WEBVTT

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Imagine you're a surgeon, you're looking at the

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scan, patient's shoulder keeps copping out anteriorly.

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You know you've got options, right? The Bankart,

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LaTarjet, maybe others. But choosing the right

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one for this patient, their specific anatomy,

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the exact injury, well, it feels less like picking

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a tool, more like a really tricky puzzle, high

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stakes. It absolutely is. It's a challenge orthopedic

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surgeons face all the time with traumatic anterior

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recurrent. shoulder instability. You know, the

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usual soft tissue fixes might not cut it if there's

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significant bone damage, but then bone procedures,

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they have their own set of issues. The big question

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has always been how do we map that out? How do

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we get a reliable guide to the best treatment

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for each person? And that is exactly what we're

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digging into today. We're looking at a systematic

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review from the Archives of Bone and Joint Surgery.

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You co -authored it, and it doesn't just summarize

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the evidence, it actually proposes a whole new

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classification system. The idea is to cut through

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that complexity, yeah, simplify those decisions

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for this specific type of instability. Yes, it's

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great to be able to talk about this. The goal

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really was to bring together the current evidence

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and offer a more structured evidence -based way

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forward for both surgeons and their patients.

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Okay, so before we jump into the nuts and bolts

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of this new system, let's just stay with that

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complexity for a second. Why weren't the old

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ways quite working? You know, just relying on

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surgeon preference or existing classifications,

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what crucial things weren't really being looked

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at together? Well, the core problem is that success

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Preventing it from happening again. It hinges

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on several things all working together not just

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one or two factors You can fix the torn tissue

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sure, but if there's say Quite a bit of bone

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loss on the socket the glenoid Or if that dent

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on the ball part of the arm bone the hill sacks

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lesion catches when they move The shoulder can

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still pop out right and equally if the patient's

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soft tissues are just well poor quality from

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repeated dislocations Even a technically good

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repair might not hold Older systems tended to

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focus on maybe just the bone loss or just the

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tissue or maybe the hill sacks. They didn't really

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combine the assessment of all three effectively.

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The bone loss, the quality of the soft tissue

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that's left, and the specific nature of that

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hill sacks lesion is engaging or not. That wasn't

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really integrated into one single pathway. So

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surgeons were sort of... mentally juggling these

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variables. Leading to less consistency, I imagine.

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It sounds like those key puzzle pieces weren't

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being fitted together properly. Exactly. And

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that's precisely the gap this new both classification

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system aims to fill. It gives a structured method

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to assess these three really critical injury

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features and then map them to the surgical strategies

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that the evidence supports most strongly. Both.

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OK, let's break that down. B -O -T -H. What's

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it stand for and how does it bring order to this?

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So both stands for bone loss, tissue quality,

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and hill sacs lesion. The whole idea is that

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by accurately classifying the severity and the

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type of damage in these three areas, you can

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seriously narrow down the surgical choices. You

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can pinpoint the procedure, or maybe combination

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of procedures, that's most likely to give that

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patient long -term stability. Right, getting

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really specific about the injury pattern. Okay.

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Okay, let's start with MO bone loss. How do you

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look at that? What are the key differences the

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system makes? Bone loss refers to how much bone

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has been chipped away or worn down from the front

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of the glen or at the socket. We usually measure

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it from CT scans, look at it as a percentage

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of the socket surface. And the research, which

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lines up with what's already known, points to

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a key threshold often around 15 to 20 percent

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damage below that level. We call that subcritical

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bone loss. More than that is critical bone loss.

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And that distinction is really fundamental because

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critical bone loss usually means you have to

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add bone back to make the socket stable again.

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Subcritical loss. Well, you might manage with

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just soft tissue work, but it depends on the

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other factors. OK, so bow is subcritical or critical

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around that 15, 20 percent mark. Got it. Yeah.

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Next up to tissue quality. What's being assessed

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there? Peace looks at the quality of the labrum

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and the capsule at the front of the shoulder.

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They're the main soft tissue stabilizers. like

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a bumper and a strong hammock holding the ball.

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If they've been torn repeatedly or they're stretched

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out or maybe just naturally a bit lax, well,

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they can't stabilize things properly. So we classify

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this quite simply as good or poor. Good or poor,

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okay. Good quality you typically see after maybe

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a first or second dislocation, someone without

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that underlying looseness. Poor quality is more

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common after lots of dislocations. The tissue

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is just degraded, worn out, can't really do its

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job as a primary stabilizer anymore. So tease

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about how resilient those soft tissues are still

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up to the job, or basically worn out. Makes sense.

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And finally, H. hilsax lesion. You mentioned

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it's a dent, but the classification isn't just

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if it's there. It's about this. on track versus

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off track thing. Precisely. A hill -sax lesion,

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that's a dent on the back of the humeral head,

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the ball. It happens when it hits the glenoid

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rim during a dislocation. Right. But the really

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critical thing, and this builds on some very

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important work in the field, is whether that

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dent engages or catches on the front edge of

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the socket when you move your arm into certain

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positions like reaching up and out. Ah, I see.

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If it doesn't catch, doesn't lever the joint

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out, we call it noontrack. But if it does engage,

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acts like a sort of cam forcing the joint out,

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then it's off -track. And an off -track hillsax

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is a major driver of instability. It needs specific

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treatment, really regardless of the other factors.

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So the both system puts these three things together.

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Bone loss, subcritical, tissue quality, good

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poor. and Hill Sachs, Noontrac off -track. That

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defines the specific injury patterns. And this

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is the map you were talking about guiding the

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treatment. Let's piece it together. What does

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both type 1 look like? Type 1 is probably the

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most straightforward scenario. You've got subcritical

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bone loss less than that 15 -20%. You've got

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good tissue quality. And the Hill Sachs lesion

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is Noontrac. So the main issue here is the initial

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soft tissue tear from the dislocation. But the

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basic structure and tissue strengths are still

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pretty sound. And based on your review, what's

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the recommended path for Type 1? For Type 1,

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the evidence strongly supports a standard bankart

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repair. This procedure just anatomically fixes

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that torn labrum, tightens up the capsule, essentially

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restores the soft tissue bumper. Since the bone's

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okay, tissues are good, HillSex isn't causing

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trouble, just fixing the soft tissue damage is

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often enough to get stability back. Okay, that

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makes sense. Fix the main problem if the foundations

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are solid. Now, Type 1 plus seri... That little

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plus suggests something extra is going on. Exactly

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right. Type 1 Plus still has that subcritical

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bone loss and the good tissue quality, just like

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Type 1. But the key difference is an off -track

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hill -sax lesion. So you still have good tissues,

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not much bone loss on the socket, but that dent

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on the humerus is actively making it unstable

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because it's catching on the glenoid edge. So

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you need to fix the tissue and deal with that

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engaging hill -sax. What does the system suggest

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then? For type 1 plus Harry, the recommendation

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is a bankart repair plus a remplasage. Remplasage

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is a technique where we take the infraspinatus

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tendon from the back of the shoulder and stitch

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it into that hillsack's dent. It effectively

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fills the hole, stops it from catching or engaging

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on the glenoid when the arm moves, so it gets

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rid of that specific way it was re -dislocating.

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Fascinating. So remplasage deals specifically

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with that off -track hillsacks when bone loss

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and tissues are otherwise alright. OK, let's

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move to type 2. How does the picture change now?

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With type 2, we're still looking at subcritical

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bone loss and still in noon -track hill sacks.

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But, and this is the crucial difference, we now

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have poor tissue quality. The soft tissues, the

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labrum and capsule, they just aren't reliable

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stabilizers anymore. Maybe stretched, frayed

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from previous dislocations. So even if the bone's

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mostly fine and the hill sacks isn't catching,

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if the tissue itself is compromised, a standard

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bank heart probably won't hold up. That's exactly

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it. Trying to repair poor quality tissue with

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a standard bankard? Well, the results are less

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predictable. So for type two, the research points

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towards dynamic interior stabilization or DAS.

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Yes. This technique uses the long head of the

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biceps tendon. It's redirected to the front of

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the glenoid and it acts like a dynamic sling

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or a hammock. The biomechanical evidence really

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supports DAS in this specific situation. It gives

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that vital dynamic support that the degraded

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native tissues just can't provide anymore. It

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compensates for the poor tissue even when bone

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loss isn't critical. That makes a lot of sense,

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bringing in a reinforcement, a dynamic one, when

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the original tissues are unreliable. And following

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the pattern, type 2 plus must be subcritical

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bone loss, poor tissue, and the off -track hill

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sacks. You've got it perfectly. Type 2 plus brings

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together subcritical bone loss, poor tissue quality,

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and an off -track hill sacks lesion. Which would

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logically need the DAS for the poor tissue and

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the remposage for the hill sacks. Precisely the

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recommendation, DAS plus remposage. You need

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both parts to tackle that specific combination

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of problems. Okay, right, so that covers all

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the situations where bone loss is subcritical.

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You layer in DAS for poor tissue, or remplassage

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for an off -track hillsax, or both if needed.

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Now, things get more complex when we cross that

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15 -20 % line into critical bone loss. What about

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both type 3? Well, once you have critical bone

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loss, just fixing soft tissue onto a deficient

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socket isn't really enough. You've got to rebuild

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the bony structure. So type 3 is defined as critical

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bone loss over 15 -20%, but importantly good

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tissue quality. and a Noontrag Hillsax. The bone

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needs sorting, but the soft tissues are still

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okay. And the suggested procedure for type 3.

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For type 3, the evidence points towards free

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glenoid bone grafting, or FGBG. This involves

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taking a piece of bone from somewhere else, often

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the patient's hip bone, the iliac crest, or sometimes

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another bit of the shoulder, like the end of

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the collarbone, and grafting it onto the front

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of the glenoid, rebuilding the socket to its

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proper shape. FGBG. So basically creating a new

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anatomical bony stop. The research mentioned

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this might be preferred over Letarjet sometimes.

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Yes. The review notes that FGBG gives you an

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anatomical reconstruction, which can be an advantage.

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For type 3, where the tissue quality is still

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good and you don't necessarily need that dynamic

00:10:09.200 --> 00:10:12.179
sling effect you get with Letarjet, FGBG is presented

00:10:12.179 --> 00:10:15.139
as a really good anatomical option. It restores

00:10:15.139 --> 00:10:17.580
the bone and potentially has a different risk

00:10:17.580 --> 00:10:20.580
profile compared to Letarjet in just this specific

00:10:20.580 --> 00:10:23.779
context. Interesting. So FGBG for critical bone

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loss if the tissues are good. Following the pattern,

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type 3 plus must be critical bone loss, good

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tissue, and the off -track hill sacs. Exactly.

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Critical bone loss, good tissue quality, and

00:10:33.759 --> 00:10:35.820
an off -track hill sacs. The treatment logically

00:10:35.820 --> 00:10:39.200
follows. FGBG plus room placage. Rebuild the

00:10:39.200 --> 00:10:41.779
socket anatomically with the graft and neutralize

00:10:41.779 --> 00:10:44.100
that engaging hill sacs lesion. All right. Nearly

00:10:44.100 --> 00:10:46.740
there. Two types left. Type 4. Critical bone

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loss again. What about the tissue and hill sacks

00:10:48.860 --> 00:10:50.799
here? Type 4 is where you have the double whammy.

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Yeah. Critical bone loss and poor tissue quality,

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but the hill sacks lesion is noontrack. So both

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the bone structure and the soft tissue restraints

00:10:58.139 --> 00:11:01.139
are significantly compromised. Ugh. Critical

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bone loss and poor tissue. This sounds like classic

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territory for the Letarjet procedure, doesn't

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it? Precisely. Letarjet is the recommended treatment

00:11:08.840 --> 00:11:12.149
for type 4. This procedure, as you know, transfers

00:11:12.149 --> 00:11:14.830
the coracoid process, that bony outcrop, from

00:11:14.830 --> 00:11:17.049
the shoulder blade along with its attached muscles,

00:11:17.450 --> 00:11:19.750
the conjoined tendon, to the front of the glenoid.

00:11:19.919 --> 00:11:22.779
The bone bit addresses the critical bone loss,

00:11:23.039 --> 00:11:25.899
rebuilds the socket depth, but crucially, that

00:11:25.899 --> 00:11:28.200
transferred tendon acts as a powerful dynamic

00:11:28.200 --> 00:11:30.639
stabilizer, provides that vital sling effect.

00:11:30.740 --> 00:11:32.899
And that's particularly helpful when the patient's

00:11:32.899 --> 00:11:35.639
own soft tissues are poor quality and can't provide

00:11:35.639 --> 00:11:38.259
enough stability on their own. So Letarjet tackles

00:11:38.259 --> 00:11:40.620
both issues, the missing bone and the poor tissue,

00:11:40.620 --> 00:11:43.659
even though it's not strictly anatomical. The

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research did mention its risks, though. It did.

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The systematic review acknowledges Letarjet.

00:11:49.080 --> 00:11:52.299
while highly effective, is definitely more complex

00:11:52.299 --> 00:11:54.799
than a straightforward bank card. It has a known

00:11:54.799 --> 00:11:57.779
learning curve, and, as the evidence we reviewed

00:11:57.779 --> 00:12:00.120
showed, it can have complications like infection

00:12:00.120 --> 00:12:02.039
or nerve issues, potentially at higher rates

00:12:02.039 --> 00:12:05.460
than, say, FGBG or bank card, when used in the

00:12:05.460 --> 00:12:07.960
right situations. Which, again, just highlights

00:12:07.960 --> 00:12:10.200
why matching the procedure to the specific injury

00:12:10.200 --> 00:12:13.039
pattern, like both aims to do, is so important.

00:12:13.399 --> 00:12:15.720
Understanding the trade -offs. Okay, and finally,

00:12:15.960 --> 00:12:18.600
type 4 plus 0. The most complex picture. Type

00:12:18.600 --> 00:12:21.159
4 plus is critical bone loss, poor tissue quality,

00:12:21.620 --> 00:12:23.639
and an off -track hill -sax lesion. You've got

00:12:23.639 --> 00:12:26.059
significant bone loss, unreliable soft tissues,

00:12:26.220 --> 00:12:28.480
and that engaging dent on the humeral head. The

00:12:28.480 --> 00:12:30.259
full house, if you like. Lethargia, again, I

00:12:30.259 --> 00:12:31.899
assume, because of the bone loss and poor tissue.

00:12:31.960 --> 00:12:34.320
What about the hill -sax in this case? It's lethargia,

00:12:34.539 --> 00:12:38.889
yes, often. Even though the Tarjet provides significant

00:12:38.889 --> 00:12:41.230
stability, especially with that sling effect,

00:12:41.809 --> 00:12:44.169
if you have a really large off -track hill sacks

00:12:44.169 --> 00:12:48.250
or very severe glenoid bone loss, adding Remplasage

00:12:48.250 --> 00:12:51.720
can give that extra bit of security. It neutralizes

00:12:51.720 --> 00:12:54.759
that specific engaging mechanism, even on top

00:12:54.759 --> 00:12:57.279
of the lethargic, kind of a belt and braces approach

00:12:57.279 --> 00:13:00.399
for the toughest cases. Wow. Okay, walking through

00:13:00.399 --> 00:13:03.139
those types really clarifies the logic behind

00:13:03.139 --> 00:13:05.220
each treatment choice when you look at all three

00:13:05.220 --> 00:13:07.460
factors together. Just to quickly recap the roles

00:13:07.460 --> 00:13:09.700
of those, let's call them advanced techniques,

00:13:10.200 --> 00:13:12.840
remplasage seems to be the go -to for that engaging

00:13:12.840 --> 00:13:15.240
the off -track hill sex, no matter the bone or

00:13:15.240 --> 00:13:17.620
tissue situation. Generally, yes, it addresses

00:13:17.620 --> 00:13:20.309
that specific mechanical issue. And DAS comes

00:13:20.309 --> 00:13:22.429
in to give that crucial dynamic support when

00:13:22.429 --> 00:13:24.509
bone loss is subcritical but the soft tissues

00:13:24.509 --> 00:13:26.830
are poor. Correct. It compensates for the poor

00:13:26.830 --> 00:13:30.269
native tissue. And FGBG is presented as the anatomical

00:13:30.269 --> 00:13:33.330
bone block choice for critical bone loss, especially

00:13:33.330 --> 00:13:35.330
if the native soft tissues are still good and

00:13:35.330 --> 00:13:37.870
you want that anatomical restoration. That's

00:13:37.870 --> 00:13:39.529
an excellent summary of their specific roles

00:13:39.529 --> 00:13:42.049
in this framework. The real strength of both

00:13:42.240 --> 00:13:44.720
is precisely that guiding the surgeon to pick

00:13:44.720 --> 00:13:48.220
which tool or combination of tools fix the exact

00:13:48.220 --> 00:13:51.539
mix of bone damage, tissue integrity, and humeral

00:13:51.539 --> 00:13:53.759
head issues. It feels like it takes something

00:13:53.759 --> 00:13:55.779
that was maybe a bit subjective and gives it

00:13:55.779 --> 00:13:58.620
an objective evidence -based structure, a roadmap

00:13:58.620 --> 00:14:01.659
really. It aims to simplify things for surgeons

00:14:01.659 --> 00:14:04.360
and hopefully lead to a more personalized approach

00:14:04.360 --> 00:14:06.559
for patients matching the fix directly to their

00:14:06.559 --> 00:14:09.809
injury, which surely has to mean better outcomes,

00:14:10.289 --> 00:14:12.889
fewer redislocations down the line. That's certainly

00:14:12.889 --> 00:14:15.870
the intention. By standardizing how we assess

00:14:15.870 --> 00:14:17.990
these injuries and linking that assessment directly

00:14:17.990 --> 00:14:20.669
to procedures backed by the best available evidence,

00:14:21.110 --> 00:14:23.090
the hope is definitely to improve consistency

00:14:23.090 --> 00:14:26.679
and optimize outcomes for patients. Our systematic

00:14:26.679 --> 00:14:28.940
review supported these distinctions, too, noting

00:14:28.940 --> 00:14:31.559
things like, yes, letharget might have lower

00:14:31.559 --> 00:14:33.399
recurrence for some complex injuries compared

00:14:33.399 --> 00:14:35.879
to bankart, but also carries a higher infection

00:14:35.879 --> 00:14:38.620
risk. It reinforces that need for careful patient

00:14:38.620 --> 00:14:40.940
selection based on the specific injury pattern.

00:14:41.539 --> 00:14:44.159
And biomechanical studies also back up the reasoning.

00:14:44.330 --> 00:14:47.289
for using techniques like DAS and those specific

00:14:47.289 --> 00:14:50.269
poor tissue scenarios. So the core message seems

00:14:50.269 --> 00:14:52.990
clear. Treating traumatic anterior recurrent

00:14:52.990 --> 00:14:55.629
shoulder instability isn't one size fits all.

00:14:56.009 --> 00:14:58.450
But this both classification offers a really

00:14:58.450 --> 00:15:01.070
powerful framework. By carefully looking at the

00:15:01.070 --> 00:15:04.029
bone loss, the tissue quality, and the hill sacs

00:15:04.029 --> 00:15:06.789
lesion, surgeons can navigate the options bank

00:15:06.789 --> 00:15:11.039
heart, DAS, FGBG, Letarjet, often adding RIMP

00:15:11.039 --> 00:15:14.059
-Lassage to hopefully deliver a surgical solution

00:15:14.059 --> 00:15:17.039
tailored precisely to your unique injury pattern.

00:15:17.379 --> 00:15:19.460
It provides that clear path through the puzzle.

00:15:19.720 --> 00:15:21.960
It helps turn a complex clinical judgment into

00:15:21.960 --> 00:15:25.399
a more structured, evidence -informed process.

00:15:25.519 --> 00:15:27.580
And that leaves us with a final thought to ponder.

00:15:27.639 --> 00:15:30.379
How does creating this kind of detailed classification

00:15:30.379 --> 00:15:33.399
system, one that meticulously breaks down a complex

00:15:33.399 --> 00:15:36.139
problem into its key parts to guide action mirror

00:15:36.139 --> 00:15:38.460
the way we perhaps need to tackle complex decisions

00:15:38.460 --> 00:15:40.940
in other professional fields, especially when

00:15:40.940 --> 00:15:42.919
dealing with lots of variables and significant

00:15:42.919 --> 00:15:45.120
potential risks. It's a very relevant question,

00:15:45.399 --> 00:15:48.139
well beyond the operating theater. Indeed. If

00:15:48.139 --> 00:15:50.759
you found this deep dive useful, please do think

00:15:50.759 --> 00:15:53.620
about rating and sharing it. It really helps

00:15:53.620 --> 00:15:56.139
other people find these discussions. Thank you

00:15:56.139 --> 00:15:58.120
for the opportunity to explore this research

00:15:58.120 --> 00:15:58.419
today.
