00:21:15.430 --> 00:21:17.910
internally. You might use small interfragmentary

00:21:17.910 --> 00:21:20.170
screws to compress the fragments, or perhaps

00:21:20.170 --> 00:21:23.230
K -wires, which are temporary pins. If ligaments

00:21:23.230 --> 00:21:25.609
need repairing, you might use suture anchors

00:21:25.609 --> 00:21:27.970
drilled into the bone. Right, like Herbert screws

00:21:27.970 --> 00:21:30.910
for fracture dislocations. Herbert screws were

00:21:30.910 --> 00:21:33.950
often used, yes, sometimes combined with K -wire

00:21:33.950 --> 00:21:36.849
stabilization across the lunatric ritual joint

00:21:36.849 --> 00:21:40.299
if needed. Okay, so... RIF is one option. Are

00:21:40.299 --> 00:21:42.599
there others? Yes. Particularly for those problematic,

00:21:42.759 --> 00:21:45.259
symptomatic non -unions. Especially if it's a

00:21:45.259 --> 00:21:47.559
small fragment and not involving the joint surface.

00:21:47.720 --> 00:21:51.180
Not articular. Precisely. In those cases, fragment

00:21:51.180 --> 00:21:53.500
excision, just removing the troublesome little

00:21:53.500 --> 00:21:55.799
piece, can be a very effective option. Just take

00:21:55.799 --> 00:21:58.700
it out. Yes. Sometimes trying to fix a tiny chip

00:21:58.700 --> 00:22:00.799
is more trouble than it's worth. There was a

00:22:00.799 --> 00:22:03.119
case report of a young man with a tiny, painful

00:22:03.119 --> 00:22:06.279
non -union, only about five millimeter. They

00:22:06.279 --> 00:22:09.480
excised it. brief immobilization, and he became

00:22:09.480 --> 00:22:12.339
pain -free. Others have reported success excising

00:22:12.339 --> 00:22:15.019
slightly larger fragments, too. It's a good option

00:22:15.019 --> 00:22:17.480
for selected cases. That makes sense for small,

00:22:17.619 --> 00:22:19.700
non -articular, persistently painful fragments.

00:22:19.900 --> 00:22:22.019
What about post -op? Still needs immobilization,

00:22:22.039 --> 00:22:24.579
of course. Maybe six weeks in a cast, then a

00:22:24.579 --> 00:22:27.220
brace, followed by rehab. Outcomes are generally

00:22:27.220 --> 00:22:30.220
good. Maybe some slight loss of flexion or extension

00:22:30.220 --> 00:22:32.900
compared to the other side. But usually satisfactory

00:22:32.900 --> 00:22:35.740
function and pain relief. And crucially, You

00:22:35.740 --> 00:22:38.200
have to address the associated injuries found

00:22:38.200 --> 00:22:41.380
earlier. Absolutely vital. If the pisiform is

00:22:41.380 --> 00:22:44.220
subloxed, you might need to excise it or fix

00:22:44.220 --> 00:22:47.019
the trichotrum. If the lunotrichotral ligament

00:22:47.019 --> 00:22:50.559
is unstable, it needs pinning or repair. Surgery

00:22:50.559 --> 00:22:53.019
has to address the whole problem, not just the

00:22:53.019 --> 00:22:56.240
trichotral fragment in isolation. It really highlights

00:22:56.240 --> 00:22:59.400
that holistic diagnostic approach again. Okay,

00:22:59.400 --> 00:23:02.329
so... After treatment, whether conservative or

00:23:02.329 --> 00:23:04.549
surgical, what's the general outlook and what

00:23:04.549 --> 00:23:06.269
potential complications should we be keeping

00:23:06.269 --> 00:23:08.789
an eye out for long term? The overall outlook

00:23:08.789 --> 00:23:11.430
is generally positive. As we said, functional

00:23:11.430 --> 00:23:13.609
recovery is typically good to excellent for most

00:23:13.609 --> 00:23:16.210
patients, even those with some associated ligament

00:23:16.210 --> 00:23:18.630
issues provided they're managed correctly. People

00:23:18.630 --> 00:23:20.730
often get back to good function relatively quickly.

00:23:20.869 --> 00:23:22.849
That's good to hear. What about non -union? You

00:23:22.849 --> 00:23:25.309
mentioned it's rare but can be symptomatic. Yes.

00:23:25.549 --> 00:23:28.029
True non -union is uncommon, especially in body

00:23:28.029 --> 00:23:31.339
fractures. Fibrous union is more frequent and

00:23:31.339 --> 00:23:34.140
usually fine. But if a true non -union does occur

00:23:34.140 --> 00:23:36.539
and becomes symptomatic, causing persistent pain

00:23:36.539 --> 00:23:39.079
or clicking, that's when it can cause significant

00:23:39.079 --> 00:23:42.000
disability and might need that surgical excision

00:23:42.000 --> 00:23:45.220
or fixation we discussed. Okay. What other long

00:23:45.220 --> 00:23:47.599
-term issues can crop up? Well, related to the

00:23:47.599 --> 00:23:50.740
associated injuries mostly. TFCC injury is common,

00:23:50.839 --> 00:23:53.420
especially with dorsal fractures. If it remains

00:23:53.420 --> 00:23:55.519
symptomatic despite initial treatment, it might

00:23:55.519 --> 00:23:57.980
need arthroscopic debridement or repair later

00:23:57.980 --> 00:24:01.119
on. Right. Pizzotrich or periosaurosis arthritis

00:24:01.119 --> 00:24:03.200
in the joint between the pisiform and trichotrum

00:24:03.200 --> 00:24:05.680
can develop if the fracture went into that joint

00:24:05.680 --> 00:24:08.660
or sometimes related to a nonunion there. If

00:24:08.660 --> 00:24:10.960
that becomes painful, excising the pisiform can

00:24:10.960 --> 00:24:13.220
be a good solution. Makes sense. And the big

00:24:13.220 --> 00:24:15.900
one, though thankfully less common if things

00:24:15.900 --> 00:24:19.140
are managed well initially, is persistent carpal

00:24:19.140 --> 00:24:22.299
instability. This usually results from missed

00:24:22.299 --> 00:24:24.859
or undertreated ligament injuries, especially

00:24:24.859 --> 00:24:27.660
involving the lunotrichotrial ligament. It really

00:24:27.660 --> 00:24:30.339
underlines why that initial thorough assessment

00:24:30.339 --> 00:24:33.180
with advanced imaging is so incredibly important

00:24:33.180 --> 00:24:36.119
to catch these instability patterns early. Absolutely.

00:24:36.680 --> 00:24:38.920
Preventing that long -term instability seems

00:24:38.920 --> 00:24:42.059
key. Well, this has been an incredibly thorough

00:24:42.059 --> 00:24:44.339
exploration. We've really covered the ground

00:24:44.339 --> 00:24:46.740
from presentation to long -term follow -up. We

00:24:46.740 --> 00:24:49.099
have. It's clear that these fractures, despite

00:24:49.099 --> 00:24:52.380
being common, are anything but simple. From understanding

00:24:52.380 --> 00:24:54.799
the shift in thinking about mechanisms away from

00:24:54.799 --> 00:24:58.420
simple, either to this complex, multifactorial

00:24:58.420 --> 00:25:02.079
picture revealed by MRI, to appreciating that

00:25:02.079 --> 00:25:05.160
staggering 95 % rate of associated ligament injuries,

00:25:05.480 --> 00:25:07.759
it really changes the game. It really does. It

00:25:07.759 --> 00:25:10.299
mandates that high index of suspicion and the

00:25:10.299 --> 00:25:12.519
readiness to use advanced imaging like CT and

00:25:12.519 --> 00:25:14.940
MRI, not just for the bone, but critically for

00:25:14.940 --> 00:25:17.299
those soft tissues. It's about seeing the whole

00:25:17.299 --> 00:25:20.259
wrist complex. Precisely. and then tailoring

00:25:20.259 --> 00:25:22.720
the management, knowing that most do well with

00:25:22.720 --> 00:25:25.799
straightforward immobilization, but also recognizing

00:25:25.799 --> 00:25:29.059
the clear indications for surgery, like ORIF,

00:25:29.359 --> 00:25:32.319
or even fragment excision when needed. This deep

00:25:32.319 --> 00:25:34.519
dive has certainly illuminated a corner of wrist

00:25:34.519 --> 00:25:37.329
trauma that deserves careful attention. It might

00:25:37.329 --> 00:25:39.289
look like a small chip, but the implications

00:25:39.289 --> 00:25:42.190
can be significant. Indeed. If you, listening,

00:25:42.450 --> 00:25:44.789
found this deep dive valuable, please do consider

00:25:44.789 --> 00:25:46.690
leaving us a rating or perhaps sharing it with

00:25:46.690 --> 00:25:49.170
a colleague. And finally, a thought to take away

00:25:49.170 --> 00:25:51.950
and perhaps ponder. Given everything we've discussed

00:25:51.950 --> 00:25:54.309
about the extensive soft tissue attachments and

00:25:54.309 --> 00:25:57.130
that near -universal ligament involvement, how

00:25:57.130 --> 00:25:59.529
might future advances perhaps in dynamic imaging

00:25:59.529 --> 00:26:02.289
techniques or maybe even intraoperative assessment

00:26:02.289 --> 00:26:04.849
tools further refine our understanding of wrist

00:26:04.849 --> 00:26:08.240
kinematics after these trick - How could that

00:26:08.240 --> 00:26:10.400
ultimately help us improve long -term stability

00:26:10.400 --> 00:26:12.299
and function for our patients even more?
