WEBVTT

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Welcome to the Deep Dive. Today we're focusing

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on a specific and actually fairly common shoulder

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issue, the acromioclavicular joint or the AC

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joint. That's right. Often called a shoulder

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separation. Exactly. And while it might sound

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minor, it can really affect daily life and particularly

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athletic function. We often see it in high impact

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sports, don't we? Indeed. Rugby, cycling falls,

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horse riding, quite common. And the impact on

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function can be, well, profound. So our goal

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in this deep dive is to really get under the

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skin of AC joint injuries. We want to provide

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you, our audience of mid -senior medical professionals,

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with a solid, practical understanding, moving

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beyond the basics to the kind of detailed insights

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that help in clinic. Looking at everything from

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diagnosis, classification, right through to the

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latest thinking on treatment and recovery. Precisely.

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So let's start at the beginning. An AC joint

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disruption isn't the same as a typical shoulder

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dislocation, is it? Can you clarify that distinction?

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Yes, that's a really crucial point. People often

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use shoulder dislocation quite loosely, but usually...

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That refers to the glenohumeral joint, the main

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ball and socket. Where the humerus comes out

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of the sock. Exactly. An AC joint disruption,

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though, is different. It's a separation right

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at the top, the apex of the shoulder. It's specifically

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where the collarbone, the clavicle, meets the

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acromeon, which is the highest point of the shoulder

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blade. So the injury is a traumatic separation

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of those two bones. The ligaments holding them

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together get damaged to varying degrees. And

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you mentioned it's common in sports. Very. Especially

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contact sports or activities where falls directly

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onto the shoulder are a risk. Think rugby tackles,

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falls in motorsports, equestrian sports. And

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it causes significant pain and limitation. Absolutely.

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It's not just a minor niggle. It can be genuinely

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debilitating, affecting simple daily tasks, let

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alone high level sport or manual work. So understanding

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the anatomy is key then. What makes this small

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joint so important for shoulder function and

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yet seemingly so vulnerable? Well, functionally,

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it's critical. It's technically a diarthrodial

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joint, a synovial joint, but its main job is

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to link the entire arm and shoulder blade assembly,

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the upper limb, to the main trunk, the axial

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skeleton. It's the strip connecting the arm to

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the body, essentially. You could think of it

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like that, yes. It allows the scapula to move

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properly, which is vital for everything from

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reaching up to throwing. Without that stable

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connection, shoulder function is severely compromised.

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And what keeps it stable? It sounds like a complex

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setup. It is. It relies on both static and dynamic

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stabilizers. First, you have the acromioclavicular

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ligaments themselves. They form the joint capsule.

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These are key for horizontal stability, stopping

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the clavicle, moving too far forwards or backwards

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relative to the acromion. Okay, so front -to

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-back stability. Precisely. But arguably even

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more important, especially for vertical stability,

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stopping the collarbone popping up, are the coracuclavicular

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ligaments, the CC ligaments. Ah, yes, the cornoid

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and trapezoid. Exactly, the cornoid and the trapezoid.

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They run from the coracoid process, that hook

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-like bit of bone on the scapula, up to the underside

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of the distal clavicle. Think of them as really

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strong suspension cables. Holding the clavicle

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down. Essentially, yes. Preventing superior displacement.

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If these are torn, that's when you typically

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see that characteristic bump on the shoulder

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in more severe injuries. Their integrity is absolutely

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vital. So those are the static ligamentous restraints.

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What about muscles? Good point. We also have

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crucial dynamic stabilizers. The large deltoid

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and trapezius muscles drape over the shoulder.

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While their main job is movement, they also provide

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dynamic compression and stability to the AC joint.

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Especially if the ligaments are injured. Yes,

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their role becomes even more important then,

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although they can't fully compensate if the main

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ligaments, particularly the CCs, are completely

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ruptured. Is there anything else within the joint

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itself? Well, sometimes there's a meniscal disc,

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a small fibrocardilage pad between the bone ends.

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It's not present in everyone, but when it is,

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it helps with load distribution and acts as a

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bit of a shock absorber. So, quite an intricate

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structure. Given this anatomy, what usually causes

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these ligaments to tear? What are the typical

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mechanisms of injury? It's almost always down

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to trauma, a significant force applied to the

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joint that overcomes the strength of those ligaments.

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The most common mechanism, by far, is direct

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trauma. This is usually a fall directly onto

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the point of the shoulder, hitting the acromeon,

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often with the arm tucked into the side, adducted.

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Like in a rugby tackle or falling off a bike?

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Exactly that scenario. The force drives the shoulder

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blade downwards, but the clavicle stays put,

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creating this massive sheer force across the

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AC joint, tearing the ligaments. OK, that makes

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sense. Are there other ways it can happen? Yes.

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Less commonly, you can get indirect trauma. The

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classic example is a foosh injury, a fall on

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an outstretched hand. Ah, yes, foosh. The force

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travels up the arm, through the humerus and scapula,

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and ultimately stresses the AC joint. Yeah. We

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also see some cases of repetitive microtrauma,

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maybe in weightlifters or overhead athletes,

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but that's usually a lower grade issue managed

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differently. You mentioned earlier it's common.

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Are there specific groups more at risk? Yes,

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there's a really striking demographic pattern.

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Males are affected much more often than females.

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The ratio is something like five to one. Five

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to one, that's significant. It is, and it's particularly

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prevalent in the 20 to 35 age group. This strongly

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correlates with higher participation rates in

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those high -risk contact sports and activities

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we discussed. It really highlights that link

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between activity levels and injury risk. That

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5 .1 ratio certainly reflects what we tend to

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see clinically. So once an injury happens, classifying

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it correctly is the next vital step for management.

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The Rockwood classification is the standard,

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isn't it? Absolutely. Classification is fundamental

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in orthopedics. It gives us a common language

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and guides treatment. The Rockwood system from

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1998 is the most comprehensive and widely used

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for AC joint injuries. And it goes beyond just

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mild, moderate, severe. It does. It defines six

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distinct types based on exactly which ligaments

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are torn, the AC and the crucial cc ligaments,

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and importantly, the direction and degree of

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clavicle displacement relative to both the acromion

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and the coracuate process. It provides a really

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robust framework for prognosis and deciding on

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treatment. Okay, let's walk through them then.

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Starting with the mildest. Type 1. Right. Type

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I is essentially a sprain of the AC ligament

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only. The fibers are stretched, maybe some minor

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tearing, but crucially, the ligament is intact

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overall and the joint remains stable. So no displacement.

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No displacement. Yeah. Radiographically, it looks

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normal. The CC ligaments are completely unharmed.

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Patients present with localized tenderness, but

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it typically settles well with conservative management

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within a few weeks. OK. Moving up to type 2.

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In type 2, the AC ligament is completely torn.

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That's the key difference. However, the CC ligaments,

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while they might be sprained, are still intact

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and functional. Ah, so the main vertical stabilizers

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are okay. Correct. Because the CCs are intact,

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you only get partial displacement or subluxation

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of the joint. On an X -ray, you'll see the clavicle

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slightly elevated relative to the acrimine, maybe

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some overlap, but it's not a complete dislocation.

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And management is usually non -operative for

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type 2 as well? Generally, yes. Because the crucial

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cc ligaments are holding things relatively stable,

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non -operative treatment with structured rehab

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usually yields good results. Now, type 3. This

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is often where the debate starts, isn't it? How

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does Rockwood define it? And perhaps more interestingly,

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how do you approach these clinically? You're

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absolutely right. Type 3 is the controversial

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one. According to the strict Rockwood definition,

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a type 3 injury involves a complete rupture of

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both the AC ligaments and the CC ligaments. So

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everything's torn? Anatomically, yes. This results

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in a complete dislocation of the joint. The clavicle

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displaces superiorly, often quite significantly,

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because there's nothing holding it down anymore.

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This is when you see that obvious step -off deformity

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or the piano key sign on examination. Right.

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Now clinically, This is where nuance comes in.

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While Rockwood says both are completely torn,

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in my practical experience managing these, I

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sometimes view a grade 3 as perhaps having a

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complete AC tear, but maybe only a significant

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sprain or partial tear of the CC ligaments. Ah,

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so not always a complete CC rupture in practice

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for what gets called a type 3. Well, the displacement

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is there, as per Rockwood 3 definition, implying

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significant CC injury. But the absolute necessity

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for surgery isn't always clear -cut for every

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single patient who fits the type 3 x -ray picture.

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It allows us to consider the patient's needs

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more closely. I see. So you'd weigh other factors.

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Absolutely. What are the patient's functional

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demands? Are they a high -level overhead athlete?

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A manual laborer needing maximum shoulder stability?

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In those cases, Even with a standard type 3 appearance,

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surgery might be the better option to restore

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optimal mechanics. Versus someone more sedentary.

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Exactly. A less active individual might do perfectly

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well functionally with non -operative management,

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despite the radiographic appearance. This contrasts

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sharply with what I might term category three

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injuries in my thinking, encompassing rockwood

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types for V insects. The more severe types. Yes.

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In those, there's absolutely no doubt both ligament

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complexes are completely disrupted, the displacement

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is far more dramatic, and the instability is

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profound. Surgery is almost always required for

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those higher grades. So for type 3, it's about

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bridging that gap between the strict anatomical

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classification and the individual patient's context.

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That's a really helpful clinical perspective.

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Okay, let's look at those higher grades then,

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starting with type 4. What's the defining feature

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there? Type 4 is defined by posterior displacement.

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The clavicle doesn't just go up, it goes backwards,

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often getting trapped behind or even punching

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through the trapezius muscle. Button -holing

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through the muscle. That's the term, yes. This

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makes it incredibly unstable and very difficult

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to reduce and hold non -operatively. Because

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of this inherent instability and the soft tissue

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damage, early surgical stabilization is pretty

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much always necessary to avoid long -term problems.

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Right. And type V, how much worse does it get?

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Type V represents a very severe superior displacement.

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We're talking significant vertical distance between

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the clavicle and the acromeon, often 100 % or

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more displacement compared to the normal side.

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Wow. This indicates not just complete AC and

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CC ligament rupture, but also extensive stripping

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of the muscle attachments, the deltoid and trapezius

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fascia from the distal clavicle. The deformity

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is usually very obvious. And surgery is mandatory.

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Almost invariably, yes. The instability is profound.

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The functional deficit is huge. Non -operative

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management simply doesn't work for type V. You

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need surgery to restore function and stability.

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And finally, the rare type VI. Type VI is indeed

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very uncommon. Here, the clavicle displaces inferiorly,

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downwards, getting lodged underneath the acromand,

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or even more rarely, the coracoid process. Downwards?

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How does that happen? Usually requires a very

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specific high energy impact. Like the higher

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grades of IV and V, it represents profound instability

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due to massive ligamentous and muscular disruption.

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Surgery is required here too. So that covers

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the adult classifications. But you sometimes

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see these in children. Are there special considerations

00:11:20.419 --> 00:11:24.159
for pediatric AC injuries? Ah, yes. A very important

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distinction. Children aren't just small adults,

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especially skeletally. The key difference is

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the periosteum around the distal clavicle in

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a child's that fibrous sleeve covering the bone.

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It's stronger in kids. It's relatively much thicker

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and stronger compared to the ligaments themselves.

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So when a child sustains a force that would tear

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ligaments in an adult, in the child, it's more

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likely to cause an injury through the bone's

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growth plate. or cause the periosteal sleeve

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to tear or avulse off the bone rather than a

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pure mid -substance ligament tear. So the injury

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pattern is different. Exactly. The periosteal

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sleeve can sometimes even remain partially intact,

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acting like a natural tether or hinge, which

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can influence the degree of displacement and

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often leads to better healing potential and remodeling

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capacity compared to adults. While you can still

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see injuries resembling the higher rockwood types

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in children, the underlying pathology and healing

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response are different. That's fascinating. Okay,

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let's shift to diagnosis. We suspect an AC joint

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injury. How does the patient typically present

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and what's the diagnostic pathway? Well, the

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presentation really mirrors the severity, the

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walk would type. For lower grades, type I and

00:12:34.950 --> 00:12:37.990
II, the main feature is localized pain right

00:12:37.990 --> 00:12:41.009
over the AC joint. It's often quite tender to

00:12:41.009 --> 00:12:43.230
touch. And worst with certain movements? Definitely.

00:12:43.850 --> 00:12:46.350
Bringing the arm across the chest, horizontal

00:12:46.350 --> 00:12:48.769
adduction usually provokes it. Lying on that

00:12:48.769 --> 00:12:50.809
side at night is often very uncomfortable, too.

00:12:51.190 --> 00:12:53.129
You might see some mild swelling, but usually

00:12:53.129 --> 00:12:55.629
no obvious deformity. And for the higher grades,

00:12:55.990 --> 00:12:57.850
three and above? It's usually much more obvious.

00:12:58.309 --> 00:13:00.190
Besides more intense pain, you'll likely see

00:13:00.190 --> 00:13:02.730
that visible deformity, the step off or bump

00:13:02.730 --> 00:13:04.649
where the clavicle is riding high. The whole

00:13:04.649 --> 00:13:06.730
arm might hang slightly lower on the affected

00:13:06.730 --> 00:13:09.549
side. So the visual inspection is key. What about

00:13:09.549 --> 00:13:11.529
the hands -on clinical examination? What are

00:13:11.529 --> 00:13:13.710
you specifically looking for? The physical exam

00:13:13.710 --> 00:13:16.710
is crucial. First, visual inspection for that

00:13:16.710 --> 00:13:20.009
asymmetry or step -off deformity. Then, careful

00:13:20.009 --> 00:13:22.809
palpation directly over the AC joint itself tenderness

00:13:22.809 --> 00:13:25.669
is a very consistent sign. And testing stability.

00:13:25.950 --> 00:13:28.830
Yes. We gently assess for abnormal movement.

00:13:29.389 --> 00:13:31.090
Can you press the clavicle down? Does it reduce?

00:13:31.350 --> 00:13:33.389
Does it spring back up when you release pressure?

00:13:33.769 --> 00:13:36.389
Panache sign. That indicates instability. Do

00:13:36.389 --> 00:13:40.009
you ever hear or feel anything? Sometimes. Yes.

00:13:40.350 --> 00:13:42.389
During testing, or even with a patient's own

00:13:42.389 --> 00:13:45.409
movement, you might hear or feel a painful clunk

00:13:45.409 --> 00:13:48.649
as the joint subluxes or reduces. That's a strong

00:13:48.649 --> 00:13:51.309
indicator of significant ligament damage. We

00:13:51.309 --> 00:13:54.090
also check range of motion, noting pain on specific

00:13:54.090 --> 00:13:56.769
movements like adduction or elevation. Okay,

00:13:56.909 --> 00:13:58.769
so a clinical exam gives you a strong suspicion.

00:13:59.330 --> 00:14:02.070
How does imaging confirm things and help classify

00:14:02.070 --> 00:14:04.629
the injury accurately? Let's start with x -rays.

00:14:05.070 --> 00:14:07.610
X -rays are the first -line investigation. Absolutely

00:14:07.610 --> 00:14:10.169
essential. They confirm the diagnosis by showing

00:14:10.169 --> 00:14:11.970
the relationship between the clavicle and the

00:14:11.970 --> 00:14:14.190
acromion, revealing the degree of displacement.

00:14:14.789 --> 00:14:17.330
They also crucially rule out associated fractures.

00:14:17.549 --> 00:14:19.649
Are there specific views you need? A standard

00:14:19.649 --> 00:14:22.210
AP view is useful, but often a Zonka view is

00:14:22.210 --> 00:14:25.090
preferred. It's angled slightly to give a clearer,

00:14:25.289 --> 00:14:27.809
unobstructed view of the AC joint itself. What

00:14:27.809 --> 00:14:30.110
about stress views? You hear about those. Yes,

00:14:30.330 --> 00:14:32.570
stress x -rays can be very helpful, especially

00:14:32.570 --> 00:14:35.149
if you suspect a type 3 injury. But the resting

00:14:35.149 --> 00:14:37.509
films look only mildly displaced, or perhaps

00:14:37.509 --> 00:14:39.950
like a type 2. The patient holds weights in each

00:14:39.950 --> 00:14:42.750
hand. To pull the arm down. Exactly. The weight

00:14:42.750 --> 00:14:45.250
stresses the joint, pulling the scapula and arm

00:14:45.250 --> 00:14:48.230
down, which exaggerates any underlying instability

00:14:48.230 --> 00:14:50.809
and makes the clavicle displacement much more

00:14:50.809 --> 00:14:53.090
obvious if the CC ligaments are indeed torn.

00:14:53.230 --> 00:14:55.629
It helps differentiate between stable type 2

00:14:55.629 --> 00:14:57.870
and unstable type 3 injuries sometimes. Right.

00:14:58.090 --> 00:15:01.919
When might you use a CT scan? CT gives much better

00:15:01.919 --> 00:15:04.779
bony detail than x -rays. It's particularly useful

00:15:04.779 --> 00:15:07.200
if you suspect an associated fracture, maybe

00:15:07.200 --> 00:15:10.340
of the distal clavicle, the acromeon, or, importantly,

00:15:10.519 --> 00:15:13.179
the coracoid process. It can also show the degree

00:15:13.179 --> 00:15:15.679
of static displacement very clearly, especially

00:15:15.679 --> 00:15:18.500
any posterior displacement seen in type 4. An

00:15:18.500 --> 00:15:21.379
MRI. Is that commonly used? MRI isn't usually

00:15:21.379 --> 00:15:23.679
the first -line test for an acute, straightforward

00:15:23.679 --> 00:15:26.480
AC separation. Its strength isn't showing soft

00:15:26.480 --> 00:15:28.620
tissues in great detail. So the ligaments themselves?

00:15:29.019 --> 00:15:32.840
Precisely. MRI is excellent for visualizing the

00:15:32.840 --> 00:15:35.539
ligaments directly, assessing the extent of tearing,

00:15:35.940 --> 00:15:38.340
partial versus complete, and identifying any

00:15:38.340 --> 00:15:40.779
associated injuries to other soft tissues like

00:15:40.779 --> 00:15:43.919
the rotator cuff or labrum. It's most useful

00:15:43.919 --> 00:15:46.379
when the diagnosis is uncertain after x -rays,

00:15:46.820 --> 00:15:48.820
perhaps in lower grade injuries with persistent

00:15:48.820 --> 00:15:51.500
symptoms or if you suspect complications later

00:15:51.500 --> 00:15:54.080
on. Okay, that clarifies the imaging pathway.

00:15:54.779 --> 00:15:57.139
So diagnosis made, classification determined.

00:15:57.480 --> 00:15:59.720
Now we face the treatment decision non -operative

00:15:59.720 --> 00:16:01.919
versus operative. Let's start with the non -operative

00:16:01.919 --> 00:16:04.019
route. What's the approach and who's it best

00:16:04.019 --> 00:16:06.620
suited for? Non -operative management is definitely

00:16:06.620 --> 00:16:09.070
the mainstay for lower grade injuries. Rockwood

00:16:09.070 --> 00:16:11.809
type I and II, and often considered for type

00:16:11.809 --> 00:16:14.570
III, as we discussed. The initial focus is purely

00:16:14.570 --> 00:16:16.629
on symptom control. How do you achieve that?

00:16:16.889 --> 00:16:19.669
Rest is key, initially. Using an arm sling provides

00:16:19.669 --> 00:16:21.929
comfort and support, ideally a broad arm sling

00:16:21.929 --> 00:16:23.470
that supports the elbow and takes the weight

00:16:23.470 --> 00:16:25.490
off the shoulder, rather than a simple collar

00:16:25.490 --> 00:16:28.230
and cuff. We also use ice therapy frequently

00:16:28.230 --> 00:16:30.549
in the acute phase to reduce swelling and pain,

00:16:30.929 --> 00:16:33.830
alongside NSAIDs, nonsteroidal anti -inflammatory

00:16:33.830 --> 00:16:36.559
drugs. And how long does that initial phase last?

00:16:36.879 --> 00:16:39.100
It depends. For a type Y, the sling might only

00:16:39.100 --> 00:16:41.539
be needed for comfort for a week or so. For type

00:16:41.539 --> 00:16:45.059
2, maybe longer, perhaps four to six weeks for

00:16:45.059 --> 00:16:47.740
symptoms to really settle. Once the acute pain

00:16:47.740 --> 00:16:50.679
starts to subside, the focus shifts very quickly

00:16:50.679 --> 00:16:53.159
towards rehabilitation. So rehab starts quite

00:16:53.159 --> 00:16:55.460
early then. Can you walk us through the typical

00:16:55.460 --> 00:16:58.500
phases? Yes. Rehab shouldn't be delayed. It's

00:16:58.500 --> 00:17:01.620
a structured, phased process. Phase one is the

00:17:01.620 --> 00:17:04.319
acute phase. Pain control, protecting the joint,

00:17:04.799 --> 00:17:06.759
but starting gentle range of motion exercises,

00:17:07.160 --> 00:17:09.779
often passive or active assisted, and isometric

00:17:09.779 --> 00:17:11.980
exercises, contracting muscles without moving

00:17:11.980 --> 00:17:14.059
the joint much. Keeping things moving gently.

00:17:14.299 --> 00:17:16.319
Exactly. Then phase two is about strengthening.

00:17:16.650 --> 00:17:19.750
As pain allows, we introduce isotonic exercises,

00:17:19.990 --> 00:17:22.190
moving against resistance, initially keeping

00:17:22.190 --> 00:17:24.730
movements below shoulder height to avoid stressing

00:17:24.730 --> 00:17:27.009
the joint too much. We focus on strengthening

00:17:27.009 --> 00:17:29.309
the rotator cuff, the scapular stabilizers, the

00:17:29.309 --> 00:17:31.269
bigger shoulder muscles, building back the support

00:17:31.269 --> 00:17:34.910
system, precisely. Phase three involves progressing

00:17:34.910 --> 00:17:37.450
to more functional movements, increasing strength,

00:17:37.529 --> 00:17:40.329
power, endurance, and crucially, neuromuscular

00:17:40.329 --> 00:17:42.430
control, teaching the muscles to work together

00:17:42.430 --> 00:17:45.029
effectively again, getting ready for more demanding

00:17:45.029 --> 00:17:48.769
tasks. And the final phase. Phase 4 is the return

00:17:48.769 --> 00:17:51.869
to activity. This is highly individualized, focusing

00:17:51.869 --> 00:17:54.890
on sport -specific or work -specific drills,

00:17:55.470 --> 00:17:57.509
gradually reintroducing the demands the patient

00:17:57.509 --> 00:18:00.390
needs to meet. For example, return to contact

00:18:00.390 --> 00:18:02.769
sports might be considered around 6 -12 weeks,

00:18:03.089 --> 00:18:05.190
depending on progress and injury severity. That

00:18:05.190 --> 00:18:07.809
sounds very thorough. What are the typical outcomes

00:18:07.809 --> 00:18:10.349
for non -operative treatments, say for type I

00:18:10.349 --> 00:18:12.849
and 2, and maybe select a type 3? For type I

00:18:12.849 --> 00:18:14.470
and 2, the outcomes are generally excellent.

00:18:14.680 --> 00:18:16.880
Most patients get significant pain relief within

00:18:16.880 --> 00:18:19.299
a few weeks, and many athletes can return to

00:18:19.299 --> 00:18:21.660
sport quite quickly, sometimes within 2 -4 weeks

00:18:21.660 --> 00:18:24.240
for type 2. Full recovery might take up to 12

00:18:24.240 --> 00:18:26.940
weeks, perhaps a bit longer, for some type 3s

00:18:26.940 --> 00:18:29.559
managed this way. Are there any potential downsides

00:18:29.559 --> 00:18:31.880
or long -term issues with non -operative management?

00:18:32.200 --> 00:18:35.480
While most do very well, a minority might experience

00:18:35.480 --> 00:18:38.579
persistent symptoms. This could be some residual

00:18:38.579 --> 00:18:41.740
ache, discomfort with specific activities, or

00:18:41.740 --> 00:18:44.000
potentially developing AC joint arthritis down

00:18:44.000 --> 00:18:46.210
the line. There's also some evidence suggesting

00:18:46.210 --> 00:18:48.089
a slight reduction in things like bench press

00:18:48.089 --> 00:18:50.430
strength. In some individuals, although most

00:18:50.430 --> 00:18:53.309
don't find it functionally limiting, if symptoms

00:18:53.309 --> 00:18:55.650
are still significant at six months, they're

00:18:55.650 --> 00:18:58.829
more likely to persist beyond a year. Okay. So

00:18:58.829 --> 00:19:00.690
when does surgery become the recommendation?

00:19:00.890 --> 00:19:03.210
What are the main indications and the goals of

00:19:03.210 --> 00:19:05.700
operating? Surgery is generally reserved for

00:19:05.700 --> 00:19:07.819
the higher grade injuries, definitely types 4,

00:19:07.920 --> 00:19:10.420
V, and 6. And it's strongly considered for type

00:19:10.420 --> 00:19:13.140
3 injuries, particularly in active individuals

00:19:13.140 --> 00:19:16.559
with high demands or when non -operative treatment

00:19:16.559 --> 00:19:18.940
for a type 3 has failed to provide satisfactory

00:19:18.940 --> 00:19:21.220
function or pain relief. And what are you trying

00:19:21.220 --> 00:19:24.079
to achieve with surgery? The main goals are,

00:19:24.299 --> 00:19:26.539
firstly, an anatomical reduction, getting the

00:19:26.539 --> 00:19:28.980
clavicle back into its proper position relative

00:19:28.980 --> 00:19:33.160
to the acromion and coracoid. Secondly, secure

00:19:33.160 --> 00:19:35.259
a fixation to hold it there while the tissues

00:19:35.259 --> 00:19:39.180
heal. And thirdly, restoring stability both horizontally

00:19:39.180 --> 00:19:42.019
and vertically to allow for good long -term function.

00:19:42.559 --> 00:19:45.259
Ultimately, we want a stable, pain -free shoulder.

00:19:45.960 --> 00:19:48.319
How have surgical techniques changed over the

00:19:48.319 --> 00:19:51.240
years? Oh, considerably. Older techniques, like

00:19:51.240 --> 00:19:54.079
the Weaver Dun procedure, often involved resecting

00:19:54.079 --> 00:19:56.339
the end of the clavicle and using local tissues

00:19:56.339 --> 00:19:58.940
like the coracoacromial ligament for reconstruction.

00:19:59.200 --> 00:20:01.980
Now, the trend is much more towards anatomical

00:20:01.980 --> 00:20:04.500
reconstruction, often using stronger fixation

00:20:04.500 --> 00:20:07.240
methods. Like what? Things like suspensory fixation

00:20:07.240 --> 00:20:10.059
devices have become very popular. These use strong

00:20:10.059 --> 00:20:12.279
sutures or synthetic tapes passed around the

00:20:12.279 --> 00:20:14.240
coracoid and through tunnels in the clavicle

00:20:14.240 --> 00:20:17.039
to replicate the function of the native cc ligaments.

00:20:17.339 --> 00:20:20.460
Some techniques involve plates or screws, though

00:20:20.460 --> 00:20:23.180
these often require removal later. In chronic

00:20:23.180 --> 00:20:25.599
cases, sometimes biological grafts are used to

00:20:25.599 --> 00:20:27.460
augment the repair. You mentioned there are over

00:20:27.460 --> 00:20:29.900
a hundred techniques described. That suggests

00:20:29.900 --> 00:20:32.539
there isn't one single best way. That's absolutely

00:20:32.539 --> 00:20:34.640
right. The sheer number of techniques reflects

00:20:34.640 --> 00:20:37.000
the ongoing challenge and the lack of universal

00:20:37.000 --> 00:20:40.480
consensus on the perfect repair. Options range

00:20:40.480 --> 00:20:43.700
from direct ligament repair, if possible acutely,

00:20:44.140 --> 00:20:46.619
using local tissue transfers, synthetic ligaments,

00:20:47.000 --> 00:20:49.819
suture button devices, hook plates. The list

00:20:49.819 --> 00:20:52.859
goes on. The choice depends on the injury pattern,

00:20:53.559 --> 00:20:56.019
the surgeon's preference and experience, the

00:20:56.019 --> 00:20:58.819
patient's needs, and whether the injury is acute

00:20:58.819 --> 00:21:01.259
or chronic. It's fascinating though that even

00:21:01.259 --> 00:21:04.259
with surgery aiming for perfect anatomical reduction,

00:21:04.599 --> 00:21:06.700
research hasn't always shown a direct link between

00:21:06.700 --> 00:21:09.559
that perfect x -ray and better pain or function

00:21:09.559 --> 00:21:12.200
outcomes. That's a really key observation in

00:21:12.200 --> 00:21:15.099
the field. While we strive for anatomical correction,

00:21:15.619 --> 00:21:17.880
the correlation between radiographic appearance

00:21:17.880 --> 00:21:20.900
and patient -reported outcomes isn't always straightforward.

00:21:21.259 --> 00:21:23.599
So a perfect x -ray doesn't guarantee a perfect

00:21:23.599 --> 00:21:26.269
result for the patient? Not necessarily. You

00:21:26.269 --> 00:21:28.589
can have a patient with a radiographically flawless

00:21:28.589 --> 00:21:31.430
reduction who still has some pain or functional

00:21:31.430 --> 00:21:34.089
limitation. And conversely, someone with slight

00:21:34.089 --> 00:21:36.609
residual displacement on x -ray might be completely

00:21:36.609 --> 00:21:38.910
asymptomatic and function at a very high level.

00:21:38.990 --> 00:21:41.970
Why is that? It's complex. Factors like muscle

00:21:41.970 --> 00:21:44.930
strength, neuromuscular control, individual pain

00:21:44.930 --> 00:21:48.049
perception, biological healing response, and

00:21:48.049 --> 00:21:50.349
the demands placed on the shoulder all play a

00:21:50.349 --> 00:21:52.980
huge role. It underscores that our ultimate goal

00:21:52.980 --> 00:21:55.660
isn't just to fix the x -ray image, but to achieve

00:21:55.660 --> 00:21:58.500
a stable, painless shoulder that allows the patient

00:21:58.500 --> 00:22:01.329
to return to their desired activities. Patient

00:22:01.329 --> 00:22:03.769
function and satisfaction are the real measures

00:22:03.769 --> 00:22:06.609
of success. A very important perspective. So,

00:22:06.710 --> 00:22:09.430
looking at the bigger picture of prognosis, how

00:22:09.430 --> 00:22:11.910
do the recovery paths and long -term outcomes

00:22:11.910 --> 00:22:14.410
generally compare between non -op and surgical

00:22:14.410 --> 00:22:16.890
approaches? Well, for types 1 and 2, non -op

00:22:16.890 --> 00:22:19.509
is the clear winner, good outcomes, quicker return

00:22:19.509 --> 00:22:21.890
to activities generally. Sling use is short.

00:22:22.079 --> 00:22:25.259
maybe 7 -10 days for type 1, perhaps 4 -6 weeks

00:22:25.259 --> 00:22:27.579
for type 2 protection. Long -term studies show

00:22:27.579 --> 00:22:29.400
good results, though as mentioned, a few might

00:22:29.400 --> 00:22:31.740
have lingering issues or develop arthritis later.

00:22:32.180 --> 00:22:34.279
If symptoms persist at 6 months, they might linger

00:22:34.279 --> 00:22:36.380
longer. And for the higher grades where surgery

00:22:36.380 --> 00:22:39.339
is more common? For types 3 and above, surgery

00:22:39.339 --> 00:22:41.720
generally provides better anatomical reduction

00:22:41.720 --> 00:22:44.279
and stability. However, it comes with a higher

00:22:44.279 --> 00:22:47.119
risk of complications compared to non -operative

00:22:47.119 --> 00:22:48.869
treatment. What kind of complications are we

00:22:48.869 --> 00:22:51.809
talking about? Things like mild residual instability

00:22:51.809 --> 00:22:55.369
can still occur, despite the surgery. Hardware

00:22:55.369 --> 00:22:58.170
issues are possible migration, prominence needing

00:22:58.170 --> 00:23:01.410
removal. Infection is a risk, though thankfully

00:23:01.410 --> 00:23:05.349
low, maybe around 1%. Nerve injury is rare, but

00:23:05.349 --> 00:23:08.410
possible. Stiffness or soft tissue ossification

00:23:08.410 --> 00:23:10.990
can also occur. So there are definite trade -offs?

00:23:11.210 --> 00:23:13.380
Absolutely. Non -operative might mean quicker

00:23:13.380 --> 00:23:15.900
initial recovery, but potentially more residual

00:23:15.900 --> 00:23:18.180
instability or cosmetic deformity for higher

00:23:18.180 --> 00:23:21.660
grades. Surgery aims for better anatomical stability,

00:23:22.000 --> 00:23:24.160
but has a longer recovery and carries those surgical

00:23:24.160 --> 00:23:27.059
risks. Despite the risks though, meta -analyses

00:23:27.059 --> 00:23:30.359
show high return to sport rates, around 94%,

00:23:30.359 --> 00:23:32.539
for patients having surgery for type 3 or higher

00:23:32.539 --> 00:23:34.980
injuries. And rehabilitation after surgery is

00:23:34.980 --> 00:23:37.579
presumably just as crucial. Even more so, arguably.

00:23:37.869 --> 00:23:40.430
Post -surgical rehab is absolutely vital to protect

00:23:40.430 --> 00:23:43.109
the repair, regain motion safely, build strength,

00:23:43.450 --> 00:23:46.329
and ultimately optimize the outcome. It's carefully

00:23:46.329 --> 00:23:48.549
progressed. What does that involve initially?

00:23:49.450 --> 00:23:52.109
Initially, there might be restrictions, perhaps

00:23:52.109 --> 00:23:54.549
limiting abduction above 90 degrees for a period.

00:23:55.369 --> 00:23:57.769
Gentle pendulum exercises are often started early

00:23:57.769 --> 00:24:00.309
to maintain some motion and prevent stiffness

00:24:00.309 --> 00:24:02.990
without stressing the repair. And then building

00:24:02.990 --> 00:24:05.430
up. Yes, then progressively introducing range

00:24:05.430 --> 00:24:07.849
of motion followed by strengthening exercises

00:24:07.849 --> 00:24:10.710
for the rotator cuff, deltoid, and scapular muscles.

00:24:11.289 --> 00:24:13.569
It's all about providing dynamic support to the

00:24:13.569 --> 00:24:16.930
reconstructed joint. The protocol needs tailoring

00:24:16.930 --> 00:24:20.490
to the specific surgery performed. So, summing

00:24:20.490 --> 00:24:22.549
up the long -term picture. It really hinges on

00:24:22.549 --> 00:24:24.769
the initial injury, severity, and the chosen

00:24:24.769 --> 00:24:27.650
management. But regardless of whether it's non

00:24:27.650 --> 00:24:30.690
-op or surgical, a meticulously planned patient

00:24:30.690 --> 00:24:33.190
-specific rehabilitation program is paramount.

00:24:33.710 --> 00:24:35.829
That tailored rehab, considering the patient's

00:24:35.829 --> 00:24:37.950
individual goals and activity levels, is probably

00:24:37.950 --> 00:24:39.809
the single most important factor in achieving

00:24:39.809 --> 00:24:41.930
the best possible long -term function. Okay,

00:24:42.230 --> 00:24:44.650
that leads us nicely into modern approaches.

00:24:45.269 --> 00:24:47.450
Many listeners will be keen to hear about current

00:24:47.450 --> 00:24:50.829
thinking on reconstruction. Could you share your

00:24:50.829 --> 00:24:53.769
management philosophy, particularly how you differentiate

00:24:53.769 --> 00:24:56.890
acute versus chronic injuries when deciding on

00:24:56.890 --> 00:24:59.230
intervention? Certainly. My approach is very

00:24:59.230 --> 00:25:01.410
much guided by the timing, the acuity of the

00:25:01.410 --> 00:25:05.309
injury. I generally define acute as less than

00:25:05.309 --> 00:25:07.390
about one week from injury. And how does that

00:25:07.390 --> 00:25:10.009
guide your initial steps? For most acute injuries,

00:25:10.410 --> 00:25:13.470
one week after diagnosis, my preference is usually

00:25:13.470 --> 00:25:15.890
to start with conservative management. That means

00:25:15.890 --> 00:25:18.880
a sling, but mainly just for comfort. not strict

00:25:18.880 --> 00:25:22.500
immobilization, analgesia for pain, and crucially

00:25:22.500 --> 00:25:25.160
starting rehabilitation with early active mobilization

00:25:25.160 --> 00:25:28.480
as soon as the patient feels able. So give conservative

00:25:28.480 --> 00:25:31.400
treatment a chance first in most acute cases.

00:25:31.480 --> 00:25:34.160
In many, yes, but the initial inflammation settles

00:25:34.160 --> 00:25:36.119
see how the patient progresses functionally.

00:25:36.319 --> 00:25:37.759
However, there are definite exceptions where

00:25:37.759 --> 00:25:39.859
I'd recommend early surgery even in the acute

00:25:39.859 --> 00:25:42.480
phase, such as cases with really severe pain,

00:25:42.819 --> 00:25:45.319
especially if the clavicle is clearly buttonholed

00:25:45.319 --> 00:25:49.200
through the trapezius, a type 4. Also, high -level

00:25:49.200 --> 00:25:51.579
overhead athletes often benefit from early surgery

00:25:51.579 --> 00:25:54.759
to restore mechanics optimally. Any associated

00:25:54.759 --> 00:25:57.579
neurovascular injury or an open fracture are,

00:25:57.579 --> 00:26:00.640
of course, immediate surgical indications. What's

00:26:00.640 --> 00:26:02.880
your follow -up strategy, then, for those managed

00:26:02.880 --> 00:26:05.119
conservatively initially? I typically review

00:26:05.119 --> 00:26:07.730
them at three weeks. If they're improving well,

00:26:08.029 --> 00:26:10.369
symptoms settling, function returning, we continue

00:26:10.369 --> 00:26:12.430
with the symptomatic management and gradual return

00:26:12.430 --> 00:26:14.930
to activity plan in another review around three

00:26:14.930 --> 00:26:16.710
months. And if they're not doing well at three

00:26:16.710 --> 00:26:18.369
weeks? If they're still really struggling with

00:26:18.369 --> 00:26:21.829
pain or instability or just not coping functionally,

00:26:22.210 --> 00:26:24.309
then I'd offer early surgical reconstruction

00:26:24.309 --> 00:26:26.690
at that point. No sense in delaying further if

00:26:26.690 --> 00:26:28.690
it's clearly not working. And the three month

00:26:28.690 --> 00:26:31.630
review? At three months, if they've successfully

00:26:31.630 --> 00:26:33.910
returned to their activities with minimal issues,

00:26:34.289 --> 00:26:37.299
fantastic. They could be discharged. If they're

00:26:37.299 --> 00:26:39.980
still not coping despite extended rehab, then

00:26:39.980 --> 00:26:42.059
surgery is offered as the definitive solution.

00:26:42.440 --> 00:26:44.380
It's a pathway that allows for non -operative

00:26:44.380 --> 00:26:47.359
management where appropriate but ensures timely

00:26:47.359 --> 00:26:49.779
intervention if needed. That makes good clinical

00:26:49.779 --> 00:26:53.319
sense. When surgery is the path chosen, what

00:26:53.319 --> 00:26:55.880
constitutes an ideal reconstruction technique

00:26:55.880 --> 00:26:58.660
today? What are the key goals? Ah, the ideal

00:26:58.660 --> 00:27:00.400
technique. That's what we're all striving for.

00:27:00.980 --> 00:27:02.940
Based on current understanding, I think there

00:27:02.940 --> 00:27:07.029
are about six key criteria we aim for. One, anatomical

00:27:07.029 --> 00:27:09.990
restoration. We want to recreate both the CC

00:27:09.990 --> 00:27:12.730
and AC ligament anatomy as closely as possible.

00:27:12.869 --> 00:27:14.450
Right, putting things back where they belong.

00:27:15.150 --> 00:27:18.329
Two, strength plus flexibility. The repair needs

00:27:18.329 --> 00:27:20.630
to be strong enough immediately, but also allow

00:27:20.630 --> 00:27:23.269
some natural physiological motion at the AC joint.

00:27:23.670 --> 00:27:25.750
Two, rigid isn't good either. Makes sense. Three,

00:27:26.210 --> 00:27:28.799
allow early rehabilitation. The construct must

00:27:28.799 --> 00:27:30.759
be stable enough to let patients start moving

00:27:30.759 --> 00:27:33.000
relatively early to prevent stiffness and speed

00:27:33.000 --> 00:27:36.519
up recovery. Get them gone and quickly. 4. Biological

00:27:36.519 --> 00:27:40.019
integration. Ideally, the repair encourages the

00:27:40.019 --> 00:27:42.299
body's own tissues to heal into it, providing

00:27:42.299 --> 00:27:44.940
long -term, durable stability. Less reliance

00:27:44.940 --> 00:27:48.019
on artificial materials long -term. 5. Avoid

00:27:48.019 --> 00:27:51.059
donor site morbidity. If using the patient's

00:27:51.059 --> 00:27:53.750
own tissue, Autographed. We want to minimize

00:27:53.750 --> 00:27:56.109
any problems from the harvest site. That's an

00:27:56.109 --> 00:27:59.309
advantage of good synthetic options. And six,

00:28:00.210 --> 00:28:03.309
eliminate routine hardware removal. We want techniques

00:28:03.309 --> 00:28:05.349
where implants don't routinely need a second

00:28:05.349 --> 00:28:07.589
operation just to take them out. That's a very

00:28:07.589 --> 00:28:10.380
clear checklist. With those criteria in mind,

00:28:10.480 --> 00:28:12.480
can you tell us about your preferred approach

00:28:12.480 --> 00:28:14.700
to the triple anatomical technique? What does

00:28:14.700 --> 00:28:16.559
it involve? Yes, this technique is designed to

00:28:16.559 --> 00:28:19.380
hit those criteria, aiming for anatomical restoration

00:28:19.380 --> 00:28:22.059
with biological potential, creating a strong

00:28:22.059 --> 00:28:24.599
construct that allows early mobilization. It

00:28:24.599 --> 00:28:26.779
has three main parts. Triple technique, three

00:28:26.779 --> 00:28:30.240
parts. Makes sense. First is an anatomic corcoclavicular

00:28:30.240 --> 00:28:33.299
ligament reconstruction. I use a strong synthetic

00:28:33.299 --> 00:28:35.180
ligament, specifically the Lars ligament, which

00:28:35.180 --> 00:28:38.700
is polyethylene tetrafasolate, because it's incredibly

00:28:38.700 --> 00:28:41.019
strong, stronger than the native CC ligaments,

00:28:41.259 --> 00:28:44.200
providing immediate stability. Its braided structure

00:28:44.200 --> 00:28:46.880
also encourages tissue ingrowth, aiming for that

00:28:46.880 --> 00:28:49.900
biological integration over time. We pass it

00:28:49.900 --> 00:28:51.920
anatomically through tunnels in the clavicle

00:28:51.920 --> 00:28:54.480
at the ligament footprints and then secure it

00:28:54.480 --> 00:28:56.319
around or through the coracoid. Okay, so that

00:28:56.319 --> 00:28:58.299
replaces the CC ligaments. What's the second

00:28:58.299 --> 00:29:00.920
part? Second is an acromoclavicular ligament

00:29:00.920 --> 00:29:03.660
reconstruction, often using a transfer of the

00:29:03.660 --> 00:29:06.660
nearby coraco -chromial ligament. This directly

00:29:06.660 --> 00:29:08.799
addresses the AC joint capsule and ligament,

00:29:09.099 --> 00:29:11.180
restoring horizontal stability, which the CC

00:29:11.180 --> 00:29:13.900
reconstruction alone doesn't fully address. Got

00:29:13.900 --> 00:29:16.900
it. And the third component. The third part is

00:29:16.900 --> 00:29:20.640
a delto -tripetal fascia repair. This involves

00:29:20.640 --> 00:29:23.680
carefully repairing the torn fascia of the deltoid

00:29:23.680 --> 00:29:26.019
and trapezius muscles where they attach to the

00:29:26.019 --> 00:29:29.039
clavicle and acromion. This is especially important

00:29:29.039 --> 00:29:31.559
in higher grade injuries, like type V, where

00:29:31.559 --> 00:29:34.500
the fascia is often ripped off. It adds significant

00:29:34.500 --> 00:29:36.759
reinforcement and dynamic stability to the whole

00:29:36.759 --> 00:29:40.079
construct. So you're rebuilding the CCs, the

00:29:40.079 --> 00:29:43.200
ACs, and reinforcing the muscle envelope. Exactly.

00:29:43.369 --> 00:29:46.049
It's a comprehensive belt and braces approach.

00:29:46.609 --> 00:29:48.589
We also believe transferring a ligament like

00:29:48.589 --> 00:29:51.930
the CA ligament might help restore some proprioception

00:29:51.930 --> 00:29:54.829
that joint position sends. Overall, we find it

00:29:54.829 --> 00:29:57.849
safe, reproducible, and very effective at restoring

00:29:57.849 --> 00:30:00.250
stability, which allows for that crucial early

00:30:00.250 --> 00:30:03.210
recovery and return to sport. It certainly sounds

00:30:03.210 --> 00:30:06.500
robust. Finally, for those patients, especially

00:30:06.500 --> 00:30:08.440
athletes undergoing this kind of reconstruction,

00:30:09.039 --> 00:30:11.180
how do you manage their return to sport? What

00:30:11.180 --> 00:30:14.279
are realistic timelines? Return to sport is always

00:30:14.279 --> 00:30:17.099
a gradual, criterion -based process, not just

00:30:17.099 --> 00:30:20.000
about time. It absolutely requires teamwork between

00:30:20.000 --> 00:30:22.559
me, the physiotherapist, and often strength and

00:30:22.559 --> 00:30:24.319
conditioning coaches. Everyone has to be on the

00:30:24.319 --> 00:30:26.700
same page. Completely. We need to see the patient

00:30:26.700 --> 00:30:29.599
meet specific functional milestones, strength,

00:30:29.740 --> 00:30:32.140
range of motion, stability, sport -specific movements

00:30:32.140 --> 00:30:35.269
before they progress. But based on our experience,

00:30:36.009 --> 00:30:38.049
contact athletes like rugby players usually take

00:30:38.049 --> 00:30:40.410
around three to four months post -op before returning

00:30:40.410 --> 00:30:42.970
to full contact. They need that time for solid

00:30:42.970 --> 00:30:44.910
healing and to rebuild the strength to withstand

00:30:44.910 --> 00:30:48.009
impacts. And overhead athletes, pitchers, tennis

00:30:48.009 --> 00:30:50.650
players. They typically need longer, often six

00:30:50.650 --> 00:30:53.349
to nine months. Their sports require extreme

00:30:53.349 --> 00:30:56.089
ranges of motion, power and fine motor control.

00:30:56.329 --> 00:30:59.569
Rushing them back risks re -injury or poor performance.

00:30:59.930 --> 00:31:02.410
Any activities that allow a quicker return. Interestingly,

00:31:02.829 --> 00:31:05.289
we find horse riders and cyclists can often get

00:31:05.289 --> 00:31:07.690
back sooner, sometimes in less than three months,

00:31:08.390 --> 00:31:10.799
while falls can cause the initial injury. The

00:31:10.799 --> 00:31:13.059
actual activity itself doesn't usually place

00:31:13.059 --> 00:31:15.799
the same high -impact repetitive stress on the

00:31:15.799 --> 00:31:19.180
AC joint as contact or overhead sports. And sport

00:31:19.180 --> 00:31:22.319
-specific rehab is key throughout. Absolutely

00:31:22.319 --> 00:31:24.440
critical. A rugby player needs drills with a

00:31:24.440 --> 00:31:27.099
ball, controlled contact simulation. A swimmer

00:31:27.099 --> 00:31:30.140
needs pool -based rehab. Tailoring the exercises

00:31:30.140 --> 00:31:32.000
to mimic the demands of their specific sport

00:31:32.000 --> 00:31:34.140
ensures they are truly ready and confident when

00:31:34.140 --> 00:31:36.240
they return to competition. Professor, this has

00:31:36.240 --> 00:31:38.380
been incredibly insightful. We've covered a huge

00:31:38.380 --> 00:31:40.319
amount from the basic anatomy right through to

00:31:40.319 --> 00:31:42.420
your specific advanced surgical technique for

00:31:42.420 --> 00:31:45.119
AC joint disruptions. Thank you for clarifying

00:31:45.119 --> 00:31:47.140
so much complexity. It's been a real pleasure.

00:31:47.420 --> 00:31:50.119
These are common injuries, and getting the management

00:31:50.119 --> 00:31:53.339
right based on a detailed understanding really

00:31:53.339 --> 00:31:55.579
does make a difference for our patients. Absolutely.

00:31:56.190 --> 00:31:58.630
Just as a final thought for our listeners, as

00:31:58.630 --> 00:32:01.369
our understanding of biomechanics and biology

00:32:01.369 --> 00:32:03.930
continues to advance, perhaps with things like

00:32:03.930 --> 00:32:07.490
better biological augmentation or even more refined,

00:32:07.710 --> 00:32:10.269
minimally invasive techniques, how do you see

00:32:10.269 --> 00:32:12.910
the approach to AC joint stability and recovery

00:32:12.910 --> 00:32:15.390
evolving further in the future? That's the exciting

00:32:15.390 --> 00:32:17.950
part, isn't it? Always looking ahead. Indeed.

00:32:18.289 --> 00:32:20.309
If this deep dive has sharpened your perspective

00:32:20.309 --> 00:32:22.829
on AC joint injuries, please do consider rating

00:32:22.829 --> 00:32:24.450
and sharing it with your colleagues. Thank you

00:32:24.450 --> 00:32:25.609
for joining us. Thank you.
