WEBVTT

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Imagine this for a moment. You're active, maybe

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really dedicated to a sport, perhaps football,

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running, dance, whatever it is, or maybe you

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just love being active. And then, this ache starts,

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deep in your hip, maybe you're groin, not from

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a specific injury, it just creeps up. Sometimes

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it's sharp, especially after activity or even

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just sitting for a while. It's a very common

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story, unfortunately. And you live with it for

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months, maybe years, adjusting how you move,

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hoping it'll go away. but it often doesn't. Well,

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that nagging pain, that mechanical niggle might

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be femurocetabular impingement or FAI. Indeed.

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It affects a huge number of symptomatic athletes,

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maybe 55%. But it's also present, often silently,

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in about 10, 15 % of the general adult population.

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Today, we're diving deep into FAI. I'm your host,

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here to navigate through the complex details,

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and I'm joined by our expert, Jest. He has this

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fantastic ability to take really complex medical

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information, see the patterns, and explain it

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all in a way that makes sense. It's great to

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be here. The hip joint really is an incredible

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structure, designed for both power and subtle

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movement. But yeah. even small changes in its

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shape can cause significant problems like FAI.

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Absolutely. And that's our mission today to really

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get under the skin of femurocetabular impingement.

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We'll look at what it is, the different types,

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who gets it, why they get it, and crucially,

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how it's diagnosed and what the latest treatment

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options look like from physio right through to

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surgery and what it all means for long term hip

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health. So let's get stuck in. OK, let's start

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right at the beginning. Femoral acetabular impingement,

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FAI. What exactly is it? It doesn't sound like

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a typical disease, does it? More like a physical

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fit issue. That's a very good way to put it,

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actually. It's not a disease like an infection.

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It's what we call a pathomechanical process.

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It's half a mechanical, right? Yeah. Think of

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your hip as a near -perfect ball and socket joint.

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You have the femoral head, the ball at the top

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of your thigh bone, fitting snugly into the acetabulum,

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which is the socket in your pelvis. Normally,

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they glide smoothly. In FAI, that smooth gliding

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is disrupted. You get abnormal contact, basically.

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The femur and the acetabulum repeatedly collide

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or rub against each other incorrectly during

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movement. A collision. Exactly, a mechanical

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collision. And this isn't gentle. It's often

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a forceful pinching or grinding. Over time, this

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constant abnormal contact starts to cause damage.

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Damage to what, specifically? Well, typically

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the first structure to suffer is the labrum.

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That's a ring of strong fiber cartilage around

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the rim of the socket. Right. Think of it like

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a gasket or a seal. Okay. the rubbing damages

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it, causes tears, then the articular cartilage,

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that incredibly smooth white lining in the bone

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surfaces that lets them glide, that gets injured

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too. Right, the slippery stuff. Precisely. And

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this ongoing damage, this mechanical insult,

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is what leads to the progressive hip pain and

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stiffness that people experience. So yes, fundamentally,

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it's a problem of shape and mechanics. That makes

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sense. And you mentioned different types, that

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this extra bone or abnormal shape shows up in

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distinct ways. That's right. Although the underlying

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issue is this abnormal bone shape causing the

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collision, where that abnormality is located,

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defines the type, and often correlates with who

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tends to get it. We generally talk about two

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main types, though very often people have a mix

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of both. OK. The first is cam impingement. The

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name comes from engineering, like a camshaft

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that isn't perfectly round. Ah, OK. This is a

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femoral based disorder. The problem is on the

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ball side, the femur. We tend to see this more

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in young athletic males. Young men, okay. Yes.

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Essentially, the femoral head, or usually the

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junction between the head and the neck, isn't

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perfectly spherical. It's a bit aspherical or

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too broad, often with a characteristic bump on

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the front outer side. A bump on the ball. Exactly.

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We're talking about a decreased head -to -neck

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ratio or offset. Basically, the smooth contour

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is lost. Now imagine this non -spherical ball

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trying to move fully within the socket. It's

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gonna jam, isn't it? Precisely. As a hip flexes

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and rotates inwards, that bump, the cam lesion,

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effectively levers against the rim of the socket.

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This creates a shearing force right where the

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cartilage meets the labrum. Ouch. Yes. Instead

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of gliding, it peels the cartilage away from

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the bone that's cartilage delamination, and it

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can tear the labrum, causing labral separation.

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On x -rays, you might see what's called a pistol

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grip deformity because of the shape. Pistol grip.

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Got it. So that's cam. What's the other type?

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The second type is pincer impingement. This is

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an acetabular -based disorder. The issue lies

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with the socket, the acetabular. So the socket's

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the problem this time. Correct. We tend to see

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this more frequently in active middle -aged women.

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Here, the socket rim has too much coverage, an

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overhang, usually at the front and top part.

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Too much socket. In effect, yes. It might be

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because the socket itself is angled slightly

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backwards, it's acetabular retroversion, or it

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might be just generally too deep, what we call

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acetabular protrusia or coxoprofinda. So with

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pincer, when the hip flexes, instead of the bump

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shearing things, the overhanging rim of the socket

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directly pinches or crushes the labrum against

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the femoral neck. Crushing the seal. Exactly.

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This often causes tearing within the substance

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of the labrum itself. And interestingly, You

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can get what's called a contracoup cartilage

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injury. Contracoup? Like in head injuries? Similar

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principle. As the front of the neck hits the

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overhanging socket rim, it levers the back of

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the femoral head against the back wall of the

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socket, damaging the cartilage there opposite

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the main impingement site. Wow. Okay. Any specific

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signs on x -ray for pincer? A classic one, especially

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with retroversion, is the crossover sign on a

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pelvis x -ray. where the front and back walls

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of the socket seemed to cross over. Right, CAM

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and PINCER. But you said people often have both.

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Yes, absolutely. It's really important to stress

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that. Combined impingement, a mix of both CAM

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and PINCER features, is actually the most common

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scenario we see in symptomatic patients, perhaps

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up to 80 % of cases. So often, we need to address

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both aspects surgically. OK. That's clear. These

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aren't just minor anacomical quirks then. They

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cause real mechanical problems. And it sounds

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like this isn't just about the immediate pain

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of that collision. It sets off a kind of chain

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reaction, doesn't it? Leading to bigger problems

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down the line. Precisely. That's the critical

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thing to understand about the long -term picture.

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The immediate pain is from the pinching and rubbing,

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yes. But it's the chronic, repetitive nature

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of that mechanical collision that does the real

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damage over time. This constant friction, whether

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it is shearing from a cam lesion or crushing

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from a pincer, leads to progressive labral degeneration

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and tears. The labrum just isn't designed to

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withstand that kind of abuse, so it wears down

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phrase tears. And the cartilage too. And the

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cartilage, too. That smooth gliding surface suffers

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significant cartilage damage. It might soften,

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fray, develop blisters, or even full thickness

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tears or flaps. And the trouble with articular

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cartilage is its very limited ability to heal

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itself. It doesn't repair well. Not really, no.

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So over time, this relentless wear and tear transforms

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localized damage into broader joint degeneration.

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which is why FAI is now recognized as a major

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known risk factor for the development of osteoarthritis

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of the hip. Ah, so it leads to arthritis earlier.

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It certainly seems to accelerate the process,

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yes, leading to hip dysfunction and potentially

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the need for hip replacement surgery much earlier

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in life than we might otherwise expect. It's

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a pathway from subtle shape difference to significant

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joint failure, potentially. That's quite sobering.

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So if these bone shapes are the root cause, what

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causes them? Is it something you're born with?

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Is it genetics, or does activity play a role?

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You mentioned athletes. It's a really key question.

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And the honest answer is we're still figuring

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out all the details. But the current thinking

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points towards a combination of factors. It's

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rarely just one thing. Firstly, there does seem

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to be a genetic predisposition. Research suggests

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genetic factors may contribute. Specific small

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variations in our DNA, things called SNPs, single

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nucleotide polymorphisms, tiny genetic differences,

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some. like variants in genes called GDF5, FRZB,

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DI02, HOX9, have been linked to particular hip

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shapes, both on the femur and socket side, and

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also to an increased risk of developing osteoarthritis.

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So your genes can literally shape your hip in

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a way that makes FAI more likely. It seems that

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way, yes. It sets the stage, potentially. Interesting.

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And the athlete connection, does playing sport

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cause it? That's the athlete paradox, as you

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called it. We definitely see more FAI, particularly

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the cam type in athletes. But, and this is really

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crucial, exercise does not cause FAI in a normal

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hip. Okay, that's important. Yes. The leading

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theory is that during adolescence, when the bones

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are still growing and developing, the increased

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stress along the great plate of a hip from high

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intensity sports, think repetitive deep flexion,

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rotation, like in football, hockey, martial arts,

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might trigger a sort of stress reaction. The

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bone reacts to the stress? Exactly an increased

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stress reaction bone formation the body adapts

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by laying down more bone But in this area it

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can result in that cam deformity that bump So

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intense activity during those key growth years

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and someone perhaps already predisposed might

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lead to the shape forming So activity doesn't

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cause it from scratch, but it can contribute

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to the shape developing, or at least make an

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existing shape symptomatic earlier. Precisely.

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If you already have that subtle anatomical difference,

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vigorous activity involving lots of hip movement

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will provoke the impingement more often and more

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forcefully, leading to symptoms sooner. It unmasks

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the problem. Right. Are there other factors?

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Childhood conditions? Yes, definitely. Childhood

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influences are important. A history of slipped

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capital femoral bypasses or SEFE is a major one.

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That's where the ball part slips off the thigh

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bone at the growth plate during childhood. Even

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after it's fixed surgically, the residual shape

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change, the deformity, can directly cause impingement

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later in life. Other conditions like perthase

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disease, which affects blood supply to the hip

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in childhood, and hip dysplasia, where the socket

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is too shallow, are also linked to a higher chance

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of developing FAI. So it's often multifactorial.

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Genetics, developmental stress, maybe previous

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hip issues. OK, that makes sense. But then...

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If these shapes are actually quite common, as

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you said earlier, why do some people have terrible

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symptoms while others with the same x -ray findings

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feel Absolutely fine. It's confusing. It is confusing

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and it's a huge area of research You're right.

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The prevalence of the bone shapes is surprisingly

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high We mentioned 10 15 percent in the general

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population having the morphology But remember

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that study the meta -analysis by Frank at all

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They found cam deformity in 37 percent and pincer

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deformity in a massive 67 percent of asymptomatic

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volunteers people with no hip pain at all 67

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percent Wow Exactly. So a huge number of people

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are walking around with these FAI bone shapes

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without any symptoms. The morphology itself doesn't

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automatically equal pain. We also see those gender

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differences in prevalence can more in men, pincer

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more in women. But again, many are asymptomatic.

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So what's the tipping point? Why do symptoms

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start for some people? The general consensus

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is that symptoms develop when the mechanical

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collision finally starts causing significant

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damage to the cartilage or the labrum. It's when

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those soft tissues, which have nerve endings,

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start to break down under the repetitive strain.

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Ah, so it's the damage, not just the shape. Exactly.

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The onset of pain, clicking stiffness that usually

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signals that the pathological process is now

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actively injuring the joint structures. And once

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that happens, if the impingement continues, the

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condition is likely to worsen. The damage progresses.

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Having the shape is one thing. Having symptoms

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means the joint is now suffering. Right. Which

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brings us neatly to diagnosis. For someone with

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that nagging hip or groin pain, what are the

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telltale signs? How do doctors start to suspect

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FAI specifically, given hip pain can be so vague?

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It can be very vague, you're right. That's why

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a careful history and a thorough physical exam

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are absolutely paramount. What do patients typically

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report? Usually a gradual onset of hip pain.

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It's often not a sudden thing. The location is

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most commonly deep in the groin area, but as

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we said, it can radiate to the thigh, buttock,

00:12:11.370 --> 00:12:14.970
outer hip, even the back. The C sign? Yes. Often

00:12:14.970 --> 00:12:17.250
they'll use that characteristic C sign, cupping

00:12:17.250 --> 00:12:18.970
their hand around the side in front of the hip

00:12:18.970 --> 00:12:21.509
to show where it hurts. That's quite suggestive

00:12:21.509 --> 00:12:23.909
of something inside the joint. Okay. And what

00:12:23.909 --> 00:12:27.629
makes it worse? Classically, pain is exacerbated

00:12:27.629 --> 00:12:30.909
by hip flexion and internal rotation. So activities

00:12:30.909 --> 00:12:33.450
involving those movements, high intensity sports,

00:12:34.090 --> 00:12:36.840
squatting, getting in and out of cars, driving,

00:12:37.059 --> 00:12:39.340
even just sitting for prolonged periods. Makes

00:12:39.340 --> 00:12:41.679
sense that position forces the joint together.

00:12:41.879 --> 00:12:44.600
Exactly. They might also report mechanical hip

00:12:44.600 --> 00:12:46.799
symptoms, things like clicking, catching, locking,

00:12:47.039 --> 00:12:49.799
or a popping sensation. That strongly suggests

00:12:49.799 --> 00:12:52.240
a labral tear is involved. And during the physical

00:12:52.240 --> 00:12:55.039
exam, what are you looking for? We look for restricted

00:12:55.039 --> 00:12:58.190
movement. Often there's limited hip flexion and

00:12:58.190 --> 00:13:00.649
particularly limited internal rotation, especially

00:13:00.649 --> 00:13:03.450
when the hip is flexed to 90 degrees. Sometimes

00:13:03.450 --> 00:13:05.450
internal rotation is less than five degrees.

00:13:05.610 --> 00:13:09.070
Wow, very restricted. Yes, and the key provocative

00:13:09.070 --> 00:13:12.769
test is the anterior impingement test or FEI

00:13:12.769 --> 00:13:15.870
or test. We gently flex the hip to 90 degrees.

00:13:15.980 --> 00:13:18.059
bring it across the body slightly, adduction,

00:13:18.480 --> 00:13:20.580
and then rotate it inwards. And if that hurts?

00:13:20.720 --> 00:13:22.820
If that reproduces their specific pain, it's

00:13:22.820 --> 00:13:25.820
a positive test. It's highly sensitive, positive

00:13:25.820 --> 00:13:29.279
in about 88 % of patients with FAI. It really

00:13:29.279 --> 00:13:32.100
hones in on that specific conflict zone. OK.

00:13:32.620 --> 00:13:34.799
So history and exam give a strong suspicion.

00:13:35.200 --> 00:13:37.759
Then comes imaging, presumably, to confirm it

00:13:37.759 --> 00:13:40.159
and see the extent of the problem. Absolutely.

00:13:40.720 --> 00:13:42.659
Imaging is essential to confirm the bony shapes

00:13:42.659 --> 00:13:45.360
and, critically, to assess the state of the labrum

00:13:45.360 --> 00:13:47.960
and cartilage. It completes the picture and guides

00:13:47.960 --> 00:13:50.100
treatment. What imaging do you start with? We

00:13:50.100 --> 00:13:52.139
always begin with radiographs, standard x -rays.

00:13:52.759 --> 00:13:54.860
A specific set of views is needed. a standing

00:13:54.860 --> 00:13:57.279
AP pelvis view, an AP view of the affected hip,

00:13:57.419 --> 00:13:59.480
and a good lateral view of the hip. What do these

00:13:59.480 --> 00:14:01.379
show? They allow us to assess the bone structure,

00:14:01.740 --> 00:14:04.000
looking for those cam and pincer lesions. We

00:14:04.000 --> 00:14:06.820
look for the pistol grip deformity for cam, signs

00:14:06.820 --> 00:14:08.799
of over coverage, like the crossover sign for

00:14:08.799 --> 00:14:12.340
pincer, or maybe a deep socket protrusio. And

00:14:12.340 --> 00:14:14.639
you measure things on the x -rays? Yes. Precise

00:14:14.639 --> 00:14:17.320
measurements are key. The alpha angle, usually

00:14:17.320 --> 00:14:19.379
measured on a special lateral view, like a dun

00:14:19.379 --> 00:14:22.720
or frog leg view, quantifies the cam deformity.

00:14:23.049 --> 00:14:26.210
An angle over 50 -55 degrees is generally considered

00:14:26.210 --> 00:14:29.110
abnormal. We also measure acetabular coverage

00:14:29.110 --> 00:14:31.610
with things like the lateral center edge angle.

00:14:32.110 --> 00:14:34.809
An angle below 25 degrees might suggest dysplasia.

00:14:35.039 --> 00:14:37.120
not enough coverage, which is different from

00:14:37.120 --> 00:14:40.019
FAIs over coverage. Different views like the

00:14:40.019 --> 00:14:42.139
done view are best for seeing the cam lesion,

00:14:42.340 --> 00:14:44.759
while a false profile view helps assess coverage

00:14:44.759 --> 00:14:47.159
at the front. So x -rays give the bony blueprint.

00:14:47.419 --> 00:14:50.500
What about CT scans? CT scans can be a useful

00:14:50.500 --> 00:14:53.000
adjunct. They give incredibly detailed views

00:14:53.000 --> 00:14:55.279
of the structural abnormalities and allow for

00:14:55.279 --> 00:14:58.340
amazing 3D reconstructions. These are fantastic

00:14:58.340 --> 00:15:00.059
for preoperative planning, letting the surgeon

00:15:00.059 --> 00:15:01.860
really visualize the shape and plan the bone

00:15:01.860 --> 00:15:04.590
resection. The downside is the higher radiation

00:15:04.590 --> 00:15:07.690
dose. Right. And for the soft tissues, the labrum

00:15:07.690 --> 00:15:11.429
and cartilage. For that, MRI is the gold standard.

00:15:11.929 --> 00:15:14.470
Specifically, MR arthrography is often preferred.

00:15:15.049 --> 00:15:17.129
Arthrography. That involves an injection, doesn't

00:15:17.129 --> 00:15:20.250
it? Yes, exactly. An intraarticular contrast

00:15:20.250 --> 00:15:22.690
injection, a special dye, is put into the hip

00:15:22.690 --> 00:15:25.669
joint just before the MRI scan. This dye leaks

00:15:25.669 --> 00:15:28.309
into any tears or defects in the labrum or cartilage,

00:15:28.669 --> 00:15:31.490
making them show up much more clearly. It's the

00:15:31.490 --> 00:15:34.289
best modality to evaluate for articular cartilage

00:15:34.289 --> 00:15:37.250
and labral damage. It makes sense. MRI also shows

00:15:37.250 --> 00:15:40.110
the bone shape well, without radiation, and it's

00:15:40.110 --> 00:15:42.070
excellent for ruling out other causes of hip

00:15:42.070 --> 00:15:45.970
pain that might mimic FAI. like a vascular necrosis,

00:15:46.230 --> 00:15:48.669
stress fractures, or various types of tendonitis.

00:15:48.929 --> 00:15:51.330
So MRI is really comprehensive. Is there anything

00:15:51.330 --> 00:15:54.149
else diagnostically? One other tool is a diagnostic

00:15:54.149 --> 00:15:56.570
intraarticular injection of local anesthetic.

00:15:56.970 --> 00:15:59.470
We numb the hip joint directly. If the patient's

00:15:59.470 --> 00:16:01.830
pain significantly improves, even temporarily,

00:16:02.309 --> 00:16:04.269
it strongly confirms the pain is coming from

00:16:04.269 --> 00:16:06.850
inside the joint, likely the FAI. It can be very

00:16:06.850 --> 00:16:08.490
helpful when the picture isn't perfectly clear.

00:16:08.620 --> 00:16:10.840
That's a really thorough diagnostic process.

00:16:11.259 --> 00:16:14.580
So once FAI is confirmed, surgery isn't automatically

00:16:14.580 --> 00:16:17.120
the next step, is it? What about conservative

00:16:17.120 --> 00:16:19.759
approaches? No, absolutely not. Non -surgical

00:16:19.759 --> 00:16:21.940
management is always the first port of call,

00:16:22.259 --> 00:16:25.700
especially for patients who are minimally symptomatic

00:16:25.700 --> 00:16:28.299
or don't have those definite mechanical symptoms

00:16:28.299 --> 00:16:30.700
like locking. What does that involve? It's usually

00:16:30.700 --> 00:16:34.659
a combination approach. Firstly, activity modification.

00:16:34.960 --> 00:16:37.960
This means identifying and reducing or temporarily

00:16:37.960 --> 00:16:41.100
stopping activities that provoke the pain. Not

00:16:41.100 --> 00:16:43.379
stopping everything, but being smarter about

00:16:43.379 --> 00:16:46.360
movement. Avoiding the triggers. Exactly. Secondly,

00:16:46.539 --> 00:16:49.399
and crucially, targeted physical therapy. A good

00:16:49.399 --> 00:16:52.080
physio will assess movement patterns, look for

00:16:52.080 --> 00:16:54.220
weaknesses or imbalances in the kinetic chain,

00:16:54.740 --> 00:16:56.980
and work on improving hip range of motion and

00:16:56.980 --> 00:16:59.399
strengthening the surrounding muscles, glutes,

00:16:59.519 --> 00:17:01.980
core muscles. To support the joint better. Precisely.

00:17:02.080 --> 00:17:04.660
Better control, better mechanics can sometimes

00:17:04.660 --> 00:17:07.940
reduce the stress on the impingement zone. Thirdly,

00:17:08.119 --> 00:17:10.779
pain management, mainly with NSAID's non -steroidal

00:17:10.779 --> 00:17:13.539
anti -inflammatory drugs to reduce pain and inflammation.

00:17:13.819 --> 00:17:16.740
Like ibuprofen. Yes. And as we mentioned that,

00:17:17.019 --> 00:17:19.299
intraarticular injection of local anesthetic

00:17:19.299 --> 00:17:22.160
and steroid can be used therapeutically too.

00:17:22.619 --> 00:17:25.059
The steroid reduces inflammation inside the joint,

00:17:25.599 --> 00:17:27.559
providing pain relief that can last weeks or

00:17:27.559 --> 00:17:30.519
months, making physio more effective. But these

00:17:30.519 --> 00:17:33.079
don't fix the underlying shape, do they? No,

00:17:33.240 --> 00:17:35.660
and that's the critical limitation. Conservative

00:17:35.660 --> 00:17:38.099
measures manage the symptoms, they manage the

00:17:38.099 --> 00:17:40.539
inflammation, they might improve mechanics around

00:17:40.539 --> 00:17:43.019
the problem, but they don't change the bone shape.

00:17:43.279 --> 00:17:45.859
They don't stop the collision happening. So if

00:17:45.859 --> 00:17:49.240
these don't provide... lasting relief. Then surgery

00:17:49.240 --> 00:17:52.140
becomes the next consideration. If symptoms persist

00:17:52.140 --> 00:17:54.180
despite a good trial of conservative management,

00:17:54.339 --> 00:17:56.900
usually three to six months, or if mechanical

00:17:56.900 --> 00:17:59.119
symptoms are prominent, then we discuss addressing

00:17:59.119 --> 00:18:01.779
the underlying anatomy directly. Right. So tell

00:18:01.779 --> 00:18:03.740
us about the surgical options. What's the aim?

00:18:04.259 --> 00:18:07.519
The aim of surgery is to reshape the bones, the

00:18:07.519 --> 00:18:10.160
femur, the acetabulum, or both, to eliminate

00:18:10.160 --> 00:18:13.059
the abnormal contact, the impingement, basically

00:18:13.059 --> 00:18:15.900
to restore normal hip mechanics and allow smooth,

00:18:16.119 --> 00:18:18.410
pain -free movement. They're two main approaches.

00:18:18.690 --> 00:18:21.109
The most common approach now is arthroscopic

00:18:21.109 --> 00:18:24.470
osteoplasty. This is minimally invasive, done

00:18:24.470 --> 00:18:27.349
through small keyhole incisions using a camera,

00:18:27.670 --> 00:18:29.509
the arthroscoge, and specialized instruments.

00:18:29.630 --> 00:18:32.569
Keyhole surgery for the hip. Exactly. The surgeon

00:18:32.569 --> 00:18:36.130
can see inside the joint on a screen and precisely

00:18:36.130 --> 00:18:39.509
trim the bony rim of the acetabulum, if it's

00:18:39.509 --> 00:18:42.869
a pincer lesion, or shave down the bump on the

00:18:42.869 --> 00:18:46.259
femoral head or neck for a cam lesion. That's

00:18:46.259 --> 00:18:48.460
the osteoplasty part reshaping the bone. And

00:18:48.460 --> 00:18:50.519
they can fix the labrum at the same time? Yes,

00:18:50.660 --> 00:18:52.940
a huge advantage is that arthroscopy allows the

00:18:52.940 --> 00:18:55.640
surgeon to thoroughly assess and repair or clean

00:18:55.640 --> 00:18:58.339
out any damage to the labrum and articular cartilage

00:18:58.339 --> 00:19:01.079
as well. Studies show it gives equivalent results

00:19:01.079 --> 00:19:03.339
to open hip surgery, but often with a quicker

00:19:03.339 --> 00:19:05.759
return to activity. That sounds promising. What's

00:19:05.759 --> 00:19:08.200
the open surgery option? The alternative is open

00:19:08.200 --> 00:19:10.640
surgical hip dislocation. This is the previous

00:19:10.640 --> 00:19:13.059
gold standard. It involves a larger incision

00:19:13.059 --> 00:19:15.380
and a technique where part of the hip bone, the

00:19:15.380 --> 00:19:17.960
greater trochanter, is temporarily detached to

00:19:17.960 --> 00:19:20.000
allow the surgeon to safely dislocate the hip

00:19:20.000 --> 00:19:22.619
joint and get a wide direct view of the femoral

00:19:22.619 --> 00:19:25.019
head and socket. Sounds quite major. It is a

00:19:25.019 --> 00:19:28.039
bigger operation, yes, but it's designed very

00:19:28.039 --> 00:19:31.720
carefully to preserve the blood supply to the

00:19:31.720 --> 00:19:35.019
femoral head. So the risk of a vascular necrosis

00:19:35.019 --> 00:19:39.079
or AVN is very low. It provides excellent exposure,

00:19:39.380 --> 00:19:42.119
which can be really useful for significant femoral

00:19:42.119 --> 00:19:45.500
deformity or complex cases. Return to play rates

00:19:45.500 --> 00:19:48.359
for athletes after open surgery are historically

00:19:48.359 --> 00:19:51.680
very high, over 85 -90%, provided they don't

00:19:51.680 --> 00:19:54.039
already have significant arthritis. Okay, and

00:19:54.039 --> 00:19:55.740
regardless of the approach, dealing with the

00:19:55.740 --> 00:19:57.680
labrum is key, you said. Absolutely critical.

00:19:57.819 --> 00:20:00.279
If there's a labral tear, which there usually

00:20:00.279 --> 00:20:03.180
is in symptomatic FAI, it needs to be addressed

00:20:03.180 --> 00:20:05.720
repaired or sometimes reconstructed at the same

00:20:05.720 --> 00:20:08.259
time as the bone is reshaped. Just cleaning out

00:20:08.259 --> 00:20:10.440
the labral tear, isolated labral debridement,

00:20:10.720 --> 00:20:12.940
without fixing the underlying bony impingement

00:20:12.940 --> 00:20:15.059
simply doesn't work long -term. Because the cause

00:20:15.059 --> 00:20:17.299
is still there? Exactly. It's a major cause of

00:20:17.299 --> 00:20:19.200
treatment failures. You have to address both

00:20:19.200 --> 00:20:21.339
the bone shape and the soft tissue damage it's

00:20:21.339 --> 00:20:23.599
caused. Right. Are there other surgeries sometimes

00:20:23.599 --> 00:20:26.940
needed alongside? Occasionally, yes. If there's

00:20:26.940 --> 00:20:30.299
significant hip dysplasia or etabular retroversion,

00:20:30.759 --> 00:20:33.839
a more complex operation called a periocetabular

00:20:33.839 --> 00:20:36.720
osteotomy might be needed to reorient the socket.

00:20:36.940 --> 00:20:39.000
And if the joint is already badly worn out with

00:20:39.000 --> 00:20:42.039
end -stage arthritis, then hip arthroplasty,

00:20:42.319 --> 00:20:44.559
a total hip replacement, becomes the necessary

00:20:44.559 --> 00:20:46.839
solution. It really depends on the individual

00:20:46.839 --> 00:20:50.420
case. Okay. Now, surgery always carries risks.

00:20:50.619 --> 00:20:52.579
What are the potential complications people should

00:20:52.579 --> 00:20:55.940
be aware of with FAI surgery? And what's a realistic,

00:20:55.940 --> 00:20:59.079
long -term outlook? That's a crucial conversation

00:20:59.079 --> 00:21:01.480
to have with patients. While generally safe and

00:21:01.480 --> 00:21:03.599
effective, complications can occur. What are

00:21:03.599 --> 00:21:05.740
the main ones? Major, though thankfully rare,

00:21:06.019 --> 00:21:07.859
complications include things like femoral neck

00:21:07.859 --> 00:21:09.900
fracture, particularly during the bone reshaping

00:21:09.900 --> 00:21:12.400
for a cam lesion. Surgeons are very careful to

00:21:12.400 --> 00:21:14.599
limit how much bone they remove, generally less

00:21:14.599 --> 00:21:17.920
than 30 % of the femoral neck diameter, to minimize

00:21:17.920 --> 00:21:20.920
this risk. With arthroscopy, very rare risks

00:21:20.920 --> 00:21:23.880
include things like abdominal compartment syndrome

00:21:23.880 --> 00:21:26.559
from fluid leaking where it shouldn't, or nerve

00:21:26.559 --> 00:21:29.099
injury. Other potential major issues, though

00:21:29.099 --> 00:21:33.440
uncommon, are blood clots, DVTPE, deep infection,

00:21:33.880 --> 00:21:37.279
or dislocation after surgery. A vascular necrosis

00:21:37.279 --> 00:21:39.619
is a risk, though minimized by careful technique.

00:21:39.700 --> 00:21:42.480
And minor complications. These are more common,

00:21:42.539 --> 00:21:44.910
but usually manageable. Things like heterotopic

00:21:44.910 --> 00:21:47.329
ossification where extra bone forms in the soft

00:21:47.329 --> 00:21:50.789
tissues afterwards. We often give endocides like

00:21:50.789 --> 00:21:52.990
endomethacin post -operatively to help prevent

00:21:52.990 --> 00:21:56.230
this. Temporary nerve irritation or praxious

00:21:56.230 --> 00:21:58.970
causing numbness maybe in the thigh or perineal

00:21:58.970 --> 00:22:01.650
area from traction during arthroscopy can happen

00:22:01.650 --> 00:22:04.309
but usually resolves. Superficial infection or

00:22:04.309 --> 00:22:06.849
bruising are also possible. Okay and the long

00:22:06.849 --> 00:22:10.359
-term prognosis. Does surgery stop arthritis?

00:22:10.700 --> 00:22:12.299
That's the million dollar question, isn't it?

00:22:12.440 --> 00:22:14.700
We know the natural history of untreated FAI

00:22:14.700 --> 00:22:17.500
likely leads to early onset hip dysfunction and

00:22:17.500 --> 00:22:19.720
arthritis. Surgery definitely needs to change

00:22:19.720 --> 00:22:21.640
that course. It's very successful at reducing

00:22:21.640 --> 00:22:23.839
symptoms and improving function in the short

00:22:23.839 --> 00:22:26.619
to medium term, and it can prevent future damage

00:22:26.619 --> 00:22:29.619
by stopping the impingement. But... The current

00:22:29.619 --> 00:22:32.420
scientific evidence is still unclear whether

00:22:32.420 --> 00:22:35.779
it will definitively delay or prevent the development

00:22:35.779 --> 00:22:38.759
of hip arthritis in the very long run, say 15,

00:22:38.779 --> 00:22:41.039
20 years or more. So it might still happen later.

00:22:41.359 --> 00:22:43.480
It might. Patients need to understand that while

00:22:43.480 --> 00:22:45.700
surgery aims for joint preservation and symptom

00:22:45.700 --> 00:22:48.460
relief, they may still develop arthritis in the

00:22:48.460 --> 00:22:50.960
future and potentially need a hip replacement

00:22:50.960 --> 00:22:53.980
eventually. The goal is to push that possibility

00:22:53.980 --> 00:22:56.750
as far down the road as possible. or lessen its

00:22:56.750 --> 00:22:59.109
severity. That's important expectation setting.

00:22:59.650 --> 00:23:02.069
And after surgery, rehab is vital, I imagine.

00:23:02.230 --> 00:23:04.910
Absolutely essential. Post -operative rehabilitation

00:23:04.910 --> 00:23:07.730
is just as important as the surgery itself. It's

00:23:07.730 --> 00:23:10.970
a structured, progressive process guided by physical

00:23:10.970 --> 00:23:13.170
therapists. What does it involve? It starts with

00:23:13.170 --> 00:23:15.130
a period of protected weight -bearing, using

00:23:15.130 --> 00:23:17.440
crutches for maybe... two to six weeks depending

00:23:17.440 --> 00:23:19.940
on the surgery. Early passive range of motion

00:23:19.940 --> 00:23:21.759
starts almost immediately to stop stiffness.

00:23:22.160 --> 00:23:24.460
Then comes active range of motion followed by

00:23:24.460 --> 00:23:26.680
intensive strengthening exercises for the hip

00:23:26.680 --> 00:23:29.759
and core and gait training to normalize walking.

00:23:29.880 --> 00:23:32.400
Getting back to sport. That comes later usually

00:23:32.400 --> 00:23:35.460
after 12 weeks or so with gradual reintroduction

00:23:35.460 --> 00:23:39.279
of jogging, jumping and agility exercises. It's

00:23:39.279 --> 00:23:41.720
a careful progression tailored to the individual.

00:23:42.059 --> 00:23:45.359
The physio's role is absolutely critical in guiding

00:23:45.359 --> 00:23:48.019
this safely. It sounds like a long process. It

00:23:48.019 --> 00:23:49.799
requires patience and commitment, definitely.

00:23:50.400 --> 00:23:52.980
Finally, looking ahead, what's new in the world

00:23:52.980 --> 00:23:55.920
of FAI? Where is the research heading? It's a

00:23:55.920 --> 00:23:58.519
very active field. The number of scientific publications

00:23:58.519 --> 00:24:01.460
on FAI has exploded in the last 15 -20 years.

00:24:02.039 --> 00:24:04.519
We're seeing advancements like computer -based

00:24:04.519 --> 00:24:07.220
navigation systems being developed to help surgeons

00:24:07.220 --> 00:24:09.460
perform the bone resection even more accurately

00:24:09.460 --> 00:24:12.309
during arthroscopy. Improving precision. Exactly.

00:24:12.809 --> 00:24:14.650
But there's also an acknowledged need for more

00:24:14.650 --> 00:24:16.829
high quality research. We need more mechanistic

00:24:16.829 --> 00:24:19.369
studies to really understand why the damage occurs

00:24:19.369 --> 00:24:21.690
at a cellular level. And critically, we need

00:24:21.690 --> 00:24:24.250
more long -term outcome data studies following

00:24:24.250 --> 00:24:27.009
patients for many, many years to truly understand

00:24:27.009 --> 00:24:30.329
the natural course of FAI and the real long -term

00:24:30.329 --> 00:24:32.789
impact of non -surgical and surgical treatment

00:24:32.789 --> 00:24:36.450
on preventing arthritis. So still learning? Always

00:24:36.450 --> 00:24:39.539
learning, yes. but we've come a very long way

00:24:39.539 --> 00:24:41.460
in understanding and treating it effectively.

00:24:41.599 --> 00:24:43.619
This has been incredibly informative. It really

00:24:43.619 --> 00:24:46.599
drives home how complex hip pain can be stemming

00:24:46.599 --> 00:24:49.339
from these underlying mechanical issues. Just

00:24:49.339 --> 00:24:51.420
to summarize the key takeaways for everyone listening.

00:24:51.720 --> 00:24:54.650
Firstly, FAI isn't just pain, it's a mechanical

00:24:54.650 --> 00:24:57.650
clash in the hip caused by abnormal bone shapes,

00:24:58.029 --> 00:25:01.109
cam, pincer, or usually combined. Bone hitting

00:25:01.109 --> 00:25:03.730
bone where it shouldn't. Secondly, it's a clear

00:25:03.730 --> 00:25:06.529
risk factor for developing early hip osteoarthritis,

00:25:06.730 --> 00:25:09.529
especially in active people whose movements can

00:25:09.529 --> 00:25:12.119
accelerate the wear and tear. Correct. Thirdly,

00:25:12.420 --> 00:25:14.700
diagnosis hinges on specific clinical tests,

00:25:15.200 --> 00:25:18.000
like that FADR impingement test, combined with

00:25:18.000 --> 00:25:20.900
imaging x -rays for the bone shapes, MRI, often

00:25:20.900 --> 00:25:23.339
with dye to see the laboring and cartilage damage.

00:25:24.039 --> 00:25:26.099
Yes. Fourth, treatment starts conservatively

00:25:26.099 --> 00:25:28.920
with activity changes and physio, but precise

00:25:28.920 --> 00:25:32.400
surgery, often keyhole arthroscopy, can reshape

00:25:32.400 --> 00:25:34.740
the bones and repair soft tissues to fix the

00:25:34.740 --> 00:25:37.369
mechanical problem. That's the pathway. And finally,

00:25:37.589 --> 00:25:39.549
while surgery is good for symptoms and function

00:25:39.549 --> 00:25:41.789
now and can prevent further immediate damage,

00:25:42.250 --> 00:25:45.029
its ability to completely stop arthritis developing

00:25:45.029 --> 00:25:48.029
decades later is still being studied. It's about

00:25:48.029 --> 00:25:50.730
long -term joint preservation. Well summarized.

00:25:51.410 --> 00:25:53.569
If you found this deep dive helpful, we'd really

00:25:53.569 --> 00:25:55.509
appreciate it if you could take a second to rate

00:25:55.509 --> 00:25:57.470
the show or share it with someone who might find

00:25:57.470 --> 00:25:59.210
it useful, maybe someone you know dealing with

00:25:59.210 --> 00:26:01.910
persistent hip issues. And a final thought to

00:26:01.910 --> 00:26:04.849
leave you with. Just consider how these seemingly

00:26:04.849 --> 00:26:07.190
small variations in our anatomy, things we might

00:26:07.190 --> 00:26:10.049
not even know we have, can have such a huge impact

00:26:10.049 --> 00:26:12.630
on our movement, our quality of life, and our

00:26:12.630 --> 00:26:15.309
long -term joint health. It really highlights

00:26:15.309 --> 00:26:17.089
the importance of listening to our bodies and

00:26:17.089 --> 00:26:19.509
seeking answers when things don't feel right.
