WEBVTT

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Welcome to the Deep Dive, where we cut through

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the information overload and give you the essential

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insights from a stack of sources. Think of it

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as getting the key knowledge without you having

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to sift through all the academic journals yourself.

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Our mission is to make you well -informed, quickly,

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with just enough detail to make it stick. Today,

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we are taking a deep dive into a shoulder injury

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that's surprisingly common, particularly among

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athletes who throw or do a lot of overhead work.

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But it also presents, well, a real puzzle for

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diagnosis and treatment. We're talking about

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superior labrum, anterior and posterior tears,

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or SLAP lesions. This isn't just some abstract

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medical term, it's something that sidelines athletes

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and causes persistent pain for many people. To

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understand it properly, we've pulled insights

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from peer -reviewed orthopedic literature, including

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articles from the Journal of the American Academy

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of Orthopedic Surgeons, JAAOS, and reliable foundational

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resources like OrthoBullets. It's a complex corner

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of orthopedics, isn't it? Filled with nuanced

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anatomy and... evolving treatment approaches

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and we're going to guide you through the most

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important bits. I've always been fascinated by

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SLAP tiers because they seem to be talked about

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constantly in sports medicine but figuring out

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what's really going on and how to best fix them.

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Well it seems to be a source of ongoing debate.

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Oh absolutely. They are a real challenge in clinical

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practice. The symptoms can overlap with other

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shoulder issues. You know imaging requires a

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discerning eye and as the sources highlight deciding

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on the optimal management strategy especially

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for different patient profiles. files involves

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navigating quite a few factors. There's no simple

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formula, really, which is why understanding the

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underlying anatomy and pathology is so critical.

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OK, let's unpack this then, starting with the

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absolute basics. We hear SLAP lesion. What exactly

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is this tear, and where is it located in the

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shoulder? Right. So at its most fundamental,

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a SLAP lesion is an injury to the superior labrum

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of the shoulder. The acronym SLAP stands for

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superior labrum anterior and posterior. This

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means the tear is specifically located in the

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top, the superior portion of the ring of cartilage

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surrounding the shoulder socket. And the crucial

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characteristic defining this SLAP tear is that

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this tear involves the superior labrum, and critically

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it extends from the front, the anterior, to the

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back, the posterior, encompassing the area where

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the long head of the biceps tendon attaches to

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the labrum. Ah, I see. So it's not just any tear

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of that cartilage ring. It's a specific tear

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in the upper part, right where the biceps hooks

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in. Precisely. Yeah, exactly. To fully appreciate

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this, let's maybe take a moment for a quick tour

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of the shoulder anatomy, as described in the

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sources. The shoulder is basically a ball and

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socket joint. You have the ball, which is the

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head of your humerus, your upper arm bone. Right.

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And it fits into a rather shallow socket on your

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shoulder blade called the glenoid. Now, because

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that glenoid socket is relatively flat, the labrum

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comes into play. And the labrum is sort of like

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a bumper or a seal? Yes. Think of the labrum

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as a rim or a bumper. That's a good way to put

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it. It's a ring of tough fibrocartilaginous tissue

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that attaches around the edge of the glenoid

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socket. Its main job is really to deepen that

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socket, making the shoulder joint inherently

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more stable. It increases the contact area between

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the humerus and the glenoid. It helps keep the

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ball centered in the socket. Beyond deepening

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the socket, the labrum also serves as an important

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attachment point for some of the key ligaments

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that provide stability to the shoulder joint.

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And, as we just mentioned, it's the attachment

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site for the long head of the biceps tendon.

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Right, let's zero in on that biceps connection

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then. It seems absolutely central to SLAP tears.

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How does the long head of the biceps attach there?

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Okay, the long head of the biceps tendon actually

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originates just above the shoulder socket at

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a point called the supraglenoid tubercle on the

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scapula. It then travels through the shoulder

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joint before blending with and attaching to the

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superior portion of the labrum. According to

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anatomical descriptions in the sources, the long

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head of the biceps tendon commonly has a sort

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of split attachment. Roughly 50 % of its fiber

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is attached directly to the superior labrum.

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And the other 50 % attached to that bony supraglinoid

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tubercle. The tubercle itself is located about

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six millimeters medial or towards the body's

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midline. from the actual articular surface of

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the glenoid. This point of attachment is generally

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located posterior to the 12 o 'clock position

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on the superior glenoid when you're looking at

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it arthroscopically. And you mentioned anatomical

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variations in how the biceps attaches. Does that

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add another layer of complexity? It absolutely

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does, yes. The way the biceps tendon attaches

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to the superior labrum isn't identical in everyone.

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There's this thing called the two -ahead classification

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system. referenced in the sources, describes

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some of these patterns. Type I is where the attachment

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is completely posterior to that 12 o 'clock position

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on the glenoid. Type II is predominantly posterior.

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Type III has fibers attaching both anteriorly

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and posteriorly. and type four is when the entire

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attachment is anterior to the 12 o 'clock position.

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The sources indicate that top eye and two attachments

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are by far the most common, accounting for over

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70 % of the anatomical variations observed. Understanding

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these normal variations is important because,

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well, they can influence how a tear presents

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or how a surgeon views the anatomy during surgery.

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Another relevant anatomical detail is that the

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long head of the biceps tendon, It actually has

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its porous blood supply. Precisely where it versus

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through the shoulder joint. This limited blood

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flow could potentially impact its ability to

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heal after injury. Limited blood flow. That sounds

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like a key piece of information. And what about

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the labrum itself? Does its blood supply vary?

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Yes, it does. The labrum receives its blood supply

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not from the underlying bone, but from the surrounding

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joint capsule and periosteal vessels. And critically,

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the anterior superior portion of the labrum seems

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to have the poorest blood supply compared to

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other parts of the rim. Couple this with the

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fact that the superior labrum attaches slightly

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further medially on the glenoid compared to the

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inferior labrum, making it a bit less firmly

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supported at its base, you could say. And it

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becomes clearer why this specific area where

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the biceps tendon anchors can be considered something

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of a weak link if prone to injury under certain

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forces, resulting in a SLAP tear. So the very

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anatomy of that superior labrum and biceps anchor

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area kind of predisposes it to tearing. And speaking

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of anatomy looking like tears, you mentioned

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those tricky anatomical variants that can sometimes

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fool even experienced eyes. That sounds like

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a major diagnostic hurdle. It's a very significant

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point, yes. particularly for surgeons looking

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inside the joint. There are two main anatomical

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variants in the superior labrum area that can

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easily be mistaken for a tear on imaging or during

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arthroscopy if you're not acutely aware of them.

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One is called the sublaboral recess or sublaboral

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foramen. This is a normal non -pathological separation

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or space between the superior labrum and the

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underlying glenoid bone. It's just how the anatomy

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is configured in some people. It can sometimes

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be associated with a thicker than usual middle

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glenohumeral ligament, which is another ligament

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in the shoulder. The key challenge is distinguishing

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this normal space from a true tear, where the

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labrum is detached and actually causing symptoms.

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On an MRI arthrogram, dye might enter this normal

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recess, which can be confusing. I can see that.

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But the pattern is usually distinct from dye

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entering a tear. It tends to be smoother. OK,

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and the second variant? The second crucial variant

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is the Buford complex. This is a specific anatomical

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arrangement where the middle glenohumeral ligament

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is very thick and cord -like, and it originates

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directly from the superior labrum. When this

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happens, there is a congenital absence, or a

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bare area, of the antero -superior labrum. It

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looks like a piece of the labrum is missing or

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torn anteriorly. But it's not. But it's actually

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a normal developmental variant present in a small

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percentage of the population. And why is recognizing

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the Buford complex so critical? What happens

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if a surgeon mistakes it for a tear and tries

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to fix that bare area? This is where the clinical

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pitfall lies. It's really important. If a surgeon

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doesn't recognize the Buford complex and mistakenly

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repair that bare, antro -superior area of the

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labrum to the glenoid, believing it's a tear,

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they will significantly restrict the patient's

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external rotation of the shoulder. Oh, wow. because

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that thick cord -like ligament is meant to be

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in that position, trying to anchor tissue there

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that isn't supposed to be just restricts normal

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shoulder movement. Recognizing these variants,

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the sublaboral recess, and especially the Buford

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complex, is absolutely paramount for accurate

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diagnosis and avoiding unnecessary surgery that

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can actually cause functional problems. That

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really emphasizes how understanding the normal

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variations is just as important as understanding

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the pathology. It's not just a simple case of,

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is there a tear or isn't there? Okay, so we know

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what SLAP lesion is anatomically and some of

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the quirks in that region. Who was most likely

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to get one of these tears and what typically

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causes them? Well, while SLAP lesions account

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for a relatively small percentage of all shoulder

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injuries, less than 5 % according to the sources.

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Less than 5%. They are disproportionately seen

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in specific groups. The classic patient is the

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overhead or throwing athlete. Often in their

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dominant arm think baseball pitchers, javelin

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throwers, volleyball or tennis players, that

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sort of thing. However, they can also occur in

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older patients, particularly as a result of a

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traumatic fall onto an outstretched arm. Are

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there specific risk factors or certain movements

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that are known to cause these tears? Yes, repetitive

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overhead activities are the prime suspect in

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athletes. The sheer volume and force of throwing

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or soothing motions put immense stress on that

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superior labrum and biceps anchor. Other common

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mechanisms of injury include falling directly

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onto an outstretched arm, especially if the arm

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is slightly bent and the biceps muscle is tensed

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at the time of impact. A sudden forceful pull

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on the arm, like trying to catch a heavy object

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unexpectedly, or rapid forceful movements of

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the arm above shoulder level can also generate

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enough force to tear the superior labrum. And

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interestingly, a shoulder dislocation can sometimes

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peel the labrum off the glenoid rim, occasionally

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including the superior portion involved in a

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SLAP tear. You mentioned a theory about throwers

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specifically linking tightness to the injury.

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Can you elaborate a bit on that biomechanics?

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Absolutely. This is where some of the deeper

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insight into the pathology comes from. There's

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a well regarded theory that in throwing athletes

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who often develop tightness in the posterior

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capsule and the posterior inferior glenohumeral

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ligament. Right, the back of the shoulder. Exactly.

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Which is a key stabilizing structure at the back

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and bottom of the shoulder. This tightness can

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alter the mechanics of the throwing motion. As

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the arm moves through the late cocking phase

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and into acceleration, this posterior tightness

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can cause the ball of the humerus to shift post

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-rosuperiorly upwards and backwards relative

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to the glenoid socket. This abnormal shift increases

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the sheer force applied to the superior labrum,

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effectively sort of grinding or peeling it away

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from the bone, especially where the biceps tend

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to detaches. Furthermore, the sources point out

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that once a SLAP lesion is present, this detached

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superior labrum and biceps anchor can then increase

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the strain on the anterior band of the inferior

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glenohumeral ligament. Which is the main stabilizer

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at the front. Precisely, the primary anterior

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stabilizer of the shoulder. This can lead to

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a sort of vicious cycle where the SLAP tear contributes

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to subtle instability, further disrupting normal

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mechanics. That's fascinating. So the injury

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itself can potentially worsen the underlying

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problem or contribute to instability. It's not

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just about the tear itself, but how the tear

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changes the shoulders function. And you noted

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that in older patients, it might not be from

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trauma, but just wear and tear. Correct, yeah.

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In individuals over the age of, say, 30 or 40,

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it's not uncommon to see some degree of fraying

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or degenerative changes in the superior labrum

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on MRI or during arthroscopy. This could just

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be a normal part of aging. and doesn't necessarily

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cause symptoms or require treatment unless there

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was a specific traumatic event superimposed on

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these degenerative changes. Right. Differentiating

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between a symptomatic tear and just age -related

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fraying is another critical diagnostic step.

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It's not always easy. And are SLAP tears usually

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isolated injuries or do they often come with

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other problems in the shoulder joint? They frequently

00:12:22.870 --> 00:12:25.870
have company, yes. SLAP lesions are often associated

00:12:25.870 --> 00:12:27.330
with other shoulder conditions, particularly

00:12:27.330 --> 00:12:29.389
in throwing athletes. These can include things

00:12:29.389 --> 00:12:31.710
like internal impingement. Or the cuff gets pinched.

00:12:31.830 --> 00:12:34.070
Yeah, where the rotator cuff tendon gets pinched

00:12:34.070 --> 00:12:37.230
in certain overhead positions. Also glenohumeral

00:12:37.230 --> 00:12:40.350
internal rotation deficit, or GIRD, which is

00:12:40.350 --> 00:12:42.929
that posterior tightness we discussed. Rotator

00:12:42.929 --> 00:12:45.350
cuff tears are common too. Often partial thickness

00:12:45.350 --> 00:12:48.549
tears on the articular or joint side. Subtle

00:12:48.549 --> 00:12:50.809
shoulder instability and scapular dyskinesis

00:12:50.809 --> 00:12:52.950
that's abnormal movement or positioning of the

00:12:52.950 --> 00:12:55.850
shoulder blade during arm motion. Okay. Addressing

00:12:55.850 --> 00:12:58.190
these associated conditions is often really important

00:12:58.190 --> 00:13:00.450
for successful management, whether you go surgical

00:13:00.450 --> 00:13:03.710
or non -operative. Okay. We understand the anatomy,

00:13:03.789 --> 00:13:06.309
who gets them, and how they might happen. Now,

00:13:06.330 --> 00:13:08.950
for the detective work, how do doctors actually

00:13:08.950 --> 00:13:11.759
figure out if someone has a celipate tear? What

00:13:11.759 --> 00:13:14.019
does the patient feel and what does the examination

00:13:14.019 --> 00:13:16.740
involve? Right. Well, from the patient's perspective,

00:13:17.340 --> 00:13:19.620
the symptoms of a cell A pay tear can be somewhat

00:13:19.620 --> 00:13:22.340
inconsistent and often mimic other shoulder problems,

00:13:22.559 --> 00:13:24.059
which, as we said, is part of the diagnostic

00:13:24.059 --> 00:13:27.259
challenge. Common complaints include a deep aching

00:13:27.259 --> 00:13:29.139
pain within the shoulder that's often difficult

00:13:29.139 --> 00:13:31.919
to localize precisely. People struggle to point

00:13:31.919 --> 00:13:34.549
exactly where it hurts. A key symptom is often

00:13:34.549 --> 00:13:37.230
mechanical people describe hearing or feeling

00:13:37.230 --> 00:13:40.789
popping, clicking, catching, or grinding sensations

00:13:40.789 --> 00:13:43.399
in the shoulder with certain movements. Pain

00:13:43.399 --> 00:13:45.799
is typically aggravated by specific positions,

00:13:46.360 --> 00:13:48.299
especially those involving overhead activities

00:13:48.299 --> 00:13:52.100
or when lifting heavy objects. Athletes, particularly

00:13:52.100 --> 00:13:54.519
throwers, might notice a significant decrease

00:13:54.519 --> 00:13:56.620
in their performance, maybe loss of velocity

00:13:56.620 --> 00:13:59.500
or control, feeling weakness, or experiencing

00:13:59.500 --> 00:14:01.679
what's sometimes called a dead arm sensation

00:14:01.679 --> 00:14:04.519
after throwing. The dead arm, yeah. Some patients

00:14:04.519 --> 00:14:06.899
also report a feeling of apprehension, or like

00:14:06.899 --> 00:14:09.500
the shoulder might pop out. And studies suggest

00:14:09.500 --> 00:14:11.740
apprehension tests are actually positive in a

00:14:11.740 --> 00:14:15.179
high percentage, around 85 % of patients with

00:14:15.179 --> 00:14:19.259
SLAP tears. 85%, that's quite high. It is. Reduced

00:14:19.259 --> 00:14:21.820
range of motion or easy fatigue ability can also

00:14:21.820 --> 00:14:24.419
occur. And it's worth noting that sometimes symptoms

00:14:24.419 --> 00:14:26.679
might not fully manifest immediately after the

00:14:26.679 --> 00:14:29.200
injury, but rather develop gradually over time.

00:14:29.480 --> 00:14:31.620
So the symptoms are broad and can sound like

00:14:31.620 --> 00:14:33.620
other things. How does a doctor approach trying

00:14:33.620 --> 00:14:36.519
to pin down if it's actually a SLI peer? Well,

00:14:36.600 --> 00:14:39.340
the diagnostic process begins, as with any injury

00:14:39.340 --> 00:14:42.240
really, with a detailed medical history. The

00:14:42.240 --> 00:14:44.139
doctor will ask about the onset of the pain,

00:14:44.259 --> 00:14:46.340
whether there's a specific injury or if it came

00:14:46.340 --> 00:14:48.860
on gradually. What activities make it worse?

00:14:49.129 --> 00:14:52.690
especially work or sports, the exact location

00:14:52.690 --> 00:14:55.950
and character of the pain, and any previous treatments

00:14:55.950 --> 00:14:58.649
they might have tried. The patient's age and

00:14:58.649 --> 00:15:01.330
activity level are also critical pieces of information.

00:15:02.789 --> 00:15:05.230
Following the history, a thorough physical examination

00:15:05.230 --> 00:15:08.389
is performed. This involves assessing the shoulder's

00:15:08.389 --> 00:15:10.850
range of motion in different planes, evaluating

00:15:10.850 --> 00:15:13.470
muscle strength, and testing the overall stability

00:15:13.470 --> 00:15:16.019
of the joint. A key component of the physical

00:15:16.019 --> 00:15:19.179
exam for suspected SLA key tears involves performing

00:15:19.179 --> 00:15:21.379
what are called provocative tests. Provocative

00:15:21.379 --> 00:15:23.799
tests. These are specific maneuvers where the

00:15:23.799 --> 00:15:26.039
doctor moves the patient's arm into positions

00:15:26.039 --> 00:15:28.000
designed to put stress on the superior labrum

00:15:28.000 --> 00:15:30.179
and biceps anchor to see if they can reproduce

00:15:30.179 --> 00:15:32.399
the patient's pain or mechanical symptoms. Are

00:15:32.399 --> 00:15:34.799
these provocative tests definitive? Like, if

00:15:34.799 --> 00:15:36.940
one test is positive, you know for sure it's

00:15:36.940 --> 00:15:40.220
a SLA P tear. Ah, this is a critical point from

00:15:40.220 --> 00:15:43.059
the sources and really highlights the diagnostic

00:15:43.059 --> 00:15:47.340
difficulty. There is no single physical examination

00:15:47.340 --> 00:15:50.539
test that is perfectly sensitive or specific

00:15:50.539 --> 00:15:54.139
for diagnosing a SLAP lesion on its own. Right.

00:15:54.399 --> 00:15:58.440
No silver bullet test. Exactly. A diagnosis is

00:15:58.440 --> 00:16:00.779
based on the combination of the patient's history,

00:16:01.259 --> 00:16:03.720
their reported symptoms, and the findings from

00:16:03.720 --> 00:16:06.460
multiple provocative tests, along with of course

00:16:06.460 --> 00:16:08.279
the imaging results. It's putting all the pieces

00:16:08.279 --> 00:16:10.429
together. Can you walk us through a couple of

00:16:10.429 --> 00:16:13.250
these provocative tests and maybe explain why

00:16:13.250 --> 00:16:16.370
they might reproduce symptoms if a SLIP is present?

00:16:16.750 --> 00:16:19.309
What's the thinking behind them? Certainly. The

00:16:19.309 --> 00:16:21.710
sources list several. Some are general biceps

00:16:21.710 --> 00:16:24.649
provocation tests like Speed's test or Jurgesson's

00:16:24.649 --> 00:16:26.269
test, though these can be positive with other

00:16:26.269 --> 00:16:28.269
biceps issues too, so they're not super specific.

00:16:28.889 --> 00:16:31.769
More specific SLAP tests include the active compression

00:16:31.769 --> 00:16:34.029
test, often known as O 'Brien's test. O 'Brien's

00:16:34.029 --> 00:16:35.289
test, yeah, I've heard of that one. The crank

00:16:35.289 --> 00:16:37.309
test, the dynamic labral shear test, and the

00:16:37.309 --> 00:16:39.029
Kibler anterior slide test. There are quite a

00:16:39.029 --> 00:16:41.549
few. Let's look at the O 'Brien's test as it's

00:16:41.549 --> 00:16:44.440
commonly used. The patient flexes their arm forward

00:16:44.440 --> 00:16:47.419
to 90 degrees, adducts it about 10, 15 degrees

00:16:47.419 --> 00:16:50.139
across the body, and internally rotates it so

00:16:50.139 --> 00:16:53.240
the thumb points down. Right, thumb down, arm

00:16:53.240 --> 00:16:56.279
across. Exactly. The doctor then applies downward

00:16:56.279 --> 00:16:59.840
resistance, and the patient resists. This position,

00:17:00.200 --> 00:17:02.559
with the arm slightly across the body and internally

00:17:02.559 --> 00:17:05.319
rotated, is thought to put a twisting or torsional

00:17:05.319 --> 00:17:08.099
stress on the superior labrum and biceps anchor.

00:17:08.859 --> 00:17:11.480
Then the arm is externally rotated, so the palm

00:17:11.480 --> 00:17:14.230
is up, and the resistance is applied again. A

00:17:14.230 --> 00:17:16.509
positive test is if the pain or clicking experience

00:17:16.509 --> 00:17:19.049
with the thumb down position is reduced or eliminated

00:17:19.049 --> 00:17:21.430
when the palm is up, particularly if the pain

00:17:21.430 --> 00:17:24.210
is felt deep inside the shoulder. The idea is

00:17:24.210 --> 00:17:26.269
that with the thumb down, the superior labrum

00:17:26.269 --> 00:17:28.750
is being loaded and perhaps peeled away slightly,

00:17:29.190 --> 00:17:31.490
while with the palm up, that stress is relieved.

00:17:31.650 --> 00:17:33.589
I see. And the crank test, how does that one

00:17:33.589 --> 00:17:35.569
work? For the crank test, the arm is abducted,

00:17:35.730 --> 00:17:38.269
so lift it away from the body to about 120 degrees.

00:17:38.730 --> 00:17:41.509
The doctor applies an axial load, basically pushing

00:17:41.509 --> 00:17:43.609
the humerus head into the socket gently, and

00:17:43.609 --> 00:17:45.910
then rotates the arm internally and externally.

00:17:46.410 --> 00:17:48.829
This maneuver is designed to try and catch or

00:17:48.829 --> 00:17:51.970
pinch a torn labral flap between the humerus

00:17:51.970 --> 00:17:54.549
head and the glenoid. It's trying to reproduce

00:17:54.549 --> 00:17:56.849
those catching or clicking mechanical symptoms.

00:17:57.450 --> 00:18:00.250
The presence of pain or a click, especially during

00:18:00.250 --> 00:18:02.950
external rotation with that axial load, can suggest

00:18:02.950 --> 00:18:06.190
a superior label tear. Right. But again, it's

00:18:06.190 --> 00:18:08.970
crucial to remember a positive result on any

00:18:08.970 --> 00:18:11.710
single test isn't proof positive. It just raises

00:18:11.710 --> 00:18:13.650
suspicion that needs to be confirmed by other

00:18:13.650 --> 00:18:16.329
means like imaging. That makes sense. You're

00:18:16.329 --> 00:18:19.230
essentially trying to manually stress that specific

00:18:19.230 --> 00:18:21.789
area where the tear might be to see if you can

00:18:21.789 --> 00:18:24.609
reproduce the patient's symptoms. Given that

00:18:24.609 --> 00:18:27.109
physical tests aren't definitive, imaging must

00:18:27.109 --> 00:18:29.980
play a really crucial role then. What do they

00:18:29.980 --> 00:18:32.359
use and how good is it at actually seeing these

00:18:32.359 --> 00:18:35.119
tears? You're right. Imaging is essential. Standard

00:18:35.119 --> 00:18:37.380
x -rays are usually the first step, but they

00:18:37.380 --> 00:18:40.000
primarily show bone, not soft tissues like the

00:18:40.000 --> 00:18:42.339
labrum. So they'll likely be normal. Exactly.

00:18:42.920 --> 00:18:45.259
X -rays in a patient with a cesolactare are typically

00:18:45.259 --> 00:18:47.680
normal. They're mostly useful for ruling out

00:18:47.680 --> 00:18:49.859
other potential causes of pain, like arthritis,

00:18:50.420 --> 00:18:54.200
bone spurs, or maybe a fracture. Okay. To visualize

00:18:54.200 --> 00:18:56.809
the labrum itself, Magnetic Resonance Imaging,

00:18:57.089 --> 00:19:00.569
or MRI, is the primary tool. On a standard MRI,

00:19:00.970 --> 00:19:03.890
a cell AP p -tear might appear as increased signal

00:19:03.890 --> 00:19:07.349
a brighter area on certain sequences, particularly

00:19:07.349 --> 00:19:09.930
T2 -weighted images between the superior labrum

00:19:09.930 --> 00:19:12.650
and the glenoid bone, usually posterior to where

00:19:12.650 --> 00:19:15.769
the biceps attaches. However, standard MRI sensitivity

00:19:15.769 --> 00:19:18.170
for detaching a cell AP p -tears is only around

00:19:18.170 --> 00:19:21.039
50%. 50%. That sounds pretty low, especially

00:19:21.039 --> 00:19:23.720
for such a key diagnostic tool. It is quite low.

00:19:23.799 --> 00:19:25.660
It means it misses about half of them, although

00:19:25.660 --> 00:19:28.279
its specificity is higher, around 90%. So if

00:19:28.279 --> 00:19:30.559
it sees something, it's likely real, but it misses

00:19:30.559 --> 00:19:32.599
a lot. So how do they improve its accuracy? There

00:19:32.599 --> 00:19:35.119
must be a better way. This is where an MRI arthrogram

00:19:35.119 --> 00:19:37.660
becomes invaluable. This involves injecting a

00:19:37.660 --> 00:19:40.460
contrast agent, basically a dye, directly into

00:19:40.460 --> 00:19:42.619
the shoulder joint before the MRI scan is performed.

00:19:42.779 --> 00:19:45.289
Ah, injecting dye into the joint first. Exactly.

00:19:45.609 --> 00:19:48.269
The dye fills the joint space. If there's a tear

00:19:48.269 --> 00:19:50.609
in the labrum... the dye will leak into that

00:19:50.609 --> 00:19:52.690
tear, highlighting the defect and making it much

00:19:52.690 --> 00:19:55.430
more visible on the scan. The sources are clear

00:19:55.430 --> 00:19:58.490
that an MRI arthrogram significantly increases

00:19:58.490 --> 00:20:01.349
the accuracy for diagnosing labral tears compared

00:20:01.349 --> 00:20:04.369
to a standard MRI. It's really the gold standard

00:20:04.369 --> 00:20:06.769
for imaging these. You can visually distinguish

00:20:06.769 --> 00:20:09.289
between, say, a normal sublabor recess where

00:20:09.289 --> 00:20:11.309
the dye might pool slightly in a smooth pattern

00:20:11.309 --> 00:20:14.190
and a true tear, where the dye seeps irregularly

00:20:14.190 --> 00:20:17.210
into the torn tissue or the defect. The presence

00:20:17.210 --> 00:20:20.180
of a paralabral gangly insist is another important

00:20:20.180 --> 00:20:22.880
imaging finding. These are fluid -filled sacs

00:20:22.880 --> 00:20:25.799
that often form adjacent to a labral tear, particularly

00:20:25.799 --> 00:20:28.200
in the spinoculnoid notch near the shoulder blade.

00:20:28.259 --> 00:20:31.019
Assist near the tear. Yes. And their presence

00:20:31.019 --> 00:20:33.859
on MRI is considered highly specific for an underlying

00:20:33.859 --> 00:20:36.660
labral tear, even if the tear itself is subtle

00:20:36.660 --> 00:20:39.539
or difficult to see directly on the scan. It's

00:20:39.539 --> 00:20:43.269
a strong secondary sign. The diagnosis is really

00:20:43.269 --> 00:20:45.609
a puzzle pieced together from the patient's story,

00:20:45.970 --> 00:20:48.289
the physical exam findings, even if they're not

00:20:48.289 --> 00:20:51.190
definitive individually, and then ideally, a

00:20:51.190 --> 00:20:53.670
specific type of imaging, like an MRI arthrogram.

00:20:54.349 --> 00:20:56.809
Once a tear is identified, the sources talk about

00:20:56.809 --> 00:21:00.250
classification systems. Why bother classifying

00:21:00.250 --> 00:21:02.930
these tears? What's the point? Classification

00:21:02.930 --> 00:21:05.230
systems are used mainly to standardize the description

00:21:05.230 --> 00:21:07.319
of the tear pattern. This helps in communication

00:21:07.319 --> 00:21:11.019
among clinicians, surgeons, physios, and importantly,

00:21:11.259 --> 00:21:13.180
it helps guide treatment decisions. Different

00:21:13.180 --> 00:21:14.940
types of tears might be managed differently.

00:21:15.680 --> 00:21:17.799
The most widely referenced system is the Snyder

00:21:17.799 --> 00:21:20.339
classification, which uniquely describes four

00:21:20.339 --> 00:21:22.940
main types based on the anatomical location and

00:21:22.940 --> 00:21:24.700
the involvement of the biceps tendon anchor.

00:21:25.140 --> 00:21:27.559
Could you briefly explain those Snyder types

00:21:27.559 --> 00:21:30.240
for us? Type I to IV? Certainly. Let's run through

00:21:30.240 --> 00:21:32.559
them. Type I. This is considered more of a degenerative

00:21:32.559 --> 00:21:35.279
tear. It involves fraying of the superior labrum

00:21:35.279 --> 00:21:37.819
and sometimes the biceps tendon itself, but the

00:21:37.819 --> 00:21:39.819
key thing is the biceps anchor remains firmly

00:21:39.819 --> 00:21:42.079
attached to the glenoid. These account for about

00:21:42.079 --> 00:21:45.700
11 % of SLAP lesions. Just fraying anchors okay.

00:21:45.819 --> 00:21:48.960
Got it. Type 2. This is the most common type,

00:21:49.059 --> 00:21:51.059
particularly in throwing athletes, making up

00:21:51.059 --> 00:21:54.240
around 41 % of cases. It involves fraying of

00:21:54.240 --> 00:21:57.279
the superior labrum and a detachment of the biceps

00:21:57.279 --> 00:22:00.380
tendon anchor from the superior glenoid. The

00:22:00.380 --> 00:22:02.319
labrum and biceps anchor are essentially peeled

00:22:02.319 --> 00:22:04.140
away from the bone. Okay, that's the detached

00:22:04.140 --> 00:22:06.680
anchor one. Exactly. Type 3. This is described

00:22:06.680 --> 00:22:09.039
as a bucket -handle tear of the superior labrum.

00:22:09.579 --> 00:22:11.900
A flap of the labrum tears and can displace into

00:22:11.900 --> 00:22:14.220
the joint, but critically, the biceps tendon

00:22:14.220 --> 00:22:16.920
anchor remains intact and attached to the glenoid.

00:22:17.519 --> 00:22:19.799
The biceps actually separates from the torn labral

00:22:19.799 --> 00:22:22.759
flap. These are about 33 % of cases. So the labrum

00:22:22.759 --> 00:22:25.299
tears, but the biceps anchor stays put. Correct.

00:22:25.440 --> 00:22:27.990
And type 4. This is also a bucket -handle tear,

00:22:28.089 --> 00:22:30.750
like type 3, but in this case, the tear extends

00:22:30.750 --> 00:22:33.430
into the biceps tendon itself, and the biceps

00:22:33.430 --> 00:22:35.789
tendon anchor is also detached and remains attached

00:22:35.789 --> 00:22:37.930
to the torn labral flap. These are less common,

00:22:38.049 --> 00:22:40.329
around 15%. Right, so the tear goes into the

00:22:40.329 --> 00:22:42.410
biceps tendon too, and the anchor comes off with

00:22:42.410 --> 00:22:44.089
it. Precisely. And there are more types added

00:22:44.089 --> 00:22:46.170
later. I think you mentioned MAFIT. Yes, that's

00:22:46.170 --> 00:22:49.309
right. The MAFIT classification and others expanded

00:22:49.309 --> 00:22:51.849
the system to include tears extending into other

00:22:51.849 --> 00:22:54.390
parts of the labrum, or associated with other

00:22:54.390 --> 00:22:57.750
ligamentous injuries. For example, type V is

00:22:57.750 --> 00:23:00.609
a type II tear that extends down and forward

00:23:00.609 --> 00:23:03.890
into the antero -inferior labrum, similar to

00:23:03.890 --> 00:23:06.150
a bankart lesion which you see in dislocations.

00:23:06.829 --> 00:23:10.470
Type VI is a type II with an unstable labral

00:23:10.470 --> 00:23:13.890
flap, and type VII involves extension into the

00:23:13.890 --> 00:23:16.930
middle glenohumeral ligament. There are even

00:23:16.930 --> 00:23:20.579
more types described, up to X, but... While these

00:23:20.579 --> 00:23:23.759
extended classifications exist, the Snyder R5

00:23:23.759 --> 00:23:26.000
are really the foundational ones clinicians often

00:23:26.000 --> 00:23:28.619
refer back to. Given the complexity in those

00:23:28.619 --> 00:23:30.740
variants we talked about, is classifying these

00:23:30.740 --> 00:23:33.019
tears always straightforward even when the surgeon

00:23:33.019 --> 00:23:35.480
is looking right at it during surgery? Unfortunately,

00:23:35.680 --> 00:23:37.460
no. This is another challenge highlighted in

00:23:37.460 --> 00:23:40.079
the sources. There's significant inter -observer

00:23:40.079 --> 00:23:42.240
variability. Meaning different surgeons might

00:23:42.240 --> 00:23:44.480
grade it differently. Exactly, and even intra

00:23:44.480 --> 00:23:46.640
-observer variability, where the same surgeon

00:23:46.640 --> 00:23:49.000
might classify the same care differently if they

00:23:49.000 --> 00:23:51.579
looked at it again at different times. This variability

00:23:51.579 --> 00:23:54.240
really underscores the subjective nature of assessing

00:23:54.240 --> 00:23:57.599
these tears. Even with direct visualization during

00:23:57.599 --> 00:24:00.720
arthroscopy. It reinforces that it's not just

00:24:00.720 --> 00:24:03.720
a simple clear -cut diagnosis or classification.

00:24:04.240 --> 00:24:07.059
There's judgment involved. Okay, so we've diagnosed

00:24:07.059 --> 00:24:09.220
it. We've classified it maybe with some variability.

00:24:10.000 --> 00:24:15.000
How do you treat a cell AP tear? Is surgery always

00:24:15.000 --> 00:24:17.980
the answer or is there a non -operative approach

00:24:17.980 --> 00:24:20.279
first? The good news is that surgery is not always

00:24:20.279 --> 00:24:22.839
necessary. And the sources strongly recommend

00:24:22.839 --> 00:24:24.920
starting with non -operative treatment in most

00:24:24.920 --> 00:24:27.240
cases. This is considered the first line of management.

00:24:27.420 --> 00:24:29.859
OK, so try physio first. What does non -operative

00:24:29.859 --> 00:24:32.339
treatment typically involve? Non -operative management

00:24:32.339 --> 00:24:35.319
includes several components. Firstly, rest or

00:24:35.319 --> 00:24:37.319
a modification of activities, especially avoiding

00:24:37.319 --> 00:24:39.119
the movements that cause pain that's particularly

00:24:39.119 --> 00:24:41.220
important for athletes, obviously. Secondly,

00:24:41.680 --> 00:24:44.420
a structured physical therapy program, or physio,

00:24:44.700 --> 00:24:47.559
is crucial. This therapy focuses on regaining

00:24:47.559 --> 00:24:50.160
full, pain -free range of motion, strengthening

00:24:50.160 --> 00:24:52.420
the muscles around the shoulder, especially the

00:24:52.420 --> 00:24:54.319
rotator cuff and the muscles that control the

00:24:54.319 --> 00:24:56.680
shoulder blade. Addressing that scapular dyskinesis

00:24:56.680 --> 00:24:59.259
you mentioned. Precisely. Addressing scapular

00:24:59.259 --> 00:25:01.980
dyskinesis, and importantly, stretching out any

00:25:01.980 --> 00:25:05.299
associated tightness like GIRD, that posterior

00:25:05.299 --> 00:25:09.440
tightness. Thirdly, non -steroidal anti -inflammatory

00:25:09.440 --> 00:25:12.680
medications, NSAIDIDES, can be used to help manage

00:25:12.680 --> 00:25:16.069
pain and reduce inflammation. This non -operative

00:25:16.069 --> 00:25:18.269
approach is typically pursued for a significant

00:25:18.269 --> 00:25:21.029
period usually between three and six months to

00:25:21.029 --> 00:25:22.950
really see if symptoms improve. Three to six

00:25:22.950 --> 00:25:25.130
months, okay. Corticosteroid injections into

00:25:25.130 --> 00:25:27.329
the shoulder joint can also be used often to

00:25:27.329 --> 00:25:30.589
help calm inflammation and sometimes as a diagnostic

00:25:30.589 --> 00:25:32.789
aid if the exact source of the pain is a bit

00:25:32.789 --> 00:25:35.329
unclear. How effective is this non -operative

00:25:35.329 --> 00:25:37.430
approach, especially for that high -demand overhead

00:25:37.430 --> 00:25:39.730
athlete the pitcher may be, who wants to get

00:25:39.730 --> 00:25:42.230
back to throwing at full speed? Ah, this is a

00:25:42.230 --> 00:25:43.970
key distinction made in the sources and it's

00:25:43.970 --> 00:25:46.470
quite important. While non -operative treatment

00:25:46.470 --> 00:25:49.089
can be successful for some individuals, particularly

00:25:49.089 --> 00:25:51.410
those with lower activity demands or maybe more

00:25:51.410 --> 00:25:54.450
degenerative tiers, the data on high -level pitching

00:25:54.450 --> 00:25:56.930
athletes is less encouraging. The number of pitchers

00:25:56.930 --> 00:25:58.970
who successfully return to their prior level

00:25:58.970 --> 00:26:01.349
of performance using only non -surgical methods

00:26:01.349 --> 00:26:04.640
has been described in the literature as Unsatisfactory.

00:26:05.640 --> 00:26:08.039
Unsatisfactory. That's quite blunt. It is. It's

00:26:08.039 --> 00:26:10.559
a critical insight. It suggests that for this

00:26:10.559 --> 00:26:14.140
specific, highly demanding population, non -operative

00:26:14.140 --> 00:26:17.240
treatment, while a necessary first step, often

00:26:17.240 --> 00:26:18.519
doesn't actually get them back to where they

00:26:18.519 --> 00:26:21.599
need to be competitively. That makes sense, unfortunately,

00:26:21.640 --> 00:26:24.359
for them. So if non -operative treatment fails,

00:26:24.700 --> 00:26:27.140
or perhaps if the tear is severe or unstable

00:26:27.140 --> 00:26:29.680
from the outset, surgery comes into consideration.

00:26:29.920 --> 00:26:32.269
What does that involve? Surgical treatment for

00:26:32.269 --> 00:26:35.089
SLAP lesions is typically performed arthroscopically

00:26:35.089 --> 00:26:38.130
keyhole surgery using small incisions, a camera,

00:26:38.369 --> 00:26:40.750
the arthroscope, and specialized miniature instruments.

00:26:41.430 --> 00:26:43.490
The goal of surgery is to address the tear based

00:26:43.490 --> 00:26:46.089
on its type, the patient's age, their activity

00:26:46.089 --> 00:26:48.410
level, and the surgeon's assessment of stability

00:26:48.410 --> 00:26:51.329
and tissue quality. This is also the area where,

00:26:51.529 --> 00:26:54.190
particularly for type 2 tears, there is significant

00:26:54.190 --> 00:26:57.130
ongoing discussion and, as the sources call it,

00:26:57.390 --> 00:26:59.220
quite a bit of controversy. Right, you mentioned

00:26:59.220 --> 00:27:02.480
controversy around type 2 tears. What's the debate

00:27:02.480 --> 00:27:05.299
here? What are the options? Okay, the core debate

00:27:05.299 --> 00:27:07.940
for type 2 tears, remember they're the most common

00:27:07.940 --> 00:27:10.740
in throwers and involve the detached biceps anchor,

00:27:11.279 --> 00:27:13.799
is whether to actually repair the torn labrum

00:27:13.799 --> 00:27:16.779
and reattach the biceps anchor back to the bone.

00:27:16.990 --> 00:27:18.930
The traditional approach. The traditional approach,

00:27:18.930 --> 00:27:22.829
yes. Or, alternatively, whether to perform a

00:27:22.829 --> 00:27:25.490
procedure on the biceps tendon itself, either

00:27:25.490 --> 00:27:28.690
a tenotomy or a tenodesis, and essentially leave

00:27:28.690 --> 00:27:31.690
the superior labrum alone, or perhaps just de

00:27:31.690 --> 00:27:33.910
-bride it, trim it up. Okay, let's clarify those

00:27:33.910 --> 00:27:36.630
biceps procedures again. What exactly are tenotomy

00:27:36.630 --> 00:27:39.369
and tenodesis? Right. Biceps tenotomy is simply

00:27:39.369 --> 00:27:41.490
cutting the long head of the biceps tendon near

00:27:41.490 --> 00:27:43.849
its attachment inside the shoulder joint, allowing

00:27:43.849 --> 00:27:46.150
it to retract down the arm. It essentially removes

00:27:46.150 --> 00:27:48.420
the and pull on the superior labrum. Just cut

00:27:48.420 --> 00:27:51.380
it loose. Pretty much. Biceps tendinesis also

00:27:51.380 --> 00:27:53.660
involves cutting the tendon, but then the cut

00:27:53.660 --> 00:27:55.839
end is reattached, usually with an anchor or

00:27:55.839 --> 00:27:58.900
screw, to the humerus, the upper arm bone, lower

00:27:58.900 --> 00:28:01.559
down, typically outside the shoulder joint itself.

00:28:01.799 --> 00:28:04.059
Okay, cut and reattach it lower down. Exactly.

00:28:04.670 --> 00:28:06.950
Tenodesis is generally preferred over tenotomy

00:28:06.950 --> 00:28:09.309
in active patients because it helps maintain

00:28:09.309 --> 00:28:12.410
biceps strength and the normal shape of the muscle,

00:28:13.049 --> 00:28:14.950
preventing the potential cosmetic deformity called

00:28:14.950 --> 00:28:17.730
a Popeye muscle bulge that can sometimes occur

00:28:17.730 --> 00:28:20.130
with just a tenotomy. Right, the Popeye bulge.

00:28:20.650 --> 00:28:23.470
So traditionally, type 2 tears, especially in

00:28:23.470 --> 00:28:26.569
younger athletes, were repaired. Why the shift

00:28:26.569 --> 00:28:29.170
towards considering tenodesis more often now?

00:28:29.420 --> 00:28:31.940
This is really where the outcomes data is influencing

00:28:31.940 --> 00:28:34.660
practice. While repairing the labrum and biceps

00:28:34.660 --> 00:28:37.759
anchor seems intuitively correct, you know, restoring

00:28:37.759 --> 00:28:39.859
the anatomy. Yeah, it sounds like the right thing

00:28:39.859 --> 00:28:42.519
to do. It does. But recent investigations have

00:28:42.519 --> 00:28:45.700
shown a notable disconnect. A high incidence

00:28:45.700 --> 00:28:48.940
of SLAP repairs being performed, but often disappointing

00:28:48.940 --> 00:28:51.769
return to play rates. particularly for pitchers.

00:28:52.170 --> 00:28:53.829
Studies cited in the sources have shown that

00:28:53.829 --> 00:28:56.609
return -to -play rates after SLAP repairs are

00:28:56.609 --> 00:28:58.950
significantly lower for pitchers compared to

00:28:58.950 --> 00:29:01.109
athletes in other overhead sports like tennis

00:29:01.109 --> 00:29:03.450
or volleyball. Lower for pitchers specifically.

00:29:03.809 --> 00:29:06.690
Interesting. Yes. And importantly, return -to

00:29:06.690 --> 00:29:08.630
-play rates for throwers are actually similar

00:29:08.630 --> 00:29:12.089
whether they undergo a SLAP repair or a biceps

00:29:12.089 --> 00:29:14.970
tenodesis. So the repair doesn't seem to offer

00:29:14.970 --> 00:29:18.009
an advantage for getting back to pitching. Specifically.

00:29:18.289 --> 00:29:20.470
So similar outcomes for return to play, but maybe

00:29:20.470 --> 00:29:23.210
other factors. Exactly. Furthermore, the sources

00:29:23.210 --> 00:29:26.029
highlight a strong association between age and

00:29:26.029 --> 00:29:29.769
the success of SLAP repair. Failures after SLAP

00:29:29.769 --> 00:29:31.890
repair are specifically linked to patients over

00:29:31.890 --> 00:29:35.450
the age of 36. Over 36. OK. This suggests that

00:29:35.450 --> 00:29:38.089
perhaps tissue quality or healing potential might

00:29:38.089 --> 00:29:40.569
be poorer in older patients, making the repair

00:29:40.569 --> 00:29:43.369
less likely to hold reliably or possibly leading

00:29:43.369 --> 00:29:45.349
to complications like stiffness, which we'll

00:29:45.349 --> 00:29:47.799
talk about. Because of these findings, poorer

00:29:47.799 --> 00:29:50.019
return to throwing after repair compared to non

00:29:50.019 --> 00:29:52.160
-throwing sports, similar return to throwing

00:29:52.160 --> 00:29:54.900
between repair and tenodesis, and higher failure

00:29:54.900 --> 00:29:57.259
and stiffness rates in older patients after repair,

00:29:57.660 --> 00:29:59.640
the general consensus among orthopedic surgeons.

00:30:00.259 --> 00:30:02.180
As reflected in the literature, is increasingly

00:30:02.180 --> 00:30:05.319
favoring bicep synotomy or tenodesis, especially

00:30:05.319 --> 00:30:07.500
in patients over the age of 40. Right, so over

00:30:07.500 --> 00:30:10.279
40 lean towards the biceps procedure. Generally,

00:30:10.460 --> 00:30:13.680
yes. For younger, high -level overhead athletes,

00:30:14.180 --> 00:30:16.359
the decision is still debated, it's more nuanced,

00:30:16.779 --> 00:30:18.819
but the pendulum is certainly swinging towards

00:30:18.819 --> 00:30:20.980
considering biceps procedures more frequently.

00:30:21.579 --> 00:30:24.079
Even in this group, if return to high -level

00:30:24.079 --> 00:30:26.960
throwing is the primary goal, simply due to the

00:30:26.960 --> 00:30:29.240
less -than -ideal results seen with repair in

00:30:29.240 --> 00:30:32.160
that specific population. That's a really significant

00:30:32.160 --> 00:30:35.220
shift in thinking, isn't it? Moving away from

00:30:35.220 --> 00:30:38.299
trying to restore the anatomy with repair towards

00:30:38.299 --> 00:30:41.019
essentially just offloading the biceps tension

00:30:41.019 --> 00:30:43.279
because the repair outcomes, particularly for

00:30:43.279 --> 00:30:45.859
throwers or older patients, haven't been as successful

00:30:45.859 --> 00:30:48.359
as everyone hoped. What about the other types

00:30:48.359 --> 00:30:50.980
of tears, types 1, 3, and 4? Right, for type

00:30:50.980 --> 00:30:53.359
I tears, which are just fraying with an intact

00:30:53.359 --> 00:30:55.960
anchor, and maybe some type III or V tears involving

00:30:55.960 --> 00:30:58.140
less than one -third of the biceps tendon without

00:30:58.140 --> 00:31:00.220
significant symptoms after non -operative treatment,

00:31:00.700 --> 00:31:03.079
simple arthroscopic debridement is often sufficient.

00:31:03.240 --> 00:31:05.319
Just trimming the frayed bits. Exactly, just

00:31:05.319 --> 00:31:07.559
trimming away the unstable frayed edges of the

00:31:07.559 --> 00:31:11.400
labrum or biceps. For type 4 tears, where more

00:31:11.400 --> 00:31:13.500
than one -third of the biceps tendon is involved,

00:31:13.720 --> 00:31:15.599
causing severe symptoms that haven't responded

00:31:15.599 --> 00:31:18.099
to non -operative care, the options are typically

00:31:18.099 --> 00:31:21.099
debridement plus either labor repair or biceps

00:31:21.099 --> 00:31:24.059
tendon imidinodesis. The choice here would again

00:31:24.059 --> 00:31:26.200
depend heavily on the patient's age, activity

00:31:26.200 --> 00:31:28.440
level, and the surgeon's preference, considering

00:31:28.440 --> 00:31:30.420
all the same factors we just discussed for type

00:31:30.420 --> 00:31:33.519
2. So treatment is highly individualized based

00:31:33.519 --> 00:31:37.009
on tear type. age, activity goals, and this evolving

00:31:37.009 --> 00:31:40.079
evidence. Once surgery is done, whichever route

00:31:40.079 --> 00:31:42.579
is chosen, what does the road to recovery look

00:31:42.579 --> 00:31:45.720
like? Rehabilitation must be absolutely critical.

00:31:46.160 --> 00:31:48.299
Oh, rehabilitation is absolutely paramount to

00:31:48.299 --> 00:31:50.420
the success of any surgical intervention for

00:31:50.420 --> 00:31:53.180
a cesilite tear, whether it's a repair or a biceps

00:31:53.180 --> 00:31:55.680
procedure. It's not optional, it's essential.

00:31:55.980 --> 00:31:58.839
It's a structured multi -stage process that progresses

00:31:58.839 --> 00:32:01.319
gradually to protect the healing issues while

00:32:01.319 --> 00:32:03.160
carefully restoring motion and strength. Okay,

00:32:03.240 --> 00:32:05.480
what's the initial phase like after the surgery?

00:32:05.720 --> 00:32:08.559
Immediately after surgery, the shoulder is typically

00:32:08.559 --> 00:32:11.819
immobilized in a sling. The duration varies depending

00:32:11.819 --> 00:32:13.940
on the surgery's complexity and the surgeon's

00:32:13.940 --> 00:32:16.160
protocol, but usually ranges from about two to

00:32:16.160 --> 00:32:19.400
six weeks. The initial phase, typically the first

00:32:19.400 --> 00:32:22.099
one to four weeks, focuses heavily on protection

00:32:22.099 --> 00:32:24.619
and controlled motion. Patients are usually allowed

00:32:24.619 --> 00:32:27.339
gentle passive range of motion, where the therapist

00:32:27.549 --> 00:32:30.470
moves the arm, and maybe some active assisted

00:32:30.470 --> 00:32:32.970
range of motion exercises, often in the scapular

00:32:32.970 --> 00:32:35.069
plane, which is arm moving slightly forward from

00:32:35.069 --> 00:32:37.809
the side, kind of a safe zone. Crucially, during

00:32:37.809 --> 00:32:40.990
this phase, it is vital to avoid any extremes

00:32:40.990 --> 00:32:43.430
of abduction and external rotation. Right. Don't

00:32:43.430 --> 00:32:45.910
stretch it out too much. Exactly. And absolutely

00:32:45.910 --> 00:32:48.529
no resisted biceps exercises, especially if the

00:32:48.529 --> 00:32:50.849
biceps anchor was repaired, to protect that healing

00:32:50.849 --> 00:32:52.900
site. You don't want to pull on it. Makes sense.

00:32:53.279 --> 00:32:55.839
What happens as you move beyond that initial

00:32:55.839 --> 00:32:57.920
protective phase, say after the first month or

00:32:57.920 --> 00:33:01.140
so? Around weeks 4 -6, as healing progresses

00:33:01.140 --> 00:33:04.240
and pain allows, the patient typically transitions

00:33:04.240 --> 00:33:07.099
to active range of motion exercises, trying to

00:33:07.099 --> 00:33:09.160
move the arm themselves under their own power.

00:33:09.940 --> 00:33:12.059
They also usually start isometric strengthening

00:33:12.059 --> 00:33:14.240
tensing muscles without actually moving the joint.

00:33:14.410 --> 00:33:17.809
The mid -stage, generally from week 6 -12, involves

00:33:17.809 --> 00:33:19.990
progressing to more functional exercises and

00:33:19.990 --> 00:33:22.049
beginning light -resistant strengthening, really

00:33:22.049 --> 00:33:24.609
focusing on the rotator cuff and those periscapular

00:33:24.609 --> 00:33:26.450
muscles, the ones controlling the shoulder blade.

00:33:27.029 --> 00:33:29.569
The goal is to build foundational strength and

00:33:29.569 --> 00:33:31.869
control. Okay, building back the support system.

00:33:32.069 --> 00:33:34.529
Precisely. The late stage, from 12 weeks onwards,

00:33:35.130 --> 00:33:37.809
involves advancing strengthening exercises, restoring

00:33:37.809 --> 00:33:40.490
full range of motion, and initiating sport -specific

00:33:40.490 --> 00:33:43.329
training if the patient is an athlete. This might

00:33:43.329 --> 00:33:45.630
involve progressive drills mimicking the movements

00:33:45.630 --> 00:33:47.970
required for their sport, like starting a throwing

00:33:47.970 --> 00:33:50.809
program. When can someone realistically expect

00:33:50.809 --> 00:33:53.450
to return to full activity or sport after all

00:33:53.450 --> 00:33:56.069
this? A typical return to full activity or sport

00:33:56.069 --> 00:33:58.990
after SLIP surgery is often quoted around six

00:33:58.990 --> 00:34:01.869
months. However, this timeline is highly variable.

00:34:02.039 --> 00:34:04.779
It really depends on the specific type and severity

00:34:04.779 --> 00:34:07.119
of the injury, the patient's individual healing

00:34:07.119 --> 00:34:10.099
rate, their overall health, and critically, their

00:34:10.099 --> 00:34:12.460
activity goals. Six months is just a ballpark

00:34:12.460 --> 00:34:15.519
figure, them. Very much so. For throwing athletes,

00:34:15.760 --> 00:34:17.800
an early interval throwing program might begin

00:34:17.800 --> 00:34:20.300
around three, four months. But a full return

00:34:20.300 --> 00:34:22.659
to competitive pitching can easily take six months

00:34:22.659 --> 00:34:25.380
or even longer, sometimes nine months to a year,

00:34:25.619 --> 00:34:28.750
to feel truly back to normal. The sources emphasize

00:34:28.750 --> 00:34:30.849
that complete recovery can take several months,

00:34:31.250 --> 00:34:34.130
particularly in more complicated cases. And diligently

00:34:34.130 --> 00:34:36.449
following the surgeon's post -operative instructions

00:34:36.449 --> 00:34:39.769
and the prescribed rehabilitation plan is absolutely

00:34:39.769 --> 00:34:41.969
vital for optimizing the outcome. You simply

00:34:41.969 --> 00:34:43.989
can't rush the biology of healing. That sounds

00:34:43.989 --> 00:34:46.349
like a long and very dedicated process. What

00:34:46.349 --> 00:34:48.530
are the potential roadblocks or complications

00:34:48.530 --> 00:34:51.869
that can occur after SLAP surgery? What can go

00:34:51.869 --> 00:34:54.590
wrong? Well, like any surgical procedure. There

00:34:54.590 --> 00:34:57.690
are potential complications. For SLAP repair

00:34:57.690 --> 00:34:59.949
specifically, the most common complication reported

00:34:59.949 --> 00:35:02.929
in the sources is post -operative stiffness.

00:35:03.789 --> 00:35:05.650
It occurs in a significant percentage of patients,

00:35:05.769 --> 00:35:10.309
one source mentioned, around 78%. 78%. That's

00:35:10.309 --> 00:35:13.230
a very high number for stiffness. Are there specific

00:35:13.230 --> 00:35:15.949
factors that make someone more likely to experience

00:35:15.949 --> 00:35:20.159
stiffness after a SLAP repair? Yes, the sources

00:35:20.159 --> 00:35:22.780
identify significant risk factors for post -operative

00:35:22.780 --> 00:35:26.420
stiffness after SLAP repair. A particularly strong

00:35:26.420 --> 00:35:29.360
risk factor seems to be performing an incidental

00:35:29.360 --> 00:35:32.440
SLAP repair in older patients, specifically those

00:35:32.440 --> 00:35:35.000
over the age of 45, when doing surgery for a

00:35:35.000 --> 00:35:37.239
different primary problem, such as a rotator

00:35:37.239 --> 00:35:40.239
cuff repair. So finding a cell AP tear while

00:35:40.239 --> 00:35:42.239
fixing something else and deciding to repair

00:35:42.239 --> 00:35:44.719
it in an older patient is risky for stiffness.

00:35:45.059 --> 00:35:47.059
Exactly. It reinforces the clinical trend away

00:35:47.059 --> 00:35:49.059
from routinely repairing these lesions in older

00:35:49.059 --> 00:35:51.639
patients and perhaps opting for a biceps procedure

00:35:51.639 --> 00:35:54.219
instead, which tends to have less risk of stiffness.

00:35:54.639 --> 00:35:56.420
And if someone does develop stiffness, how is

00:35:56.420 --> 00:35:58.880
it treated? Can it be fixed? Early recognition

00:35:58.880 --> 00:36:02.280
and intervention are key. Rehabilitation protocols

00:36:02.280 --> 00:36:04.619
are designed to start controlled motion relatively

00:36:04.619 --> 00:36:07.599
early, even in the protective phase. Things like

00:36:07.599 --> 00:36:09.840
pendulum swings are often encouraged, sometimes

00:36:09.840 --> 00:36:12.539
beginning as early as one week post -op, specifically

00:36:12.539 --> 00:36:15.980
to help prevent stiffness setting in. If stiffness

00:36:15.980 --> 00:36:18.780
develops and persists despite dedicated physio,

00:36:19.159 --> 00:36:21.519
a secondary procedure called a capsular release

00:36:21.519 --> 00:36:24.639
might be necessary. This involves arthroscopically

00:36:24.639 --> 00:36:26.820
releasing the tightened tissues, the capsule,

00:36:27.079 --> 00:36:30.480
around the joint to restore motion. Okay. Are

00:36:30.480 --> 00:36:32.820
there other potential complications besides stiffness?

00:36:33.579 --> 00:36:36.219
Yes, other potential complications include, though

00:36:36.219 --> 00:36:38.840
less commonly, injury to the suprascapular nerve.

00:36:39.619 --> 00:36:41.500
This is a nerve near the shoulder blade that

00:36:41.500 --> 00:36:44.019
controls some of the rotator cuff muscles. This

00:36:44.019 --> 00:36:45.980
could potentially happen if the glenoid bone

00:36:45.980 --> 00:36:48.159
is over -drilled during the process of placing

00:36:48.159 --> 00:36:50.900
anchors for a prey pair, as the nerve runs quite

00:36:50.900 --> 00:36:53.780
close by. Careful technique is needed. Right.

00:36:53.960 --> 00:36:56.000
And unfortunately, sometimes the SLAP repair

00:36:56.000 --> 00:36:58.719
simply fails or the patient continues to experience

00:36:58.719 --> 00:37:00.820
symptoms despite the surgery. What typically

00:37:00.820 --> 00:37:03.480
causes a failed SLAP repair? Why might it not

00:37:03.480 --> 00:37:06.280
work? Failures can be multifactorial. There isn't

00:37:06.280 --> 00:37:09.579
always one single reason. Causes can include

00:37:09.579 --> 00:37:12.519
the repair anchors pulling out of the bone, failure

00:37:12.519 --> 00:37:14.760
of the labral tissue itself to heal back to the

00:37:14.760 --> 00:37:17.760
bone properly, or perhaps the presence of associated

00:37:17.760 --> 00:37:20.940
injuries. like articular cartilage damage that

00:37:20.940 --> 00:37:23.219
weren't fully addressed initially or that progressed

00:37:23.219 --> 00:37:26.539
over time. The sources again highlight the association

00:37:26.539 --> 00:37:29.280
of failures with age, specifically noting that

00:37:29.280 --> 00:37:31.119
failures are more common in patients over the

00:37:31.119 --> 00:37:33.619
age of 36. Right, reinforcing that age factor

00:37:33.619 --> 00:37:36.480
again. Exactly. Further supporting the rationale

00:37:36.480 --> 00:37:39.099
for considering biceps tenodesis in this age

00:37:39.099 --> 00:37:41.780
group. It's also really important to note that

00:37:41.780 --> 00:37:44.820
the outcomes after surgical management of a failed

00:37:44.820 --> 00:37:48.019
SLAP repair. So going back in to try and revise

00:37:48.019 --> 00:37:50.699
the initial surgery are generally inferior to

00:37:50.699 --> 00:37:53.079
the outcomes achieved after a successful primary

00:37:53.079 --> 00:37:54.880
repair. So it's much better to get it right the

00:37:54.880 --> 00:37:57.099
first time. Absolutely. It really emphasizes

00:37:57.099 --> 00:37:58.960
the importance of selecting the right procedure

00:37:58.960 --> 00:38:00.699
for the right patient the first time around.

00:38:01.380 --> 00:38:03.840
So getting that initial treatment decision correct

00:38:03.840 --> 00:38:07.059
based on all the factors, age, activity, tear

00:38:07.059 --> 00:38:10.260
type, evidence, is really paramount for the best

00:38:10.260 --> 00:38:13.280
chance of success. Putting it all together then,

00:38:13.699 --> 00:38:16.219
what's the overall prognosis for someone who

00:38:16.219 --> 00:38:18.179
undergoes treatment for a cell AP tear? What

00:38:18.179 --> 00:38:21.119
can they expect? Well, the prognosis for a cell

00:38:21.119 --> 00:38:23.400
AP after treatment is generally positive in terms

00:38:23.400 --> 00:38:25.260
of reducing pain and improving shoulder strength

00:38:25.260 --> 00:38:27.659
for the majority of patients, particularly those

00:38:27.659 --> 00:38:29.699
with symptoms directly attributable to the tear

00:38:29.699 --> 00:38:31.639
who undergo appropriate surgical intervention

00:38:31.639 --> 00:38:33.739
after feeling a good course of non -operative

00:38:33.739 --> 00:38:36.710
care. However, as we've discussed quite a bit,

00:38:37.170 --> 00:38:39.429
the ability to return to a prior high level of

00:38:39.429 --> 00:38:41.929
performance, especially for elite overhead throwers,

00:38:42.289 --> 00:38:44.809
remains variable and can be unpredictable. It's

00:38:44.809 --> 00:38:47.250
not guaranteed. Achieving a successful outcome

00:38:47.250 --> 00:38:49.590
relies heavily on the correct diagnosis in the

00:38:49.590 --> 00:38:51.750
first place, appropriate treatment selection

00:38:51.750 --> 00:38:54.710
tailored to the individual, addressing any associated

00:38:54.710 --> 00:38:57.489
shoulder pathologies concurrently, and critically,

00:38:57.719 --> 00:38:59.900
the patient's commitment to that structured and

00:38:59.900 --> 00:39:02.239
often lengthy post -operative rehabilitation

00:39:02.239 --> 00:39:05.400
process. It really is a complex injury requiring

00:39:05.400 --> 00:39:07.980
a comprehensive approach from everyone involved.

00:39:08.199 --> 00:39:10.460
This has truly been a deep dive into the world

00:39:10.460 --> 00:39:13.659
of SLAP lesions. We've covered the nuanced anatomy,

00:39:14.059 --> 00:39:16.320
including those tricky variants like the Buford

00:39:16.320 --> 00:39:18.980
complex, gone through the detective work of diagnosis

00:39:18.980 --> 00:39:21.619
using history, physical exam, and specialized

00:39:21.619 --> 00:39:24.800
imaging like MRI orthograms, and explored the

00:39:24.800 --> 00:39:26.860
evolving and sometimes controversial landscape

00:39:26.860 --> 00:39:28.960
of treatment decisions, particularly that shift

00:39:28.960 --> 00:39:31.579
towards biceps procedures for type 2 pairs in

00:39:31.579 --> 00:39:34.119
certain patient populations. Plus the challenges

00:39:34.119 --> 00:39:36.619
of rehabilitation and potential complications

00:39:36.619 --> 00:39:39.880
like that risk of stiffness. It's absolutely

00:39:39.880 --> 00:39:41.900
clear that this is far from a simple injury,

00:39:42.159 --> 00:39:44.719
and managing it effectively requires a deep understanding

00:39:44.719 --> 00:39:47.059
of all these interconnected factors. That's a

00:39:47.059 --> 00:39:49.840
very accurate summary, I think. The diagnostic

00:39:49.840 --> 00:39:52.920
challenges, the importance of recognizing anatomical

00:39:52.920 --> 00:39:55.420
variants, the controversy surrounding the optimal

00:39:55.420 --> 00:39:58.179
management of type 2 care, especially in different

00:39:58.179 --> 00:40:00.719
age groups and activity levels, and the critical

00:40:00.719 --> 00:40:03.980
role of rehabilitation. Those are all key takeaways

00:40:03.980 --> 00:40:06.650
from the literature we reviewed today. And understanding

00:40:06.650 --> 00:40:09.530
why certain decisions are made, like opting for

00:40:09.530 --> 00:40:12.050
a biceps tenodesis over a repair in an older

00:40:12.050 --> 00:40:14.309
patient, because of that increased risk of failure

00:40:14.309 --> 00:40:16.389
or stiffness, that's crucial information for

00:40:16.389 --> 00:40:18.750
both clinicians and patients to understand, isn't

00:40:18.750 --> 00:40:21.670
it? Indeed. And the evidence base is constantly

00:40:21.670 --> 00:40:24.150
evolving, which is why this area remains a topic

00:40:24.150 --> 00:40:26.349
of significant research and clinical discussion.

00:40:26.809 --> 00:40:28.789
It keeps changing. Okay, thinking about everything

00:40:28.789 --> 00:40:31.989
we've covered. The anatomical complexities, the

00:40:31.989 --> 00:40:34.250
difficulty in definitively diagnosing these tiers

00:40:34.250 --> 00:40:36.690
sometimes, the ongoing debate about the best

00:40:36.690 --> 00:40:38.670
way to fix certain types, especially for athletes

00:40:38.670 --> 00:40:40.809
trying to get back to peak performance, and the

00:40:40.809 --> 00:40:43.190
impact of age on outcomes and complications.

00:40:43.849 --> 00:40:45.710
Here's a provocative thought to leave you, our

00:40:45.710 --> 00:40:48.320
listeners, with. Given these persistent challenges,

00:40:48.460 --> 00:40:51.079
how might future advancements, maybe in areas

00:40:51.079 --> 00:40:53.579
like artificial intelligence, helping to analyze

00:40:53.579 --> 00:40:57.119
subtle patterns on MRI scans, or more sophisticated

00:40:57.119 --> 00:40:59.579
biomechanical modeling to understand forces on

00:40:59.579 --> 00:41:02.199
the labor in real time during movement, or even

00:41:02.199 --> 00:41:04.360
biological approaches to enhance laboral healing

00:41:04.360 --> 00:41:06.940
itself, how might these things fundamentally

00:41:06.940 --> 00:41:09.599
change how we diagnose and treat SLAP tiers in

00:41:09.599 --> 00:41:11.860
the future? Could we reach a point where we can

00:41:11.860 --> 00:41:14.500
much more accurately predict who needs what treatment

00:41:14.500 --> 00:41:16.519
and who is most likely to achieve their desired

00:41:16.519 --> 00:41:19.280
outcome? Or conversely, is the inherent variability

00:41:19.280 --> 00:41:21.880
in human anatomy, tissue quality, and individual

00:41:21.880 --> 00:41:24.360
healing potential simply something we will always

00:41:24.360 --> 00:41:26.619
have to navigate with clinical judgment and experience

00:41:26.619 --> 00:41:29.179
no matter how good the tech gets? Those are certainly

00:41:29.179 --> 00:41:31.619
the questions driving future research and innovation

00:41:31.619 --> 00:41:34.559
in orthopedic sports medicine. Big questions

00:41:34.559 --> 00:41:37.239
for the future. This has been the Deep Dive.

00:41:37.400 --> 00:41:39.840
We hope this detailed exploration has given you

00:41:39.840 --> 00:41:43.320
a clearer picture of SLAP lesions. If you found

00:41:43.320 --> 00:41:45.960
this insightful, please do consider reading and

00:41:45.960 --> 00:41:48.099
sharing this deep dive with colleagues or friends

00:41:48.099 --> 00:41:50.059
who might find it useful. Thank you for joining

00:41:50.059 --> 00:41:50.380
us.
